Association of Galectin-3 and Soluble ST2, and Their Changes, with Echocardiographic Parameters and Development of Heart Failure after ST-Segment Elevation Myocardial Infarction.


Journal

Disease markers
ISSN: 1875-8630
Titre abrégé: Dis Markers
Pays: United States
ID NLM: 8604127

Informations de publication

Date de publication:
2019
Historique:
received: 23 04 2019
accepted: 23 08 2019
entrez: 6 11 2019
pubmed: 7 11 2019
medline: 17 4 2020
Statut: epublish

Résumé

To investigate the association of galectin-3 (Gal-3) and soluble ST2 (sST2) and their follow-up changes with the development of heart failure (HF) and echocardiographic parameters of HF (ejection fraction, atrial and ventricular size, left ventricular hypertrophy, e', and E/e') in patients with ST-segment elevation myocardial infarction (STEMI) treated with primary percutaneous coronary intervention (pPCI). A prospective, observational study, BIOSTRAT (Biomarkers for Risk Stratification After STEMI), enrolled 117 patients between October 2014 and April 2017. Gal-3 and sST2 serum collection and echocardiography were performed twice (during index hospitalization and on a control visit at one-year follow-up). The primary endpoint was HF onset at one-year follow-up. Secondary assessments included associations of biomarker concentration with echocardiographic indices of systolic and diastolic dysfunction at baseline and at one year. Mean baseline concentrations of Gal-3 and sST2 (7.5 and 26.4 ng/mL, respectively) were significantly increased at one-year follow-up (8.5 ng/mL and Baseline, but not one-year, changes of Gal-3 and sST2 concentrations may be useful for risk stratification after STEMI. However, only Gal-3 was the independent predictor of HF development at one-year observation. This trial is registered with NCT03735719.

Identifiants

pubmed: 31687050
doi: 10.1155/2019/9529053
pmc: PMC6811795
doi:

Substances chimiques

Biomarkers 0
Blood Proteins 0
Galectin 3 0
Galectins 0
IL1RL1 protein, human 0
Interleukin-1 Receptor-Like 1 Protein 0
LGALS3 protein, human 0

Banques de données

ClinicalTrials.gov
['NCT03735719']

Types de publication

Clinical Trial Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

9529053

Informations de copyright

Copyright © 2019 Agata Tymińska et al.

Déclaration de conflit d'intérêts

The authors declare that there is no conflict of interest regarding the publication of this article.

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Auteurs

Agata Tymińska (A)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Agnieszka Kapłon-Cieślicka (A)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Krzysztof Ozierański (K)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Monika Budnik (M)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Anna Wancerz (A)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Piotr Sypień (P)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Michał Peller (M)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Paweł Balsam (P)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Grzegorz Opolski (G)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

Krzysztof J Filipiak (KJ)

1st Department of Cardiology, Medical University of Warsaw, Warsaw, Poland.

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