Consequences of HLA-associated mutations in HIV-1 subtype C Nef on HLA-I downregulation ability.


Journal

Journal of medical virology
ISSN: 1096-9071
Titre abrégé: J Med Virol
Pays: United States
ID NLM: 7705876

Informations de publication

Date de publication:
08 2020
Historique:
received: 08 10 2019
accepted: 07 01 2020
pubmed: 17 1 2020
medline: 13 7 2021
entrez: 17 1 2020
Statut: ppublish

Résumé

Identification of CD8+ T lymphocyte (CTL) escape mutations that compromise the pathogenic functions of the Nef protein may be relevant for an HIV-1 attenuation-based vaccine. Previously, HLA-associated mutations 102H, 105R, 108D, and 199Y were individually statistically associated with decreased Nef-mediated HLA-I downregulation ability in a cohort of 298 HIV-1 subtype C infected individuals. In the present study, these mutations were introduced by site-directed mutagenesis into different patient-derived Nef sequence backgrounds of high similarity to the consensus C Nef sequence, and their ability to downregulate HLA-I was measured by flow cytometry in a CEM-derived T cell line. A substantial negative effect of 199Y on HLA-I downregulation and Nef expression was observed, while 102H and 105R displayed negative effects on HLA-I downregulation ability and Nef expression to a lesser extent. The total magnitude of CTL responses in individuals harboring the 199Y mutation was lower than those without the mutation, although this was not statistically significant. Overall, a modest positive relationship between Nef-mediated HLA-I downregulation ability and total magnitude of CTL responses was observed, suggesting that there is a higher requirement for HLA-I downregulation with increased CTL pressure. These results highlight a region of Nef that could be targeted by vaccine-induced CTL to reduce HLA-I downregulation and maximize CTL efficacy.

Identifiants

pubmed: 31944317
doi: 10.1002/jmv.25676
doi:

Substances chimiques

nef Gene Products, Human Immunodeficiency Virus 0
nef protein, Human immunodeficiency virus 1 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1182-1190

Subventions

Organisme : Howard Hughes Medical Institute
Pays : United States

Informations de copyright

© 2020 Wiley Periodicals, Inc.

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Auteurs

Jaclyn K Mann (JK)

HIV Pathogenesis Programme, University of KwaZulu-Natal, Durban, South Africa.

Erasha Rajkoomar (E)

HIV Pathogenesis Programme, University of KwaZulu-Natal, Durban, South Africa.

Steven W Jin (SW)

Faculty of Health Sciences, Simon Fraser University, Burnaby, Burnaby, BC, Canada.

Qiniso Mkhize (Q)

HIV Pathogenesis Programme, University of KwaZulu-Natal, Durban, South Africa.

Omolara Baiyegunhi (O)

Africa Health Research Institute, Durban, South Africa.

Pholisiwe Mbona (P)

HIV Pathogenesis Programme, University of KwaZulu-Natal, Durban, South Africa.

Mark A Brockman (MA)

Faculty of Health Sciences, Simon Fraser University, Burnaby, Burnaby, BC, Canada.
Molecular Biology and Biochemistry, Simon Fraser University, Burnaby, BC, Canada.
British Columbia Centre for Excellence in HIV/AIDS, Vancouver, BC, Canada.

Thumbi Ndung'u (T)

HIV Pathogenesis Programme, University of KwaZulu-Natal, Durban, South Africa.
Africa Health Research Institute, Durban, South Africa.
Ragon Institute of MGH, MIT and Harvard University, Cambridge, Maryland.
Max Planck Institute for Infection Biology, Berlin, Germany.
Division of Infection and Immunity, University College London, London, United Kingdom.

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