Rosuvastatin protects isolated hearts against ischemia-reperfusion injury: role of Akt-GSK-3β, metabolic environment, and mitochondrial permeability transition pore.


Journal

Journal of physiology and biochemistry
ISSN: 1877-8755
Titre abrégé: J Physiol Biochem
Pays: Spain
ID NLM: 9812509

Informations de publication

Date de publication:
Feb 2020
Historique:
received: 10 05 2019
accepted: 28 11 2019
pubmed: 10 1 2020
medline: 9 10 2020
entrez: 10 1 2020
Statut: ppublish

Résumé

The cardioprotective activity of rosuvastatin (R) is yet to be known. The objective of this study was to research whether R perfusion before global ischemia can mitigate myocardial ischemia-reperfusion damage, considering the metabolic condition in which these effects occur, and to contemplate potential mitochondrial benefits. Protein kinase B (Akt)/glycogen synthase kinase-3β (GSK-3β) and mitochondrial permeability transition pore (MPTP) are key elements in myocardial injury produced by ischemia-reperfusion. Isolated rat hearts were subjected to 25-min ischemia and 1-h reperfusion in the presence or absence of R, with or without Wortmannin (W), a phosphatidylinositol 3-kinase (PI3K)/Akt inhibitor. Akt and GSK-3β were measured by Western blot analysis; lactate, glycogen, and G6PDH were determined; and Ca

Identifiants

pubmed: 31916218
doi: 10.1007/s13105-019-00718-z
pii: 10.1007/s13105-019-00718-z
doi:

Substances chimiques

Cardiotonic Agents 0
Mitochondrial Membrane Transport Proteins 0
Mitochondrial Permeability Transition Pore 0
Rosuvastatin Calcium 83MVU38M7Q
Glycogen Synthase Kinase 3 beta EC 2.7.11.1
Gsk3b protein, rat EC 2.7.11.1
Proto-Oncogene Proteins c-akt EC 2.7.11.1

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

85-98

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Auteurs

Debora E Vélez (DE)

Physiology Unit. Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Junín 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina. velez.debora@hotmail.com.
IQUIMEFA-CONICET, Junin 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina. velez.debora@hotmail.com.

Victoria E Mestre-Cordero (VE)

Physiology Unit. Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Junín 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.
IQUIMEFA-CONICET, Junin 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.

Romina Hermann (R)

Physiology Unit. Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Junín 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.
IQUIMEFA-CONICET, Junin 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.

Juliana Perego (J)

Physiology Unit. Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Junín 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.

Sofia Harriet (S)

Physiology Unit. Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Junín 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.

María de Las Mercedes Fernandez-Pazos (MLM)

Physiology Unit. Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Junín 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.

Julieta Mourglia (J)

Physiology Unit. Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Junín 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.

M Gabriela Marina-Prendes (MG)

Physiology Unit. Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Junín 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.
IQUIMEFA-CONICET, Junin 956, 7th floor, C1113AAD, Ciudad de Buenos Aires, Argentina.

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Classifications MeSH