Neuroinflammation trajectories precede cognitive impairment after experimental meningitis-evidence from an in vivo PET study.


Journal

Journal of neuroinflammation
ISSN: 1742-2094
Titre abrégé: J Neuroinflammation
Pays: England
ID NLM: 101222974

Informations de publication

Date de publication:
04 Jan 2020
Historique:
received: 04 11 2019
accepted: 24 12 2019
entrez: 6 1 2020
pubmed: 7 1 2020
medline: 11 11 2020
Statut: epublish

Résumé

Bacterial meningitis is a devastating central nervous system (CNS) infection with acute and long-term neurological consequences, including cognitive impairment. The aim of this study was to understand the association between activated microglia-induced neuroinflammation and post-meningitis cognitive impairment. Meningitis was induced in male Wistar rats by injecting Streptococcus pneumoniae into the brain through the cisterna magna, and rats were then treated with ceftriaxone. Twenty-four hours and 10 days after meningitis induction, rats were imaged with positron emission tomography (PET) using [ Both 24-h (acute) and 10-day (long-term) groups of rats demonstrated increased [ TSPO-PET could potentially be used as an imaging biomarker for microglial activation and long-term cognitive impairment post-meningitis. Additionally, this study opens a new avenue for the potential use of TSPO ligands after infection-induced neurological sequelae.

Sections du résumé

BACKGROUND BACKGROUND
Bacterial meningitis is a devastating central nervous system (CNS) infection with acute and long-term neurological consequences, including cognitive impairment. The aim of this study was to understand the association between activated microglia-induced neuroinflammation and post-meningitis cognitive impairment.
METHOD METHODS
Meningitis was induced in male Wistar rats by injecting Streptococcus pneumoniae into the brain through the cisterna magna, and rats were then treated with ceftriaxone. Twenty-four hours and 10 days after meningitis induction, rats were imaged with positron emission tomography (PET) using [
RESULTS RESULTS
Both 24-h (acute) and 10-day (long-term) groups of rats demonstrated increased [
CONCLUSIONS CONCLUSIONS
TSPO-PET could potentially be used as an imaging biomarker for microglial activation and long-term cognitive impairment post-meningitis. Additionally, this study opens a new avenue for the potential use of TSPO ligands after infection-induced neurological sequelae.

Identifiants

pubmed: 31901235
doi: 10.1186/s12974-019-1692-0
pii: 10.1186/s12974-019-1692-0
pmc: PMC6942362
doi:

Substances chimiques

Inflammation Mediators 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

5

Subventions

Organisme : Alzheimer's Association
ID : AARGDNTF-19-619645
Pays : United States

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Auteurs

Vijayasree V Giridharan (VV)

Faillace Department of Psychiatry and Behavioral Sciences, McGovern Medical School, The University of Texas Health Science Center at Houston (UTHealth), Houston, TX, USA.

Allan Collodel (A)

Experimental Physiopathology Laboratory, Graduate Program in Health Sciences, Graduate Program in Health Sciences, Health Sciences Unit, University of Southern Santa Catarina (UNESC), Criciúma, SC, Brazil.

Jaqueline S Generoso (JS)

Experimental Physiopathology Laboratory, Graduate Program in Health Sciences, Graduate Program in Health Sciences, Health Sciences Unit, University of Southern Santa Catarina (UNESC), Criciúma, SC, Brazil.

Giselli Scaini (G)

Faillace Department of Psychiatry and Behavioral Sciences, McGovern Medical School, The University of Texas Health Science Center at Houston (UTHealth), Houston, TX, USA.

Rico Wassather (R)

Micro Analysis Group, Keyence Corporation of America, Austin, TX, USA.

Sudhakar Selvaraj (S)

Faillace Department of Psychiatry and Behavioral Sciences, McGovern Medical School, The University of Texas Health Science Center at Houston (UTHealth), Houston, TX, USA.

Rodrigo Hasbun (R)

Division of Infectious Disease, Department of Medicine, McGovern Medical School, UTHealth, Houston, TX, USA.

Felipe Dal-Pizzol (F)

Experimental Physiopathology Laboratory, Graduate Program in Health Sciences, Graduate Program in Health Sciences, Health Sciences Unit, University of Southern Santa Catarina (UNESC), Criciúma, SC, Brazil.

Fabricia Petronilho (F)

Laboratory of Neurobiology of Inflammatory and Metabolic Processes, Postgraduate Program in Health Sciences, University of South Santa Catarina (UNISUL), Tubarao, SC, Brazil.

Tatiana Barichello (T)

Faillace Department of Psychiatry and Behavioral Sciences, McGovern Medical School, The University of Texas Health Science Center at Houston (UTHealth), Houston, TX, USA. Tatiana.Barichello@uth.tmc.edu.
Experimental Physiopathology Laboratory, Graduate Program in Health Sciences, Graduate Program in Health Sciences, Health Sciences Unit, University of Southern Santa Catarina (UNESC), Criciúma, SC, Brazil. Tatiana.Barichello@uth.tmc.edu.

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Classifications MeSH