Treatment of atopic dermatitis with ruxolitinib cream (JAK1/JAK2 inhibitor) or triamcinolone cream.


Journal

The Journal of allergy and clinical immunology
ISSN: 1097-6825
Titre abrégé: J Allergy Clin Immunol
Pays: United States
ID NLM: 1275002

Informations de publication

Date de publication:
02 2020
Historique:
received: 02 04 2019
revised: 23 08 2019
accepted: 28 08 2019
pubmed: 21 10 2019
medline: 23 9 2020
entrez: 21 10 2019
Statut: ppublish

Résumé

Atopic dermatitis (AD) is a highly pruritic chronic inflammatory skin disorder. Ruxolitinib, a selective inhibitor of Janus kinase 1 and Janus kinase 2, potently suppresses cytokine signaling involved in AD pathogenesis. We sought to evaluate the efficacy and safety of ruxolitinib (RUX) cream in adults with AD. In this phase 2 study (NCT03011892), 307 adult patients with AD, an Investigator's Global Assessment score of 2 or 3 (mild or moderate), and 3% to 20% affected body surface area were equally randomized for 8 weeks of double-blind treatment to RUX (1.5% twice daily [BID], 1.5% once daily [QD], 0.5% QD, 0.15% QD), vehicle, or triamcinolone cream (0.1% BID for 4 weeks, then vehicle for 4 weeks). Subsequently, patients could apply 1.5% RUX BID for 4 additional weeks of open-label treatment. The primary end point was the comparison between 1.5% RUX cream BID and vehicle in mean percentage change from baseline in Eczema Area and Severity Index at week 4. All RUX regimens demonstrated therapeutic benefit at week 4; 1.5% BID provided the greatest improvement in Eczema Area and Severity Index (71.6% vs 15.5%; P < .0001) and Investigator's Global Assessment responses (38.0% vs 7.7%; P < .001) versus vehicle. Rapid reductions in the itch numerical rating scale score occurred within 36 hours (1.5% BID vs vehicle, ‒1.8 vs ‒0.2; P < .0001) and were sustained through 12 weeks. Patients who transitioned to 1.5% RUX BID improved in all measures. RUX was not associated with clinically significant application-site reactions. RUX cream provided rapid and sustained improvements in AD symptoms and was well tolerated.

Sections du résumé

BACKGROUND
Atopic dermatitis (AD) is a highly pruritic chronic inflammatory skin disorder. Ruxolitinib, a selective inhibitor of Janus kinase 1 and Janus kinase 2, potently suppresses cytokine signaling involved in AD pathogenesis.
OBJECTIVE
We sought to evaluate the efficacy and safety of ruxolitinib (RUX) cream in adults with AD.
METHODS
In this phase 2 study (NCT03011892), 307 adult patients with AD, an Investigator's Global Assessment score of 2 or 3 (mild or moderate), and 3% to 20% affected body surface area were equally randomized for 8 weeks of double-blind treatment to RUX (1.5% twice daily [BID], 1.5% once daily [QD], 0.5% QD, 0.15% QD), vehicle, or triamcinolone cream (0.1% BID for 4 weeks, then vehicle for 4 weeks). Subsequently, patients could apply 1.5% RUX BID for 4 additional weeks of open-label treatment. The primary end point was the comparison between 1.5% RUX cream BID and vehicle in mean percentage change from baseline in Eczema Area and Severity Index at week 4.
RESULTS
All RUX regimens demonstrated therapeutic benefit at week 4; 1.5% BID provided the greatest improvement in Eczema Area and Severity Index (71.6% vs 15.5%; P < .0001) and Investigator's Global Assessment responses (38.0% vs 7.7%; P < .001) versus vehicle. Rapid reductions in the itch numerical rating scale score occurred within 36 hours (1.5% BID vs vehicle, ‒1.8 vs ‒0.2; P < .0001) and were sustained through 12 weeks. Patients who transitioned to 1.5% RUX BID improved in all measures. RUX was not associated with clinically significant application-site reactions.
CONCLUSIONS
RUX cream provided rapid and sustained improvements in AD symptoms and was well tolerated.

Identifiants

pubmed: 31629805
pii: S0091-6749(19)31326-0
doi: 10.1016/j.jaci.2019.08.042
pii:
doi:

Substances chimiques

Anti-Inflammatory Agents 0
Nitriles 0
Pyrazoles 0
Pyrimidines 0
Triamcinolone 1ZK20VI6TY
ruxolitinib 82S8X8XX8H
JAK1 protein, human EC 2.7.10.2
JAK2 protein, human EC 2.7.10.2
Janus Kinase 1 EC 2.7.10.2
Janus Kinase 2 EC 2.7.10.2

Banques de données

ClinicalTrials.gov
['NCT03011892']

Types de publication

Clinical Trial, Phase II Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

572-582

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Brian S Kim (BS)

Washington University School of Medicine, St Louis, Mo. Electronic address: briankim@wustl.edu.

Michael D Howell (MD)

Incyte Corporation, Wilmington, Del.

Kang Sun (K)

Incyte Corporation, Wilmington, Del.

Kim Papp (K)

K. Papp Clinical Research and Probity Medical Research, Waterloo, Ontario, Canada.

Adnan Nasir (A)

Wake Research Associates LLC, Raleigh, NC.

Michael E Kuligowski (ME)

Incyte Corporation, Wilmington, Del.

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Classifications MeSH