Potential Benefits of Bevacizumab Combined With Platinum-Based Chemotherapy in Advanced Non-Small-Cell Lung Cancer Patients With EGFR Mutation.


Journal

Clinical lung cancer
ISSN: 1938-0690
Titre abrégé: Clin Lung Cancer
Pays: United States
ID NLM: 100893225

Informations de publication

Date de publication:
05 2020
Historique:
received: 31 07 2019
revised: 26 12 2019
accepted: 20 01 2020
pubmed: 24 2 2020
medline: 6 7 2021
entrez: 24 2 2020
Statut: ppublish

Résumé

Oncogenic EGFR signaling has been shown to upregulate vascular endothelial growth factor A (VEGFA) expression involved in tumor angiogenesis. However, the clinical benefits of bevacizumab plus cytotoxic chemotherapy for EGFR mutation-positive patients remain unclear. This study aimed to investigate VEGFA messenger RNA expression in patients with EGFR mutation, and to further compare the efficacy of bevacizumab combined with platinum-based chemotherapy between EGFR-mutant and wild-type patients. Gene expression of various proangiogenic factors was analyzed in nonsquamous, non-small-cell lung cancer (NSCLC) patients using The Cancer Genome Atlas dataset. Additionally, clinical data of patients receiving carboplatin and pemetrexed (CPem; n = 104) or bevacizumab plus CPem (BevCPem; n = 55) at Nagoya University hospital were retrospectively assessed for progression-free survival and best overall response rate (ORR). Among various proangiogenic factors, only VEGFA expression was significantly higher in patients with advanced nonsquamous NSCLC with EGFR mutation compared to wild-type patients (P = .0476). Progression-free survival in the BevCPem group was significantly longer in patients with EGFR mutation than in wild-type patients (10.5 vs. 6.6 months; Wilcoxon P = .0278), while the difference in the CPem group was not significant (6.6 vs. 4.5 months; Wilcoxon P = .1822). The ORRs in the BevCPem group were 54.5% and 36.4% for EGFR-mutant and wild-type patients, respectively, and the ORRs in the CPem group were 35.5% and 28.8 % in EGFR-mutant and wild-type patients, respectively. VEGFA messenger RNA expression was significantly increased in advanced nonsquamous NSCLC harboring EGFR mutation, and BevCPem provided better clinical benefits to patients with EGFR mutation than wild-type carriers.

Sections du résumé

BACKGROUND
Oncogenic EGFR signaling has been shown to upregulate vascular endothelial growth factor A (VEGFA) expression involved in tumor angiogenesis. However, the clinical benefits of bevacizumab plus cytotoxic chemotherapy for EGFR mutation-positive patients remain unclear. This study aimed to investigate VEGFA messenger RNA expression in patients with EGFR mutation, and to further compare the efficacy of bevacizumab combined with platinum-based chemotherapy between EGFR-mutant and wild-type patients.
PATIENTS AND METHODS
Gene expression of various proangiogenic factors was analyzed in nonsquamous, non-small-cell lung cancer (NSCLC) patients using The Cancer Genome Atlas dataset. Additionally, clinical data of patients receiving carboplatin and pemetrexed (CPem; n = 104) or bevacizumab plus CPem (BevCPem; n = 55) at Nagoya University hospital were retrospectively assessed for progression-free survival and best overall response rate (ORR).
RESULTS
Among various proangiogenic factors, only VEGFA expression was significantly higher in patients with advanced nonsquamous NSCLC with EGFR mutation compared to wild-type patients (P = .0476). Progression-free survival in the BevCPem group was significantly longer in patients with EGFR mutation than in wild-type patients (10.5 vs. 6.6 months; Wilcoxon P = .0278), while the difference in the CPem group was not significant (6.6 vs. 4.5 months; Wilcoxon P = .1822). The ORRs in the BevCPem group were 54.5% and 36.4% for EGFR-mutant and wild-type patients, respectively, and the ORRs in the CPem group were 35.5% and 28.8 % in EGFR-mutant and wild-type patients, respectively.
CONCLUSION
VEGFA messenger RNA expression was significantly increased in advanced nonsquamous NSCLC harboring EGFR mutation, and BevCPem provided better clinical benefits to patients with EGFR mutation than wild-type carriers.

Identifiants

pubmed: 32088115
pii: S1525-7304(20)30011-5
doi: 10.1016/j.cllc.2020.01.011
pii:
doi:

Substances chimiques

Pemetrexed 04Q9AIZ7NO
Carboplatin BG3F62OND5
EGFR protein, human EC 2.7.10.1
ErbB Receptors EC 2.7.10.1

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

273-280.e4

Informations de copyright

Copyright © 2020 Elsevier Inc. All rights reserved.

Auteurs

Ichidai Tanaka (I)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan. Electronic address: ichidai@med.nagoya-u.ac.jp.

Masahiro Morise (M)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Ayako Miyazawa (A)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Yuta Kodama (Y)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Yutaro Tamiya (Y)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Soei Gen (S)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Akira Matsui (A)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Tetsunari Hase (T)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Naozumi Hashimoto (N)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Mitsuo Sato (M)

Department of Pathophysiological Laboratory Sciences, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Yoshinori Hasegawa (Y)

Department of Respiratory Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.

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