Correction of autophagy impairment inhibits pathology in the NOD.H-2h4 mouse model of primary Sjögren's syndrome.


Journal

Journal of autoimmunity
ISSN: 1095-9157
Titre abrégé: J Autoimmun
Pays: England
ID NLM: 8812164

Informations de publication

Date de publication:
03 2020
Historique:
received: 24 12 2019
revised: 20 01 2020
accepted: 20 01 2020
pubmed: 8 2 2020
medline: 6 7 2021
entrez: 8 2 2020
Statut: ppublish

Résumé

Dysregulation of autophagy has been implicated in the development of various disease indications including autoimmune diseases. Here we identified hitherto unsuspected molecular alterations of autophagy occurring at an early stage of the macroautophagy pathway in the salivary glands and spleen of NOD.H-2h4 mice that develop a primary Sjögren's-like syndrome. In this study we investigated the capacity of phosphopeptide P140 to correct immune alteration in NOD.H-2h4 mice and the effect on neogenesis of tertiary lymphoid structures in salivary glands, which is hallmark characteristic of SS. Phosphopeptide P140 known to lower excessive autophagy processes, rescued sick NOD.H-2h4 mice from some autophagy defects and significantly reduced formation of tertiary lymphoid structures in salivary glands. Mechanistically, the frequency of activated CD44

Identifiants

pubmed: 32029330
pii: S0896-8411(20)30029-9
doi: 10.1016/j.jaut.2020.102418
pii:
doi:

Substances chimiques

Autoantibodies 0
Immunoglobulin G 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

102418

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest EV and AQ are salaried employees and own stocks of AstraZeneka; SM is a co-inventor on patents on P140 peptide. The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

Auteurs

Elisaveta Voynova (E)

Respiratory, Inflammation and Autoimmunity Group, AstraZeneca, Gaithersburg, MD, 2878, USA.

Floryna Lefebvre (F)

CNRS UMR7242, Biotechnology and Cell Signaling, Ecole Supérieure de Biotechnologie de Strasbourg, Strasbourg University/Laboratory of Excellence Medalis, France.

Ariful Qadri (A)

Respiratory, Inflammation and Autoimmunity Group, AstraZeneca, Gaithersburg, MD, 2878, USA.

Sylviane Muller (S)

CNRS UMR7242, Biotechnology and Cell Signaling, Ecole Supérieure de Biotechnologie de Strasbourg, Strasbourg University/Laboratory of Excellence Medalis, France; Fédération Hospitalo-Universitaire (FHU) OMICARE, Fédération de Médecine Translationnelle de Strasbourg (FMTS), Strasbourg University, Strasbourg, France; University of Strasbourg Institute for Advanced Study, Strasbourg, France. Electronic address: sylviane.muller@unistra.fr.

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Classifications MeSH