The effects of psychosocial stress on dopaminergic function and the acute stress response.
PET
adversity
human
imaging
neuroscience
psychosocial
stress
threat
Journal
eLife
ISSN: 2050-084X
Titre abrégé: Elife
Pays: England
ID NLM: 101579614
Informations de publication
Date de publication:
12 11 2019
12 11 2019
Historique:
received:
12
03
2019
accepted:
12
10
2019
entrez:
13
11
2019
pubmed:
13
11
2019
medline:
6
5
2020
Statut:
epublish
Résumé
Chronic psychosocial adversity induces vulnerability to mental illnesses. Animal studies demonstrate that this may be mediated by dopaminergic dysfunction. We therefore investigated whether long-term exposure to psychosocial adversity was associated with dopamine dysfunction and its relationship to psychological and physiological responses to acute stress. Using 3,4-dihydroxy-6-[
Identifiants
pubmed: 31711569
doi: 10.7554/eLife.46797
pii: 46797
pmc: PMC6850765
doi:
pii:
Substances chimiques
Dopamine
VTD58H1Z2X
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Medical Research Council
ID : MR/N027078/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/L022176/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC_U120097115
Pays : United Kingdom
Organisme : National Institute for Health Research
ID : University College London Hospitals Biomedical Research Centre
Pays : International
Organisme : Medical Research Council
ID : MR/N026063/1
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 094849/Z/10/Z
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC-A656-5QD30
Pays : United Kingdom
Organisme : Medical Research Council
ID : G0700995
Pays : United Kingdom
Organisme : National Institute for Health Research
ID : Biomedical Research Centre at South London and Maudsley NHS Foundation Trust and King's College London
Pays : International
Informations de copyright
© 2019, Bloomfield et al.
Déclaration de conflit d'intérêts
MB, RM, MK, TF No competing interests declared, OH Professor Howes has received investigator-initiated research funding from and/or participated in advisory/speaker meetings organised by Astra-Zeneca, Autifony, BMS, Eli Lilly, Heptares, Jansenn, Lundbeck, Lyden-Delta, Otsuka, Servier, Sunovion, Rand and Roche. Neither Professor Howes nor his family have been employed by or have holdings/a financial stake in any biomedical company.
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