Locations, associations and temporal evolution of intracranial arterial infundibular dilatations in children.


Journal

Journal of neurointerventional surgery
ISSN: 1759-8486
Titre abrégé: J Neurointerv Surg
Pays: England
ID NLM: 101517079

Informations de publication

Date de publication:
May 2020
Historique:
received: 09 09 2019
revised: 11 10 2019
accepted: 13 10 2019
pubmed: 9 11 2019
medline: 8 9 2020
entrez: 9 11 2019
Statut: ppublish

Résumé

There are few data in the literature on the characteristics and natural history of intracranial arterial infundibular dilatations in children. An institutional review board-approved retrospective review was performed of infundibula reported on MR angiography in patients <18 years of age at our tertiary pediatric institute from 1998 to 2016. Clinical data (age, sex, diagnosis, other vascular variants/pathologies) were recorded and images assessed for vessel of origin, infundibulum size and exact location. Ratios of infundibulum:parent artery were assessed at diagnosis and last follow-up. Temporal evolution to aneurysm was evaluated. We found 60 intracranial infundibula in 60 children (male:female=27:33; mean age 9.7±5.2 years, range 2-18 years,). Family history of aneurysms was present in 2/60 (3.3%). Syndromic association was found in 14/60 (23.3%), most frequently sickle cell disease (4/14=28.6%). Mean infundibulum size was 2.2±0.5 mm, with mean ratio to parent artery of 0.54±0.17. The most common location was on the P1-posterior cerebral artery (34/63=56.7%), whereas posterior communicating infundibula were seen in only 4/60 (6.7%) cases. Other cerebrovascular variants were seen in 12/60 (20%) patients. On follow-up imaging (in 32/60 patients over 86 patient-years, mean 32.3±35.7 months), no significant change in infundibulum:parent artery ratio was noted. None of the infundibular dilatations showed interval evolution to aneurysm. We present the largest reported cohort of pediatric intracranial arterial infundibula, which we found to be distinct from their adult counterparts with regard to location, etiology and temporal evolution. Growth over time and/or aneurysmal formation are rare, not necessitating frequent short-term imaging surveillance during childhood.

Sections du résumé

BACKGROUND BACKGROUND
There are few data in the literature on the characteristics and natural history of intracranial arterial infundibular dilatations in children.
METHODS METHODS
An institutional review board-approved retrospective review was performed of infundibula reported on MR angiography in patients <18 years of age at our tertiary pediatric institute from 1998 to 2016. Clinical data (age, sex, diagnosis, other vascular variants/pathologies) were recorded and images assessed for vessel of origin, infundibulum size and exact location. Ratios of infundibulum:parent artery were assessed at diagnosis and last follow-up. Temporal evolution to aneurysm was evaluated.
RESULTS RESULTS
We found 60 intracranial infundibula in 60 children (male:female=27:33; mean age 9.7±5.2 years, range 2-18 years,). Family history of aneurysms was present in 2/60 (3.3%). Syndromic association was found in 14/60 (23.3%), most frequently sickle cell disease (4/14=28.6%). Mean infundibulum size was 2.2±0.5 mm, with mean ratio to parent artery of 0.54±0.17. The most common location was on the P1-posterior cerebral artery (34/63=56.7%), whereas posterior communicating infundibula were seen in only 4/60 (6.7%) cases. Other cerebrovascular variants were seen in 12/60 (20%) patients. On follow-up imaging (in 32/60 patients over 86 patient-years, mean 32.3±35.7 months), no significant change in infundibulum:parent artery ratio was noted. None of the infundibular dilatations showed interval evolution to aneurysm.
CONCLUSION CONCLUSIONS
We present the largest reported cohort of pediatric intracranial arterial infundibula, which we found to be distinct from their adult counterparts with regard to location, etiology and temporal evolution. Growth over time and/or aneurysmal formation are rare, not necessitating frequent short-term imaging surveillance during childhood.

Identifiants

pubmed: 31699885
pii: neurintsurg-2019-015433
doi: 10.1136/neurintsurg-2019-015433
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

495-498

Informations de copyright

© Author(s) (or their employer(s)) 2020. No commercial re-use. See rights and permissions. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: None declared.

Auteurs

Adam A Dmytriw (AA)

Department of Diagnostic Imaging & Image Guided Therapy, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada adam.dmytriw@sickkids.ca.

Daniel-Alexandre Bisson (DA)

Department of Diagnostic Imaging & Image Guided Therapy, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada.

Kevin Phan (K)

Department of Diagnostic Imaging & Image Guided Therapy, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada.

Afsaneh Amirabadi (A)

Department of Diagnostic Imaging & Image Guided Therapy, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada.

Helen Branson (H)

Department of Diagnostic Imaging & Image Guided Therapy, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada.

Peter B Dirks (PB)

Division of Neurosurgery, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada.

Manohar Shroff (M)

Department of Diagnostic Imaging & Image Guided Therapy, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada.

Prakash Muthusami (P)

Department of Diagnostic Imaging & Image Guided Therapy, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada.

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