Beta-sitosterol attenuates insulin resistance in adipose tissue via IRS-1/Akt mediated insulin signaling in high fat diet and sucrose induced type-2 diabetic rats.


Journal

European journal of pharmacology
ISSN: 1879-0712
Titre abrégé: Eur J Pharmacol
Pays: Netherlands
ID NLM: 1254354

Informations de publication

Date de publication:
15 Apr 2020
Historique:
received: 21 07 2019
revised: 05 02 2020
accepted: 07 02 2020
pubmed: 12 2 2020
medline: 28 11 2020
entrez: 12 2 2020
Statut: ppublish

Résumé

In our previous study, we have shown that β-sitosterol (SIT) enhances glycemic control by increasing the activation of insulin receptor (IR) and glucose transporter 4 (GLUT4) proteins in adipose tissue. However, the possible role of SIT on the regulation of post-receptor insulin signal transduction is not known. Hence, the study was aimed to assess the effects of SIT on IRS-1/Akt mediated insulin signaling molecules in high-fat diet and sucrose induced type-2 diabetic rats. An oral effective dose of SIT (20 mg/kg b.wt) was given for 30 days to high fat-fed type-2 diabetic rats to find out whether SIT regulates IRS-1/Akt pathway of insulin signaling. The results showed that SIT attenuated the insulin receptor substrate-1 serine phosphorylation (p-IRS-1

Identifiants

pubmed: 32045603
pii: S0014-2999(20)30096-0
doi: 10.1016/j.ejphar.2020.173004
pii:
doi:

Substances chimiques

Insulin 0
Insulin Receptor Substrate Proteins 0
Irs1 protein, rat 0
Sitosterols 0
beta-Arrestin 2 0
Sucrose 57-50-1
gamma-sitosterol 5LI01C78DD
src-Family Kinases EC 2.7.10.2
Proto-Oncogene Proteins c-akt EC 2.7.11.1

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

173004

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare no conflict of interests.

Auteurs

Shyamaladevi Babu (S)

Department of Biochemistry, Saveetha Dental College and Hospitals, Saveetha Institute of Medical and Technical Sciences, Saveetha University, Chennai, Tamil Nadu, India.

Madhan Krishnan (M)

Department of Biochemistry, Saveetha Dental College and Hospitals, Saveetha Institute of Medical and Technical Sciences, Saveetha University, Chennai, Tamil Nadu, India.

Ponnulakshmi Rajagopal (P)

Central Research Laboratory, Meenakshi Academy of Higher Education and Research (Deemed to be University), Chennai, Tamil Nadu, India.

Vijayalakshmi Periyasamy (V)

Department of Biotechnology and Bioinformatics, Holy Cross College, Trichy, Tamilnadu, India.

Vishnupriya Veeraraghavan (V)

Department of Biochemistry, Saveetha Dental College and Hospitals, Saveetha Institute of Medical and Technical Sciences, Saveetha University, Chennai, Tamil Nadu, India.

Ramajayam Govindan (R)

Multi Disciplinary Research Unit, Madurai Medical College, TamilNadu, India.

Selvaraj Jayaraman (S)

Department of Biochemistry, Saveetha Dental College and Hospitals, Saveetha Institute of Medical and Technical Sciences, Saveetha University, Chennai, Tamil Nadu, India. Electronic address: jselvaendo@gmail.com.

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Classifications MeSH