IQ motif-containing GTPase-activating protein 1 is essential for the optimal maintenance of lung ILC2s.


Journal

International immunology
ISSN: 1460-2377
Titre abrégé: Int Immunol
Pays: England
ID NLM: 8916182

Informations de publication

Date de publication:
12 04 2020
Historique:
received: 03 10 2019
accepted: 09 12 2019
pubmed: 11 12 2019
medline: 2 10 2020
entrez: 11 12 2019
Statut: ppublish

Résumé

Group 2 innate lymphoid cells (ILC2s) play critical roles in type 2 immunity and are crucial for pathogenesis of various types of inflammatory disease. IQ motif-containing GTPase-activating protein 1 (IQGAP1) is a ubiquitously expressed scaffold protein that is involved in multiple cellular functions such as cell survival and trafficking. While the roles for IQGAP1 in T and B lymphocytes have been uncovered, the physiological significance of IQGAP1 in innate lymphocytes remains to be elucidated. In the current study, we demonstrate that using bone marrow chimeras, the deficiency of IQGAP1 caused an impaired survival of lung ILC2s in a cell-intrinsic manner and that Iqgap1-/- mice displayed decreased accumulation of ILC2s after administration of papain and thereby reduced the pathology of the disease. Moreover, Iqgap1-/- ILC2s showed a significantly enhanced apoptosis as compared to wild-type ILC2s under both steady-state and inflammatory conditions. Together these results identify for the first time that IQGAP1 is essential for homeostasis of ILC2s in the lung.

Identifiants

pubmed: 31819988
pii: 5670825
doi: 10.1093/intimm/dxz077
doi:

Substances chimiques

IQ motif containing GTPase activating protein 1 0
ras GTPase-Activating Proteins 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

233-241

Informations de copyright

© The Japanese Society for Immunology. 2019. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Auteurs

Shunichi Tayama (S)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Yuko Okuyama (Y)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Hai The Phung (HT)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Atsuko Asao (A)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Shuhei Kobayashi (S)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Tomomi Musha (T)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Tomoaki Machiyama (T)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Tsuyoshi Sakurai (T)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Chengming Zhang (C)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Masuko Ushio-Fukai (M)

Department of Medicine (Cardiology), Augusta University, Augusta, GA, USA.

Takeshi Kawabe (T)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Takanori So (T)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.
Laboratory of Molecular Cell Biology, Graduate School of Medicine and Pharmaceutical Science, University of Toyama, Toyama, Japan.

Naoto Ishii (N)

Department of Microbiology and Immunology, Tohoku University Graduate School of Medicine, Sendai, Japan.

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