Treatment-free remission after first-line dasatinib discontinuation in patients with chronic myeloid leukaemia (first-line DADI trial): a single-arm, multicentre, phase 2 trial.


Journal

The Lancet. Haematology
ISSN: 2352-3026
Titre abrégé: Lancet Haematol
Pays: England
ID NLM: 101643584

Informations de publication

Date de publication:
Mar 2020
Historique:
received: 19 06 2019
revised: 17 10 2019
accepted: 18 10 2019
pubmed: 25 1 2020
medline: 12 3 2020
entrez: 25 1 2020
Statut: ppublish

Résumé

A previous dasatinib discontinuation (DADI) trial showed that 31 (49%) of 63 patients with chronic-phase chronic myeloid leukaemia who were treated with second-line or subsequent dasatinib could discontinue the drug safely. However, the safety and efficacy of discontinuing first-line dasatinib remains unclear. In this trial (the first-line DADI trial) we aimed to assess molecular relapse-free survival at 6 months after discontinuation of dasatinib in patients with chronic myeloid leukaemia who had been treated with first-line dasatinib and had maintained deep molecular response for at least 1 year. The first-line DADI trial was a single-arm, multicentre, phase 2 trial done at 23 hospitals in Japan. Patients with newly diagnosed chronic-phase chronic myeloid leukaemia without hepatosplenomegaly and extramedullary mass, who received at least 24-month dasatinib treatment and had a sustained deep molecular response (defined as BCR-ABL1/ABL1 international scale ≤0·0069% in at least four successive samples spanning a 12 month period) were enrolled. Other eligibility criteria were an age of 15 years or older, an Eastern Cooperative Oncology Group performance status score of 0-2, and no primary organ dysfunction. The primary outcome was molecular relapse-free survival (also known as treatment-free remission) after discontinuation of dasatinib at 6 months and was analysed in all patients who completed the 12-month consolidation phase. Safety was assessed in all patients who received treatment. This study closed early due to accrual and is registered with the UMIN Clinical Trials Registry (UMIN000011099). Between Sept 20, 2013 and July 12, 2016, 68 patients who had a deep molecular response after receiving first-line dasatinib for at least 24 months were enrolled and assigned to the consolidation phase. Nine patients were excluded during the consolidation phase and one patient was excluded after study completion because of meeting exclusion criteria. 58 patients discontinued dasatinib and were assessed. 32 (55%) of 58 patients had treatment-free remission at 6 months after dasatinib discontinuation, and median follow-up was 23·3 months (IQR 11·7-31·0). Treatment-free remission at 6 months was 55·2% (95% CI 43·7-69·6). No non-haematological adverse events worse than grade 2 occurred before dasatinib discontinuation. The most common haematological adverse event was anaemia (14 [21%] of 68 treated patients); three (4%) of 68 treated patients had grade 3 neutropenia and one (1%) had grade 4 lymphopenia. Our findings suggest that dasatinib could be safely discontinued after first-line treatment in patients with chronic myeloid leukaemia who had received at least 36 months of therapy and sustained deep molecular response; however, further confirmation in larger trials is needed. Epidemiological and Clinical Research Information Network.

Sections du résumé

BACKGROUND BACKGROUND
A previous dasatinib discontinuation (DADI) trial showed that 31 (49%) of 63 patients with chronic-phase chronic myeloid leukaemia who were treated with second-line or subsequent dasatinib could discontinue the drug safely. However, the safety and efficacy of discontinuing first-line dasatinib remains unclear. In this trial (the first-line DADI trial) we aimed to assess molecular relapse-free survival at 6 months after discontinuation of dasatinib in patients with chronic myeloid leukaemia who had been treated with first-line dasatinib and had maintained deep molecular response for at least 1 year.
METHODS METHODS
The first-line DADI trial was a single-arm, multicentre, phase 2 trial done at 23 hospitals in Japan. Patients with newly diagnosed chronic-phase chronic myeloid leukaemia without hepatosplenomegaly and extramedullary mass, who received at least 24-month dasatinib treatment and had a sustained deep molecular response (defined as BCR-ABL1/ABL1 international scale ≤0·0069% in at least four successive samples spanning a 12 month period) were enrolled. Other eligibility criteria were an age of 15 years or older, an Eastern Cooperative Oncology Group performance status score of 0-2, and no primary organ dysfunction. The primary outcome was molecular relapse-free survival (also known as treatment-free remission) after discontinuation of dasatinib at 6 months and was analysed in all patients who completed the 12-month consolidation phase. Safety was assessed in all patients who received treatment. This study closed early due to accrual and is registered with the UMIN Clinical Trials Registry (UMIN000011099).
FINDINGS RESULTS
Between Sept 20, 2013 and July 12, 2016, 68 patients who had a deep molecular response after receiving first-line dasatinib for at least 24 months were enrolled and assigned to the consolidation phase. Nine patients were excluded during the consolidation phase and one patient was excluded after study completion because of meeting exclusion criteria. 58 patients discontinued dasatinib and were assessed. 32 (55%) of 58 patients had treatment-free remission at 6 months after dasatinib discontinuation, and median follow-up was 23·3 months (IQR 11·7-31·0). Treatment-free remission at 6 months was 55·2% (95% CI 43·7-69·6). No non-haematological adverse events worse than grade 2 occurred before dasatinib discontinuation. The most common haematological adverse event was anaemia (14 [21%] of 68 treated patients); three (4%) of 68 treated patients had grade 3 neutropenia and one (1%) had grade 4 lymphopenia.
INTERPRETATION CONCLUSIONS
Our findings suggest that dasatinib could be safely discontinued after first-line treatment in patients with chronic myeloid leukaemia who had received at least 36 months of therapy and sustained deep molecular response; however, further confirmation in larger trials is needed.
FUNDING BACKGROUND
Epidemiological and Clinical Research Information Network.

