Phosphosite Analysis of the Cytomegaloviral mRNA Export Factor pUL69 Reveals Serines with Critical Importance for Recruitment of Cellular Proteins Pin1 and UAP56/URH49.


Journal

Journal of virology
ISSN: 1098-5514
Titre abrégé: J Virol
Pays: United States
ID NLM: 0113724

Informations de publication

Date de publication:
31 03 2020
Historique:
received: 21 12 2019
accepted: 13 01 2020
pubmed: 24 1 2020
medline: 29 9 2020
entrez: 24 1 2020
Statut: epublish

Résumé

Human cytomegalovirus (HCMV) encodes the viral mRNA export factor pUL69, which facilitates the cytoplasmic accumulation of mRNA via interaction with the cellular RNA helicase UAP56 or URH49. We reported previously that pUL69 is phosphorylated by cellular CDKs and the viral CDK-like kinase pUL97. Here, we set out to identify phosphorylation sites within pUL69 and to characterize their importance. Mass spectrometry-based phosphosite mapping of pUL69 identified 10 serine/threonine residues as phosphoacceptors. Surprisingly, only a few of these sites localized to the N terminus of pUL69, which could be due to the presence of additional posttranslational modifications, like arginine methylation. As an alternative approach, pUL69 mutants with substitutions of putative phosphosites were analyzed by Phos-tag SDS-PAGE. This demonstrated that serines S46 and S49 serve as targets for phosphorylation by pUL97. Furthermore, we provide evidence that phosphorylation of these serines mediates

Identifiants

pubmed: 31969433
pii: JVI.02151-19
doi: 10.1128/JVI.02151-19
pmc: PMC7108844
pii:
doi:

Substances chimiques

NIMA-Interacting Peptidylprolyl Isomerase 0
RNA, Messenger 0
RNA, Viral 0
Transcription Factors 0
Threonine 2ZD004190S
Serine 452VLY9402
DDX39A protein, human EC 3.6.1.-
DDX39B protein, human EC 3.6.1.-
DEAD-box RNA Helicases EC 3.6.4.13
PIN1 protein, human EC 5.2.1.8

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2020 American Society for Microbiology.

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Auteurs

Marco Thomas (M)

Institute of Clinical and Molecular Virology, Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), Erlangen-Nuremberg, Germany.

Regina Müller (R)

Institute of Clinical and Molecular Virology, Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), Erlangen-Nuremberg, Germany.

Georg Horn (G)

Institute of Clinical and Molecular Virology, Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), Erlangen-Nuremberg, Germany.

Boris Bogdanow (B)

Max Delbrueck Center for Molecular Medicine, Berlin, Germany.

Koshi Imami (K)

Max Delbrueck Center for Molecular Medicine, Berlin, Germany.

Jens Milbradt (J)

Institute of Clinical and Molecular Virology, Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), Erlangen-Nuremberg, Germany.

Mirjam Steingruber (M)

Institute of Clinical and Molecular Virology, Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), Erlangen-Nuremberg, Germany.

Manfred Marschall (M)

Institute of Clinical and Molecular Virology, Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), Erlangen-Nuremberg, Germany.

Eva-Maria Schilling (EM)

Institute for Virology, University Hospital Ulm, Ulm, Germany.

Torgils Fossen (T)

Department of Chemistry, University of Bergen, Bergen, Norway.

Thomas Stamminger (T)

Institute for Virology, University Hospital Ulm, Ulm, Germany thomas.stamminger@uniklinik-Ulm.de.

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Classifications MeSH