Autoimmune markers for progression of Libby amphibole lamellar pleural thickening.


Journal

Inhalation toxicology
ISSN: 1091-7691
Titre abrégé: Inhal Toxicol
Pays: England
ID NLM: 8910739

Informations de publication

Date de publication:
Historique:
pubmed: 10 12 2019
medline: 27 5 2020
entrez: 10 12 2019
Statut: ppublish

Résumé

Exposure to Libby Asbestiform Amphibole (LAA) is associated with asbestos-related diseases, including mesothelioma, pulmonary carcinoma, pleural fibrosis, and systemic autoimmune diseases. The pleural fibrosis can manifest as a rapidly progressing lamellar pleural thickening (LPT), which causes thoracic pain, dyspnea, and worsening pulmonary function tests (PFT). It is refractory to treatment and frequently fatal.

Identifiants

pubmed: 31814459
doi: 10.1080/08958378.2019.1699616
pmc: PMC7450909
mid: NIHMS1621095
doi:

Substances chimiques

Antibodies, Antinuclear 0
Asbestos, Amphibole 0
Autoantibodies 0
Biomarkers 0
Cytokines 0
Collagen 9007-34-5

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

409-419

Subventions

Organisme : NCATS NIH HHS
ID : UL1 TR002319
Pays : United States

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Auteurs

Jean C Pfau (JC)

Department of Microbiology and Immunology, Montana State University, Bozeman, MT, USA.

Tracy McNew (T)

Center for Asbestos Related Diseases, Libby, MT, USA.

Kerensa Hanley (K)

Center for Asbestos Related Diseases, Libby, MT, USA.

Lindsay Swan (L)

Department of Microbiology and Immunology, Montana State University, Bozeman, MT, USA.

Brad Black (B)

Center for Asbestos Related Diseases, Libby, MT, USA.

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Classifications MeSH