Intracellular Pharmacodynamic Modeling Is Predictive of the Clinical Activity of Fluoroquinolones against Tuberculosis.


Journal

Antimicrobial agents and chemotherapy
ISSN: 1098-6596
Titre abrégé: Antimicrob Agents Chemother
Pays: United States
ID NLM: 0315061

Informations de publication

Date de publication:
20 12 2019
Historique:
received: 22 05 2019
accepted: 18 09 2019
pubmed: 16 10 2019
medline: 1 9 2020
entrez: 16 10 2019
Statut: epublish

Résumé

Clinical studies of new antitubercular drugs are costly and time-consuming. Owing to the extensive tuberculosis (TB) treatment periods, the ability to identify drug candidates based on their predicted clinical efficacy is vital to accelerate the pipeline of new therapies. Recent failures of preclinical models in predicting the activity of fluoroquinolones underline the importance of developing new and more robust predictive tools that will optimize the design of future trials. Here, we used high-content imaging screening and pharmacodynamic intracellular (PDi) modeling to identify and prioritize fluoroquinolones for TB treatment. In a set of studies designed to validate this approach, we show moxifloxacin to be the most effective fluoroquinolone, and PDi modeling-based Monte Carlo simulations accurately predict negative culture conversion (sputum sterilization) rates compared to eight independent clinical trials. In addition, PDi-based simulations were used to predict the risk of relapse. Our analyses show that the duration of treatment following culture conversion can be used to predict the relapse rate. These data further support that PDi-based modeling offers a much-needed decision-making tool for the TB drug development pipeline.

Identifiants

pubmed: 31611354
pii: AAC.00989-19
doi: 10.1128/AAC.00989-19
pmc: PMC7187570
pii:
doi:

Substances chimiques

Antitubercular Agents 0
Fluoroquinolones 0
Moxifloxacin U188XYD42P

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Wellcome Trust
ID : 105620/Z/14/Z
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/N028376/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC_PC_14111
Pays : United Kingdom
Organisme : Medical Research Council
ID : G1002586
Pays : United Kingdom
Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/L000644/1
Pays : United Kingdom

Informations de copyright

Copyright © 2019 Donnellan et al.

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Auteurs

Samantha Donnellan (S)

Centre for Drugs and Diagnostics, Department of Tropical Disease Biology, Liverpool School of Tropical Medicine, Pembroke Place, Liverpool, United Kingdom.

Ghaith Aljayyoussi (G)

Centre for Drugs and Diagnostics, Department of Tropical Disease Biology, Liverpool School of Tropical Medicine, Pembroke Place, Liverpool, United Kingdom.

Emmanuel Moyo (E)

Centre for Drugs and Diagnostics, Department of Tropical Disease Biology, Liverpool School of Tropical Medicine, Pembroke Place, Liverpool, United Kingdom.

Alison Ardrey (A)

Centre for Drugs and Diagnostics, Department of Tropical Disease Biology, Liverpool School of Tropical Medicine, Pembroke Place, Liverpool, United Kingdom.

Carmen Martinez-Rodriguez (C)

Institute of Infection and Global Health, University of Liverpool, Liverpool, United Kingdom.

Stephen A Ward (SA)

Centre for Drugs and Diagnostics, Department of Tropical Disease Biology, Liverpool School of Tropical Medicine, Pembroke Place, Liverpool, United Kingdom.

Giancarlo A Biagini (GA)

Centre for Drugs and Diagnostics, Department of Tropical Disease Biology, Liverpool School of Tropical Medicine, Pembroke Place, Liverpool, United Kingdom giancarlo.biagini@lstmed.ac.uk.

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