PGC1α Suppresses Prostate Cancer Cell Invasion through ERRα Transcriptional Control.


Journal

Cancer research
ISSN: 1538-7445
Titre abrégé: Cancer Res
Pays: United States
ID NLM: 2984705R

Informations de publication

Date de publication:
15 12 2019
Historique:
received: 17 04 2019
revised: 27 08 2019
accepted: 04 10 2019
pubmed: 9 10 2019
medline: 28 5 2020
entrez: 10 10 2019
Statut: ppublish

Résumé

The PPARγ coactivator 1 alpha (PGC1α) is a prostate tumor suppressor that controls the balance between anabolism and catabolism. PGC1A downregulation in prostate cancer is causally associated with the development of metastasis. Here we show that the transcriptional complex formed by PGC1α and estrogen-related receptor 1 alpha (ERRα) controls the aggressive properties of prostate cancer cells. PGC1α expression significantly decreased migration and invasion of various prostate cancer cell lines. This phenotype was consistent with remarkable cytoskeletal remodeling and inhibition of integrin alpha 1 and beta 4 expression, both

Identifiants

pubmed: 31594836
pii: 0008-5472.CAN-19-1231
doi: 10.1158/0008-5472.CAN-19-1231
doi:

Substances chimiques

MYC protein, human 0
PPARGC1A protein, human 0
Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha 0
Proto-Oncogene Proteins c-myc 0
Receptors, Estrogen 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

6153-6165

Subventions

Organisme : Cancer Research UK
ID : 24478
Pays : United Kingdom

Informations de copyright

©2019 American Association for Cancer Research.

Auteurs

Lorea Valcarcel-Jimenez (L)

CIC bioGUNE, Bizkaia, Spain.

Alice Macchia (A)

CIC bioGUNE, Bizkaia, Spain.

Eva Crosas-Molist (E)

Barts Cancer Institute, Queen Mary University of London, London, United Kingdom.
Randall Centre for Cell & Molecular Biophysics, King's College London, London, United Kingdom.

Ariane Schaub-Clerigué (A)

CIC bioGUNE, Bizkaia, Spain.

Laura Camacho (L)

CIC bioGUNE, Bizkaia, Spain.
Biochemistry and Molecular Biology Department, University of the Basque Country (UPV/EHU), Bilbao, Spain.

Natalia Martín-Martín (N)

CIC bioGUNE, Bizkaia, Spain.
CIBERONC, Madrid, Spain.

Paolo Cicogna (P)

CIC bioGUNE, Bizkaia, Spain.

Cristina Viera-Bardón (C)

CIC bioGUNE, Bizkaia, Spain.
CIBERONC, Madrid, Spain.

Sonia Fernández-Ruiz (S)

CIC bioGUNE, Bizkaia, Spain.
CIBERONC, Madrid, Spain.

Irene Rodriguez-Hernandez (I)

Barts Cancer Institute, Queen Mary University of London, London, United Kingdom.
Randall Centre for Cell & Molecular Biophysics, King's College London, London, United Kingdom.

Ivana Hermanova (I)

CIC bioGUNE, Bizkaia, Spain.

Ianire Astobiza (I)

CIC bioGUNE, Bizkaia, Spain.
CIBERONC, Madrid, Spain.

Ana R Cortazar (AR)

CIC bioGUNE, Bizkaia, Spain.
CIBERONC, Madrid, Spain.

Antonio Gomez-Muñoz (A)

Biochemistry and Molecular Biology Department, University of the Basque Country (UPV/EHU), Bilbao, Spain.

Victoria Sanz-Moreno (V)

Barts Cancer Institute, Queen Mary University of London, London, United Kingdom.
Randall Centre for Cell & Molecular Biophysics, King's College London, London, United Kingdom.

Verónica Torrano (V)

CIC bioGUNE, Bizkaia, Spain. acarracedo@cicbiogune.es vtorrano@cicbiogune.es.
Biochemistry and Molecular Biology Department, University of the Basque Country (UPV/EHU), Bilbao, Spain.
CIBERONC, Madrid, Spain.

Arkaitz Carracedo (A)

CIC bioGUNE, Bizkaia, Spain. acarracedo@cicbiogune.es vtorrano@cicbiogune.es.
Biochemistry and Molecular Biology Department, University of the Basque Country (UPV/EHU), Bilbao, Spain.
CIBERONC, Madrid, Spain.
Ikerbasque, Basque Foundation for Science, Bilbao, Spain.

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Classifications MeSH