Efficacy and safety of TNF blockers and of ustekinumab in palmoplantar pustulosis and in acrodermatitis continua of Hallopeau.


Journal

Journal of the European Academy of Dermatology and Venereology : JEADV
ISSN: 1468-3083
Titre abrégé: J Eur Acad Dermatol Venereol
Pays: England
ID NLM: 9216037

Informations de publication

Date de publication:
Oct 2020
Historique:
received: 28 10 2019
accepted: 21 01 2020
pubmed: 8 2 2020
medline: 15 5 2021
entrez: 8 2 2020
Statut: ppublish

Résumé

Palmoplantar pustulosis (PPP) and acrodermatitis continua of Hallopeau (ACH) are rare variants of psoriasis. Knowledge of the efficacy of biologics is scarce. To evaluate the real-life efficacy of tumour necrosis factor blockers and ustekinumab in PPP and in ACH. A multicentre retrospective descriptive study was conducted in 19 dermatology departments, including all patients with PPP or ACH seen from 2014 to 2016 who received one of the studied biologics. The data were collected by a standardized document. Factors associated with complete clearance (CC) were analysed by multivariate analysis, estimating odds ratios (ORs) and 95% confidence intervals (CIs). Among 92 patients included, 50 received adalimumab, 44 ustekinumab, 36 etanercept and 31 infliximab. Improvement and CC were observed in 83.9% and 20.0% patients receiving infliximab, 75.0% and 38.6% ustekinumab, 57.1% and 20.0% etanercept and 60.4% and 29.2% adalimumab. We found no significant difference in CC rates or duration of treatment among the biological treatments (P = 0.18 and P = 0.10, respectively). On multivariate analysis, CC with etanercept was associated with the ACH form and not smoking [OR = 9.5 (95% CI 1.1-82.7), P = 0.04 and 0.1 (0.01-0.9), P = 0.04]; with ustekinumab, male sex and absence of obesity [6.0 (1.3-28.6), P = 0.02 and 4.7 (1.0-22.7), P = 0.05]; with adalimumab, the ACH form [11.9 (2.7-52.3), P = 0.001]; and with infliximab, obesity [5.6 (1.1-29.4), P = 0.04]. We found no difference in efficacy between TNF blockers and ustekinumab and among the three different TNF blockers in real life for PPP or ACH, which reveals the heterogeneity of clinical response to biologics in pustular psoriasis as compared with plaque psoriasis.

Sections du résumé

BACKGROUND BACKGROUND
Palmoplantar pustulosis (PPP) and acrodermatitis continua of Hallopeau (ACH) are rare variants of psoriasis. Knowledge of the efficacy of biologics is scarce.
OBJECTIVES OBJECTIVE
To evaluate the real-life efficacy of tumour necrosis factor blockers and ustekinumab in PPP and in ACH.
METHODS METHODS
A multicentre retrospective descriptive study was conducted in 19 dermatology departments, including all patients with PPP or ACH seen from 2014 to 2016 who received one of the studied biologics. The data were collected by a standardized document. Factors associated with complete clearance (CC) were analysed by multivariate analysis, estimating odds ratios (ORs) and 95% confidence intervals (CIs).
RESULTS RESULTS
Among 92 patients included, 50 received adalimumab, 44 ustekinumab, 36 etanercept and 31 infliximab. Improvement and CC were observed in 83.9% and 20.0% patients receiving infliximab, 75.0% and 38.6% ustekinumab, 57.1% and 20.0% etanercept and 60.4% and 29.2% adalimumab. We found no significant difference in CC rates or duration of treatment among the biological treatments (P = 0.18 and P = 0.10, respectively). On multivariate analysis, CC with etanercept was associated with the ACH form and not smoking [OR = 9.5 (95% CI 1.1-82.7), P = 0.04 and 0.1 (0.01-0.9), P = 0.04]; with ustekinumab, male sex and absence of obesity [6.0 (1.3-28.6), P = 0.02 and 4.7 (1.0-22.7), P = 0.05]; with adalimumab, the ACH form [11.9 (2.7-52.3), P = 0.001]; and with infliximab, obesity [5.6 (1.1-29.4), P = 0.04].
CONCLUSIONS CONCLUSIONS
We found no difference in efficacy between TNF blockers and ustekinumab and among the three different TNF blockers in real life for PPP or ACH, which reveals the heterogeneity of clinical response to biologics in pustular psoriasis as compared with plaque psoriasis.

Identifiants

pubmed: 32030802
doi: 10.1111/jdv.16265
doi:

Substances chimiques

Tumor Necrosis Factor Inhibitors 0
Infliximab B72HH48FLU
Ustekinumab FU77B4U5Z0
Adalimumab FYS6T7F842
Etanercept OP401G7OJC

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

2330-2338

Informations de copyright

© 2020 European Academy of Dermatology and Venereology.

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Auteurs

B Husson (B)

Dermatology Department, Hôpital Robert-Debré, Reims, France.

C Barbe (C)

Clinical Research Unit, Hôpital Robert-Debré, Reims, France.

S Hegazy (S)

Dermatology Department, Hôpital Larrey, Toulouse, France.

J Seneschal (J)

Dermatology Department, National Reference Center for Rare Skin Diseases, Hôpital Saint-André, Bordeaux, France.

F Aubin (F)

Dermatology Department, Centre Hospitalo-Universitaire (CHU), Besançon, France.

E Mahé (E)

Dermatology Department, Centre Hospitalier (CH), Argenteuil, France.

D Jullien (D)

Clinical Immunology Department, CH Lyon-Sud, Lyon, France.

E Sbidian (E)

Dermatology Department, Hôpital Henri-Mondor, Créteil, France.

M D'Incan (M)

Dermatology Department, CHU Estaing, Clermont-Ferrand, France.

C Conrad (C)

Dermatology Department, Lausanne University Hospital, CHUV, Lausanne, Switzerland.

E Brenaut (E)

Dermatology Department, CHU, Brest, France.

C Girard (C)

Dermatology Department, CHU Lapeyronie, Montpellier, France.

M A Richard (MA)

Dermatology Department, CEReSS-EA 3279, Research Center in Health Services and Quality of Life Aix Marseille University, Universitary Hospital Timone, Assistance Publique Hôpitaux de Marseille, Marseille, France.

H Bachelez (H)

Université de Paris, Paris, France.
Dermatology Department, Assistance Publique-Hôpitaux de Paris, Hôpital Saint-Louis, Paris, France.
Laboratory of Genetics of Skin Diseases, INSERM UMR1163, Institut Imagine, Necker Hospital, Paris, France.

M Viguier (M)

Dermatology Department, Hôpital Robert-Debré, Reims, France.

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