Pharmacotherapy for metabolic and cellular stress in degenerative retinal diseases.


Journal

Drug discovery today
ISSN: 1878-5832
Titre abrégé: Drug Discov Today
Pays: England
ID NLM: 9604391

Informations de publication

Date de publication:
02 2020
Historique:
received: 24 06 2019
revised: 08 11 2019
accepted: 26 11 2019
pubmed: 7 12 2019
medline: 1 5 2021
entrez: 7 12 2019
Statut: ppublish

Résumé

Retinal photoreceptors continually endure stresses associated with prolonged light exposure and the metabolic demands of dark adaptation. Although healthy photoreceptors are able to withstand these stresses for several decades, the disease-affected retina functions at a reduced capacity and is at an increased risk for dysfunction. To alleviate cellular and metabolic stressors in degenerative retinal diseases, a new class of drugs that modulate the metabolic activity of the retina have been developed. A clinical candidate in this class (emixustat) has been shown to reduce retinal pathology in various animal models of human retinal disease and is currently under clinical study. Here, we describe the pharmacological properties of emixustat, its mechanisms of action, and potential for use in the treatment of specific retinal diseases.

Identifiants

pubmed: 31809750
pii: S1359-6446(19)30455-6
doi: 10.1016/j.drudis.2019.11.013
pii:
doi:

Substances chimiques

Phenyl Ethers 0
Propanolamines 0
emixustat 02DZ1HBF0M

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

292-304

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Ryo Kubota (R)

Acucela Inc., 818 Stewart St, Suite 1110, Seattle, WA 9810, USA. Electronic address: Dr.Kubota@acucela.com.

Jeff Gregory (J)

Acucela Inc., 818 Stewart St, Suite 1110, Seattle, WA 9810, USA.

Susan Henry (S)

Acucela Inc., 818 Stewart St, Suite 1110, Seattle, WA 9810, USA.

Nathan L Mata (NL)

Halloran Consulting Group, 266 Summer St, Boston, MA 02210, USA.

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Classifications MeSH