Altered productions of prostaglandins and prostamides by human amnion in response to infectious and inflammatory stimuli identified by mutliplex mass spectrometry.


Journal

Prostaglandins, leukotrienes, and essential fatty acids
ISSN: 1532-2823
Titre abrégé: Prostaglandins Leukot Essent Fatty Acids
Pays: Scotland
ID NLM: 8802730

Informations de publication

Date de publication:
03 2020
Historique:
received: 14 06 2019
revised: 28 11 2019
accepted: 21 01 2020
pubmed: 6 2 2020
medline: 22 9 2020
entrez: 5 2 2020
Statut: ppublish

Résumé

Prostaglandins are critical for the onset and progression of labor in mammals, and are formed by the metabolism of arachidonic acid. The products of arachidonic acid, 2-arachidonoylglycerol (2-AG), and anandamide (AEA) have a similar lipid back bone but differing polar head groups, meaning that identification of these products by immunoassay can be difficult. In the current study, we present the use of mass spectrometry as multiplex method of identifying the specific end products of arachidonic and anandamide metabolism by human derived amnion explants treated with either an infectious agent (LPS) or inflammatory mediator (IL-1β or TNF-α). Human amnion tissue explants treated with LPS, IL-1β, or TNF-α increased production of prostaglandin E The findings of the current study are in keeping with the literature which describes amnion tissues as predominantly producing PGE

Identifiants

pubmed: 32014738
pii: S0952-3278(19)30123-1
doi: 10.1016/j.plefa.2020.102059
pii:
doi:

Substances chimiques

Arachidonic Acids 0
Cytokines 0
Endocannabinoids 0
Interleukin-1beta 0
Lipopolysaccharides 0
Polyunsaturated Alkamides 0
Tumor Necrosis Factor-alpha 0
15-keto-13,14-dihydroprostaglandin F2alpha 27376-76-7
Arachidonic Acid 27YG812J1I
Dinoprost B7IN85G1HY
Dinoprostone K7Q1JQR04M
anandamide UR5G69TJKH

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

102059

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest None.

Auteurs

Hassendrini N Peiris (HN)

School of Biomedical Science, Institute of Health and Biomedical Innovation - Centre for Children's Health Research, Faculty of Health, Queensland University of Technology, 62 Graham Street, South Brisbane, QLD 4101, Australia. Electronic address: hassendrini.peiris@qut.edu.au.

Kanchan Vaswani (K)

School of Biomedical Science, Institute of Health and Biomedical Innovation - Centre for Children's Health Research, Faculty of Health, Queensland University of Technology, 62 Graham Street, South Brisbane, QLD 4101, Australia.

Olivia Holland (O)

School of Biomedical Science, Institute of Health and Biomedical Innovation - Centre for Children's Health Research, Faculty of Health, Queensland University of Technology, 62 Graham Street, South Brisbane, QLD 4101, Australia.

Yong Qin Koh (YQ)

School of Biomedical Science, Institute of Health and Biomedical Innovation - Centre for Children's Health Research, Faculty of Health, Queensland University of Technology, 62 Graham Street, South Brisbane, QLD 4101, Australia.

Fatema B Almughlliq (FB)

University of Queensland Centre for Clinical Research, Building 71/918, Royal Brisbane and Women's Hospital, Herston, QLD 4029, Australia.

Sarah Reed (S)

University of Queensland Centre for Clinical Research, Building 71/918, Royal Brisbane and Women's Hospital, Herston, QLD 4029, Australia.

Murray D Mitchell (MD)

School of Biomedical Science, Institute of Health and Biomedical Innovation - Centre for Children's Health Research, Faculty of Health, Queensland University of Technology, 62 Graham Street, South Brisbane, QLD 4101, Australia. Electronic address: murray.mitchell@qut.edu.au.

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Classifications MeSH