Effects of Interleukin 17A Inhibition on Myocardial Deformation and Vascular Function in Psoriasis.


Journal

The Canadian journal of cardiology
ISSN: 1916-7075
Titre abrégé: Can J Cardiol
Pays: England
ID NLM: 8510280

Informations de publication

Date de publication:
01 2020
Historique:
received: 26 12 2018
revised: 23 05 2019
accepted: 17 06 2019
pubmed: 14 10 2019
medline: 24 7 2020
entrez: 14 10 2019
Statut: ppublish

Résumé

Interleukin (IL)-17A activity is implicated in psoriasis. We investigated the effects of IL-17A inhibition on vascular and left ventricular (LV) function in patients with psoriasis. A total of 150 patients with psoriasis received either an anti-IL-17A agent (secukinumab, n = 50), cyclosporine (n = 50), or methotrexate treatment (n = 50). At baseline and after 4 and 12 months of treatment, we measured (1) LV global longitudinal strain (GLS), GLS rate (GLSR), GLSR at early diastole, LV twisting, and untwisting; (2) coronary flow reserve (CFR); (3) pulse wave velocity (PWV); and (4) malondialdehyde and protein carbonyl as markers of oxidative stress. Compared with cyclosporine and methotrexate, anti-IL-17A treatment resulted in a greater increase in GLS at 4 and 12 months after treatment (10% and 14% with anti-IL-17A vs 2% and 2% with cyclosporine vs 4% and 4% with methotrexate, respectively), GLSR, GLSR at early diastole (45% and 41% vs 5% and 4% vs 7% and 9%, respectively), and LV twisting (32% and 28% vs 6% and 8% vs 7% and 6%, respectively) (P < 0.05). Anti-IL-17A treatment resulted in greater improvement of CFR and PWV than cyclosporine or methotrexate (P < 0.05). PWV increased after cyclosporine treatment (+11% at 4 and +14% and 12 months) (P < 0.05). Markers of oxidative stress were reduced only after anti-IL-17A treatment (P < 0.05). Changes of myocardial deformation markers and CFR after anti-IL-17A treatment correlated with a concomitant reduction of oxidative stress. In psoriasis, inhibition of IL-17A results in a greater improvement of vascular and myocardial function compared with cyclosporine or methotrexate treatment, indicating a beneficial effect on overall cardiovascular function.

Sections du résumé

BACKGROUND
Interleukin (IL)-17A activity is implicated in psoriasis. We investigated the effects of IL-17A inhibition on vascular and left ventricular (LV) function in patients with psoriasis.
METHODS
A total of 150 patients with psoriasis received either an anti-IL-17A agent (secukinumab, n = 50), cyclosporine (n = 50), or methotrexate treatment (n = 50). At baseline and after 4 and 12 months of treatment, we measured (1) LV global longitudinal strain (GLS), GLS rate (GLSR), GLSR at early diastole, LV twisting, and untwisting; (2) coronary flow reserve (CFR); (3) pulse wave velocity (PWV); and (4) malondialdehyde and protein carbonyl as markers of oxidative stress.
RESULTS
Compared with cyclosporine and methotrexate, anti-IL-17A treatment resulted in a greater increase in GLS at 4 and 12 months after treatment (10% and 14% with anti-IL-17A vs 2% and 2% with cyclosporine vs 4% and 4% with methotrexate, respectively), GLSR, GLSR at early diastole (45% and 41% vs 5% and 4% vs 7% and 9%, respectively), and LV twisting (32% and 28% vs 6% and 8% vs 7% and 6%, respectively) (P < 0.05). Anti-IL-17A treatment resulted in greater improvement of CFR and PWV than cyclosporine or methotrexate (P < 0.05). PWV increased after cyclosporine treatment (+11% at 4 and +14% and 12 months) (P < 0.05). Markers of oxidative stress were reduced only after anti-IL-17A treatment (P < 0.05). Changes of myocardial deformation markers and CFR after anti-IL-17A treatment correlated with a concomitant reduction of oxidative stress.
CONCLUSIONS
In psoriasis, inhibition of IL-17A results in a greater improvement of vascular and myocardial function compared with cyclosporine or methotrexate treatment, indicating a beneficial effect on overall cardiovascular function.

Identifiants

pubmed: 31606265
pii: S0828-282X(19)30442-8
doi: 10.1016/j.cjca.2019.06.021
pii:
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
Biomarkers 0
IL17A protein, human 0
Immunosuppressive Agents 0
Interleukin-17 0
Cyclosporine 83HN0GTJ6D
secukinumab DLG4EML025
Methotrexate YL5FZ2Y5U1

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

100-111

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019 Canadian Cardiovascular Society. Published by Elsevier Inc. All rights reserved.

Auteurs

George Makavos (G)

Second Department of Cardiology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

Ignatios Ikonomidis (I)

Second Department of Cardiology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece. Electronic address: ignoik@gmail.com.

Ioanna Andreadou (I)

Department of Pharmaceutical Chemistry, National and Kapodistrian University of Athens School of Pharmacy, Athens, Greece.

Maria Varoudi (M)

Second Department of Cardiology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

Irini Kapniari (I)

Second Department of Dermatology and Venereology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

Eleni Loukeri (E)

Department of Pharmaceutical Chemistry, National and Kapodistrian University of Athens School of Pharmacy, Athens, Greece.

Kostas Theodoropoulos (K)

Second Department of Dermatology and Venereology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

George Pavlidis (G)

Second Department of Cardiology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

Helen Triantafyllidi (H)

Second Department of Cardiology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

John Thymis (J)

Second Department of Cardiology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

John Parissis (J)

Second Department of Cardiology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

Maria Tsoumani (M)

Department of Pharmaceutical Chemistry, National and Kapodistrian University of Athens School of Pharmacy, Athens, Greece.

Pinelopi Rafouli-Stergiou (P)

Second Department of Cardiology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

Pelagia Katsimbri (P)

Rheumatology and Clinical Immunology Unit, Fourth Department of Internal Medicine, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

Evangelia Papadavid (E)

Second Department of Dermatology and Venereology, National and Kapodistrian University of Athens Medical School, Attikon Hospital, Athens, Greece.

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