Symmetry of ictal slow waves may predict the outcomes of corpus callosotomy for epileptic spasms.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
24 12 2019
Historique:
received: 01 10 2018
accepted: 05 12 2019
entrez: 26 12 2019
pubmed: 26 12 2019
medline: 5 11 2020
Statut: epublish

Résumé

We aimed to analyse the ictal electrographic changes on scalp electroencephalography (EEG), focusing on high-voltage slow waves (HVSs) in children with epileptic spasms (ES) and tonic spasms (TS) and then identified factors associated with corpus callosotomy (CC) outcomes. We enrolled 17 patients with ES/TS who underwent CC before 20 years of age. Post-CC Engel's classification was as follows: I in 7 patients, II in 2, III in 4, and IV in 4. Welch's t-test was used to analyse the correlation between ictal HVSs and CC outcomes based on the following three symmetrical indices: (1) negative peak delay: interhemispheric delay between negative peaks; (2) amplitude ratio: interhemispheric ratio of amplitude values for the highest positive peaks; and (3) duration ratio: interhemispheric ratio of slow wave duration. Ages at CC ranged from 17-237 months. Four to 15 ictal EEGs were analysed for each patient. The negative peak delay, amplitude ratio and duration ratio ranged from 0-530 ms, 1.00-7.40 and 1.00-2.74, respectively. The negative peak delay, amplitude ratio and duration ratio were significantly higher in the seizure residual group (p = 0.017, <0.001, <0.001, respectively). Symmetry of ictal HVSs may predict favourable outcomes following CC for ES/TS.

Identifiants

pubmed: 31875025
doi: 10.1038/s41598-019-56303-3
pii: 10.1038/s41598-019-56303-3
pmc: PMC6930281
doi:

Types de publication

Clinical Trial Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

19733

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Auteurs

Sotaro Kanai (S)

Department of Child Neurology, Seirei-Hamamatsu General Hospital, 2-12-12 Sumiyoshi, Hamamatsu, 430-8558, Japan.
Division of Child Neurology, Institute of Neurological Sciences, Faculty of Medicine, Tottori University, 86 Nishimachi, Yonago, 683-8503, Japan.

Masayoshi Oguri (M)

Division of Pathobiological Science and Technology, School of Health Sciences, Faculty of Medicine, Tottori University, 86 Nishimachi, Yonago, 683-8503, Japan.

Tohru Okanishi (T)

Department of Child Neurology, Seirei-Hamamatsu General Hospital, 2-12-12 Sumiyoshi, Hamamatsu, 430-8558, Japan. okanishipediatrics@gmail.com.

Shinji Itamura (S)

Department of Child Neurology, Seirei-Hamamatsu General Hospital, 2-12-12 Sumiyoshi, Hamamatsu, 430-8558, Japan.

Shimpei Baba (S)

Department of Child Neurology, Seirei-Hamamatsu General Hospital, 2-12-12 Sumiyoshi, Hamamatsu, 430-8558, Japan.

Mitsuyo Nishimura (M)

Laboratory of Neurophysiology, Seirei-Hamamatsu General Hospital, 2-12-12 Sumiyoshi, Hamamatsu, 430-8558, Japan.

Yoichiro Homma (Y)

Department of General Internal Medicine, Seirei-Hamamatsu General Hospital, 2-12-12 Sumiyoshi, Hamamatsu, 430-8558, Japan.

Yoshihiro Maegaki (Y)

Division of Child Neurology, Institute of Neurological Sciences, Faculty of Medicine, Tottori University, 86 Nishimachi, Yonago, 683-8503, Japan.

Hideo Enoki (H)

Department of Child Neurology, Seirei-Hamamatsu General Hospital, 2-12-12 Sumiyoshi, Hamamatsu, 430-8558, Japan.

Ayataka Fujimoto (A)

Comprehensive Epilepsy Center, Seirei-Hamamatsu General Hospital, 2-12-12 Sumiyoshi, Hamamatsu, 430-8558, Japan.

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