Pharmacokinetic interaction between modified Atkins diet and antiepileptic drugs in adults with drug-resistant epilepsy.


Journal

Epilepsia
ISSN: 1528-1167
Titre abrégé: Epilepsia
Pays: United States
ID NLM: 2983306R

Informations de publication

Date de publication:
11 2019
Historique:
received: 14 03 2019
revised: 12 09 2019
accepted: 15 09 2019
pubmed: 12 10 2019
medline: 16 4 2020
entrez: 12 10 2019
Statut: ppublish

Résumé

The aim was to examine the influence of modified Atkins diet on serum concentration of antiepileptic drugs (AEDs). Prospective data from 63 adult patients with either focal or generalized drug-resistant epilepsy recruited to 12-week dietary treatment as add-on to AEDs are analyzed. AED serum concentrations, ketones, glucose, and hemoglobin A1c were measured before and after the dietary intervention. Paired t test was used and Spearman correlation coefficient, r, was estimated. Mean age was 37 years (range 16-65 years). Mean serum concentrations of carbamazepine, clobazam, and valproate were significantly reduced after 4 and 12 weeks of the diet period (<.001 ≤ P ≤ .02). Levels of lacosamide, lamotrigine, and topiramate were less reduced (.02 ≤ P ≤ .08), whereas the serum concentrations of oxcarbazepine, zonisamide, and levetiracetam were unchanged (.06 ≤ P ≤ .90). The largest reduction in serum concentration was found for clobazam: mean reduction after 12 weeks was 1.5 μmol/L (34%). Percent change in serum concentration after 4 and 12 weeks of all drugs analyzed was -10.5% (95% confidence interval [CI] -14.1 to -6.8; n = 60; P < .001) and -13.5% (95% CI -18.8 to -8.3; n = 56; P < .001), respectively. Percent change in serum concentration of AEDs was not significantly correlated to percent change in seizure frequency after 12 weeks of dietary treatment (r = .14, P = .33, n = 53) but negatively correlated to urine ketosis (r = -.43; P = .003; n = 46). A reduction in AED serum concentrations may counteract a seizure-reducing effect of the diet, and in patients without such an effect, it may cause seizure aggravation. Thus, we recommend that clinicians who are treating patients with ketogenic diets monitor serum concentrations of the concomitant AEDs.

Identifiants

pubmed: 31602644
doi: 10.1111/epi.16364
doi:

Substances chimiques

Anticonvulsants 0

Banques de données

GENBANK
['NCT01311440']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2235-2244

Informations de copyright

© 2019 The Authors. Epilepsia published by Wiley Periodicals, Inc. on behalf of International League Against Epilepsy.

Références

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Auteurs

Magnhild Kverneland (M)

National Centre for Epilepsy, Oslo University Hospital, Oslo, Norway.
Department of Nutrition, University of Oslo, Oslo, Norway.
Faculty of Medicine, University of Oslo, Oslo, Norway.

Erik Taubøll (E)

Faculty of Medicine, University of Oslo, Oslo, Norway.
Department of Neurology, Oslo University Hospital, Oslo, Norway.

Ellen Molteberg (E)

National Centre for Epilepsy, Oslo University Hospital, Oslo, Norway.
Faculty of Medicine, University of Oslo, Oslo, Norway.

Marit B Veierød (MB)

Oslo Center of Biostatistics and Epidemiology, Department of Biostatistics, University of Oslo, Oslo, Norway.

Kaja K Selmer (KK)

National Centre for Epilepsy, Oslo University Hospital, Oslo, Norway.
Department of Research and Development, Division of Clinical Neuroscience, Oslo University Hospital and the University of Oslo, Oslo, Norway.

Karl O Nakken (KO)

National Centre for Epilepsy, Oslo University Hospital, Oslo, Norway.

Per O Iversen (PO)

Department of Nutrition, University of Oslo, Oslo, Norway.
Faculty of Medicine, University of Oslo, Oslo, Norway.

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