Gastrointestinal Perforations in Adult Whole-Liver Transplant Patients: Clinical, Radiologic, and Histopathologic Analysis.


Journal

Transplantation proceedings
ISSN: 1873-2623
Titre abrégé: Transplant Proc
Pays: United States
ID NLM: 0243532

Informations de publication

Date de publication:
Nov 2019
Historique:
received: 14 12 2018
accepted: 17 02 2019
pubmed: 15 10 2019
medline: 21 1 2020
entrez: 15 10 2019
Statut: ppublish

Résumé

Gastrointestinal perforation (GIP) is a rare complication after adult liver transplant (LT) associated with high morbidity and mortality. Limited data are available about clinical risk factors and underlying pathogenic mechanisms. The retrospective study included all GIP cases from a consecutive cohort of 361 LT recipients during the period 2005-2017. Clinical variables were investigated as potential risk factors for GIP, and radiologic and histopathologic evaluations were undertaken to identify any causative mechanism. A total of 22 patients developed at least 1 episode of GIP (prevalence 6.1%) at a median time of 18.5 [interquartile range, 12.5-28.5] days after LT. The perforations occurred in the small bowel (63.6%), transverse colon (27.3%), right colon (22.7%), left colon (9.1%), and stomach (9.1%). A total of 27.3% of patients developed multiple sites of GIP, and in 31% GIP recurred after curative surgery. The 30-day mortality rate after relaparotomy was 40%. A history of previous abdominal surgery (odds ratio, 2.5) and early post-LT relaparotomy due to other complications (odds ratio, 2.6) were significant risk factors for GIP. No thromboembolic or steno-occlusive complications of any splanchnic vessel were detected at computed tomography scan, while histopathology examination on perforated gastrointestinal segments excluded cytomegalovirus infection, graft-vs-host disease, and inflammatory bowel disease. In all the cases, ischemic necrosis with aspecific microangiopathy and microembolization were the pathologic features detected. GIP is a severe complication after LT with frequent multiple gastrointestinal involvement and recurrence after curative surgery. The pathologic underlying mechanism is usually microvascular ischemia. Clinical risk factors are history of previous abdominal surgery and early post-LT relaparotomy.

Sections du résumé

BACKGROUND BACKGROUND
Gastrointestinal perforation (GIP) is a rare complication after adult liver transplant (LT) associated with high morbidity and mortality. Limited data are available about clinical risk factors and underlying pathogenic mechanisms.
METHODS METHODS
The retrospective study included all GIP cases from a consecutive cohort of 361 LT recipients during the period 2005-2017. Clinical variables were investigated as potential risk factors for GIP, and radiologic and histopathologic evaluations were undertaken to identify any causative mechanism.
RESULTS RESULTS
A total of 22 patients developed at least 1 episode of GIP (prevalence 6.1%) at a median time of 18.5 [interquartile range, 12.5-28.5] days after LT. The perforations occurred in the small bowel (63.6%), transverse colon (27.3%), right colon (22.7%), left colon (9.1%), and stomach (9.1%). A total of 27.3% of patients developed multiple sites of GIP, and in 31% GIP recurred after curative surgery. The 30-day mortality rate after relaparotomy was 40%. A history of previous abdominal surgery (odds ratio, 2.5) and early post-LT relaparotomy due to other complications (odds ratio, 2.6) were significant risk factors for GIP. No thromboembolic or steno-occlusive complications of any splanchnic vessel were detected at computed tomography scan, while histopathology examination on perforated gastrointestinal segments excluded cytomegalovirus infection, graft-vs-host disease, and inflammatory bowel disease. In all the cases, ischemic necrosis with aspecific microangiopathy and microembolization were the pathologic features detected.
CONCLUSIONS CONCLUSIONS
GIP is a severe complication after LT with frequent multiple gastrointestinal involvement and recurrence after curative surgery. The pathologic underlying mechanism is usually microvascular ischemia. Clinical risk factors are history of previous abdominal surgery and early post-LT relaparotomy.

Identifiants

pubmed: 31607622
pii: S0041-1345(18)31629-4
doi: 10.1016/j.transproceed.2019.02.068
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2974-2976

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

Auteurs

Riccardo Pravisani (R)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

Umberto Baccarani (U)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy. Electronic address: umberto.baccarani@uniud.it.

Francesco Toso (F)

Department of Radiology, ASUIUD, Udine, Italy.

Gian Luigi Adani (GL)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

Dario Lorenzin (D)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

Vittorio Cherchi (V)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

Sergio Calandra (S)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

Edoardo Scarpa (E)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

Sara Crestale (S)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

Claudio Avellini (C)

Department of Pathology, ASUIUD, Udine, Italy.

Giovanni Terrosu (G)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

Massimo Sponza (M)

Department of Radiology, ASUIUD, Udine, Italy.

Andrea Risaliti (A)

Liver-Kidney Transplant Unit - Department of Medicine, University of Udine, Udine, Italy.

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