Identifiants

pubmed: 31978329
pii: S2352-3026(19)30235-2
doi: 10.1016/S2352-3026(19)30235-2
pii:
doi:

Substances chimiques

Protein Kinase Inhibitors 0
Dasatinib RBZ1571X5H

Types de publication

Clinical Trial, Phase II Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

e218-e225

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Auteurs

Shinya Kimura (S)

Division of Hematology, Respiratory Medicine and Oncology, Department of Internal Medicine, Faculty of Medicine, Saga University, Saga, Japan. Electronic address: shkimu@cc.saga-u.ac.jp.

Jun Imagawa (J)

Department of Hematology and Oncology, Research Institute for Radiation Biology and Medicine, Hiroshima University, Hiroshima, Japan.

Kazunori Murai (K)

Department of Hematology, Iwate Prefectural Central Hospital, Morioka, Japan.

Masayuki Hino (M)

Department of Hematology, Osaka City University Hospital, Osaka, Japan.

Toshio Kitawaki (T)

Department of Hematology and Oncology, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Masaya Okada (M)

Division of Hematology, Department of Internal Medicine, Hyogo College of Medicine, Hyogo, Japan.

Hideo Tanaka (H)

Department of Hematology, Hiroshima City Asa Hospital, Hiroshima, Japan.

Motohiro Shindo (M)

Division of Gastroenterology and Hematology/Oncology, Department of Medicine, Asahikawa Medical University, Asahikawa, Japan.

Takashi Kumagai (T)

Department of Hematology, Ome Municipal General Hospital, Ome, Japan.

Takayuki Ikezoe (T)

Department of Hematology and Respiratory Medicine, Kochi Medical School, Kochi University, Kochi, Japan; Department of Hematology, Fukushima Medical School, Fukushima, Japan.

Nobuhiko Uoshima (N)

Department of Hematology, Matsushita Memorial Hospital, Osaka, Japan.

Tsutomu Sato (T)

Department of Medical Oncology and Hematology, Sapporo Medical University Hospital, Sapporo, Japan.

Reiko Watanabe (R)

Department of Hematology, Saitama Medical Center, Saitama Medical University, Saitama, Japan.

Shugo Kowata (S)

Department of Hematology, Iwate Prefecture Ofunato Hospital, Iwate, Japan.

Masaya Hayakawa (M)

Department of Hematology, Ogaki Municipal Hospital, Ogaki, Japan.

Takaaki Hosoki (T)

Department of Hematology and Oncology, Asahikawa Kosei Hospital, Asahikawa, Japan.

Kazuhiko Ikeda (K)

Department of Hematology, Fukushima Medical School, Fukushima, Japan.

Tsutomu Kobayashi (T)

Division of Hematology and Oncology, Department of Medicine, Kyoto Prefectural University of Medicine, Kyoto, Japan.

Yasutaka Kakinoki (Y)

Department of Hematology, Asahikawa City Hospital, Asahikawa, Japan.

Tetsuo Nishimoto (T)

Department of Hematology, Ashiya Municipal Hospital, Ashiya, Japan.

Naoki Takezako (N)

Division of Hematology, National Hospital Organization Disaster Medical Center, Tachikawa, Japan.

Hirohiko Shibayama (H)

Department of Hematology and Oncology, Osaka University Graduate School of Medicine, Suita, Japan.

Akifumi Takaori-Kondo (A)

Department of Hematology and Oncology, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Hirohisa Nakamae (H)

Department of Hematology, Osaka City University Hospital, Osaka, Japan.

Atsushi Kawaguchi (A)

Center for Comprehensive Community Medicine, Faculty of Medicine, Saga University, Saga, Japan.

Hiroshi Ureshino (H)

Division of Hematology, Respiratory Medicine and Oncology, Department of Internal Medicine, Faculty of Medicine, Saga University, Saga, Japan.

Junichi Sakamoto (J)

Epidemiological and Clinical Research Information Network, Okazaki, Japan.

Yoji Ishida (Y)

Department of Hematology and Oncology, Iwate Medical University, Morioka, Japan.

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