Association of Local Epicardial Adipose Tissue Depots and Left Ventricular Diastolic Performance in Patients With Preserved Left Ventricular Ejection Fraction.


Journal

Circulation journal : official journal of the Japanese Circulation Society
ISSN: 1347-4820
Titre abrégé: Circ J
Pays: Japan
ID NLM: 101137683

Informations de publication

Date de publication:
24 01 2020
Historique:
pubmed: 21 1 2020
medline: 21 10 2020
entrez: 21 1 2020
Statut: ppublish

Résumé

Although full-volume quantification of epicardial adipose tissue (EAT) is a predictor of LV diastolic dysfunction (LVDD), how localized EAT depots are linked to LVDD remains unclear. We evaluated the effect of local EAT depots on LV diastolic function parameters in patients with preserved LV ejection fraction (LVEF).Methods and Results:From 423 consecutive patients who underwent cardiac CT angiography, we recruited 252 with sinus rhythm and normal LVEF. The EAT volume index (EATV/body surface area) and the localized EAT thickness around the right coronary artery (EAT Localized EAT depots are linked to altered mitral annular motion. Further study is warranted to clarify whether localized EAT depots are functionally linked to the clinical manifestations of LVDD.

Sections du résumé

BACKGROUND
Although full-volume quantification of epicardial adipose tissue (EAT) is a predictor of LV diastolic dysfunction (LVDD), how localized EAT depots are linked to LVDD remains unclear. We evaluated the effect of local EAT depots on LV diastolic function parameters in patients with preserved LV ejection fraction (LVEF).Methods and Results:From 423 consecutive patients who underwent cardiac CT angiography, we recruited 252 with sinus rhythm and normal LVEF. The EAT volume index (EATV/body surface area) and the localized EAT thickness around the right coronary artery (EAT
CONCLUSIONS
Localized EAT depots are linked to altered mitral annular motion. Further study is warranted to clarify whether localized EAT depots are functionally linked to the clinical manifestations of LVDD.

Identifiants

pubmed: 31956209
doi: 10.1253/circj.CJ-19-0793
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

203-216

Commentaires et corrections

Type : CommentIn

Auteurs

Gulinu Maimaituxun (G)

Department of Diabetes, Endocrinology and Metabolism, School of Medicine, Fukushima Medical University.

Hirotsugu Yamada (H)

Department of Community Medicine for Cardiology, Tokushima University Graduate School of Biomedical Sciences.

Daiju Fukuda (D)

Department of Cardiovascular Medicine, Tokushima University Graduate School of Biomedical Sciences.
Department of Cardio-Diabetes Medicine, Tokushima University Graduate School of Biomedical Sciences.

Shusuke Yagi (S)

Department of Cardiovascular Medicine, Tokushima University Graduate School of Biomedical Sciences.

Kenya Kusunose (K)

Department of Cardiovascular Medicine, Tokushima University Graduate School of Biomedical Sciences.

Yukina Hirata (Y)

Department of Cardiovascular Medicine, Tokushima University Graduate School of Biomedical Sciences.

Susumu Nishio (S)

Department of Cardiovascular Medicine, Tokushima University Graduate School of Biomedical Sciences.

Takeshi Soeki (T)

Department of Cardiovascular Medicine, Tokushima University Graduate School of Biomedical Sciences.

Hiroaki Masuzaki (H)

Division of Endocrinology, Diabetes and Metabolism, Hematology, Rheumatology (Second Department of Internal Medicine), Graduate School of Medicine, University of the Ryukyus.

Masataka Sata (M)

Department of Cardiovascular Medicine, Tokushima University Graduate School of Biomedical Sciences.

Michio Shimabukuro (M)

Department of Diabetes, Endocrinology and Metabolism, School of Medicine, Fukushima Medical University.
Department of Cardio-Diabetes Medicine, Tokushima University Graduate School of Biomedical Sciences.

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