NAD
Acute Kidney Injury
/ metabolism
Adenosine Triphosphate
/ metabolism
Animals
Humans
Kidney Tubules
/ metabolism
NAD
/ metabolism
NADP
/ metabolism
Oxidation-Reduction
Poly(ADP-ribose) Polymerases
/ metabolism
Renal Insufficiency, Chronic
/ metabolism
Reperfusion Injury
/ metabolism
Sirtuins
/ metabolism
Journal
Nature reviews. Nephrology
ISSN: 1759-507X
Titre abrégé: Nat Rev Nephrol
Pays: England
ID NLM: 101500081
Informations de publication
Date de publication:
02 2020
02 2020
Historique:
accepted:
19
09
2019
pubmed:
2
11
2019
medline:
17
4
2020
entrez:
2
11
2019
Statut:
ppublish
Résumé
The mammalian kidney relies on abundant mitochondria in the renal tubule to generate sufficient ATP to provide the energy required for constant reclamation of solutes from crude blood filtrate. The highly metabolically active cells of the renal tubule also pair their energetic needs to the regulation of diverse cellular processes, including energy generation, antioxidant responses, autophagy and mitochondrial quality control. Nicotinamide adenine dinucleotide (NAD
Identifiants
pubmed: 31673160
doi: 10.1038/s41581-019-0216-6
pii: 10.1038/s41581-019-0216-6
pmc: PMC7223841
doi:
Substances chimiques
NAD
0U46U6E8UK
NADP
53-59-8
Adenosine Triphosphate
8L70Q75FXE
Poly(ADP-ribose) Polymerases
EC 2.4.2.30
Sirtuins
EC 3.5.1.-
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
99-111Références
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