MRI-Based Manual versus Automated Corpus Callosum Volumetric Measurements in Multiple Sclerosis.


Journal

Journal of neuroimaging : official journal of the American Society of Neuroimaging
ISSN: 1552-6569
Titre abrégé: J Neuroimaging
Pays: United States
ID NLM: 9102705

Informations de publication

Date de publication:
03 2020
Historique:
received: 15 07 2019
revised: 13 10 2019
accepted: 26 10 2019
pubmed: 22 11 2019
medline: 15 12 2020
entrez: 22 11 2019
Statut: ppublish

Résumé

Corpus callosum atrophy is a neurodegenerative biomarker in multiple sclerosis (MS). Manual delineations are gold standard but subjective and labor intensive. Novel automated methods are promising but require validation. We aimed to compare the robustness of manual versus automatic corpus callosum segmentations based on FreeSurfer. Nine MS patients (6 females, age 38 ± 13 years, disease duration 7.3 ± 5.2 years) were scanned twice with repositioning using 3-dimensional T Manual measurements had high intrarater (junior doctor .96 and neuroradiologist .96) and interrater agreement (.94), by intraclass correlation coefficient (P < .001). The coefficient of variation was lowest for longitudinal FreeSurfer (.96% within scanners; 2.0% between scanners) compared to cross-sectional FreeSurfer (3.7%, P = .001; 3.8%, P = .058) and the neuroradiologist (2.3%, P = .005; 2.4%, P = .33). Longitudinal FreeSurfer was also more accurate than cross-sectional (Dice scores 83.9 ± 7.5% vs. 78.9 ± 8.4%, P < .01 relative to manual segmentations). The corpus callosum measures correlated with physical disability (longitudinal FreeSurfer r = -.36, P < .01; neuroradiologist r = -.32, P < .01) and cognitive disability (longitudinal FreeSurfer r = .68, P < .001; neuroradiologist r = .64, P < .001). FreeSurfer's longitudinal stream provides corpus callosum measures with better repeatability than current manual methods and with similar clinical correlations. However, due to some limitations in accuracy, caution is warranted when using FreeSurfer with clinical data.

Sections du résumé

BACKGROUND AND PURPOSE
Corpus callosum atrophy is a neurodegenerative biomarker in multiple sclerosis (MS). Manual delineations are gold standard but subjective and labor intensive. Novel automated methods are promising but require validation. We aimed to compare the robustness of manual versus automatic corpus callosum segmentations based on FreeSurfer.
METHODS
Nine MS patients (6 females, age 38 ± 13 years, disease duration 7.3 ± 5.2 years) were scanned twice with repositioning using 3-dimensional T
RESULTS
Manual measurements had high intrarater (junior doctor .96 and neuroradiologist .96) and interrater agreement (.94), by intraclass correlation coefficient (P < .001). The coefficient of variation was lowest for longitudinal FreeSurfer (.96% within scanners; 2.0% between scanners) compared to cross-sectional FreeSurfer (3.7%, P = .001; 3.8%, P = .058) and the neuroradiologist (2.3%, P = .005; 2.4%, P = .33). Longitudinal FreeSurfer was also more accurate than cross-sectional (Dice scores 83.9 ± 7.5% vs. 78.9 ± 8.4%, P < .01 relative to manual segmentations). The corpus callosum measures correlated with physical disability (longitudinal FreeSurfer r = -.36, P < .01; neuroradiologist r = -.32, P < .01) and cognitive disability (longitudinal FreeSurfer r = .68, P < .001; neuroradiologist r = .64, P < .001).
CONCLUSIONS
FreeSurfer's longitudinal stream provides corpus callosum measures with better repeatability than current manual methods and with similar clinical correlations. However, due to some limitations in accuracy, caution is warranted when using FreeSurfer with clinical data.

Identifiants

pubmed: 31750599
doi: 10.1111/jon.12676
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

198-204

Informations de copyright

© 2019 by the American Society of Neuroimaging.

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Auteurs

Michael Platten (M)

Department of Clinical Neuroscience, Karolinska Institute, Stockholm, Sweden.
Division of Neuroradiology, Department of Radiology, Karolinska University Hospital, Stockholm, Sweden.
School of Engineering Sciences in Chemistry, Biotechnology and Health, Royal Institute of Technology, Stockholm, Sweden.

Juha Martola (J)

Department of Clinical Neuroscience, Karolinska Institute, Stockholm, Sweden.

Katharina Fink (K)

Department of Clinical Neuroscience, Karolinska Institute, Stockholm, Sweden.
Center for Neurology, Academic Specialist Center, Stockholm Health Services, Stockholm, Sweden.

Russell Ouellette (R)

Department of Clinical Neuroscience, Karolinska Institute, Stockholm, Sweden.
Division of Neuroradiology, Department of Radiology, Karolinska University Hospital, Stockholm, Sweden.

Fredrik Piehl (F)

Department of Clinical Neuroscience, Karolinska Institute, Stockholm, Sweden.
Department of Neurology, Karolinska University Hospital, Stockholm, Sweden.
Center for Neurology, Academic Specialist Center, Stockholm Health Services, Stockholm, Sweden.

Tobias Granberg (T)

Department of Clinical Neuroscience, Karolinska Institute, Stockholm, Sweden.
Division of Neuroradiology, Department of Radiology, Karolinska University Hospital, Stockholm, Sweden.

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