Methylation of the LEP gene promoter in blood at 12 months and BMI at 4 years of age-a population-based cohort study.


Journal

International journal of obesity (2005)
ISSN: 1476-5497
Titre abrégé: Int J Obes (Lond)
Pays: England
ID NLM: 101256108

Informations de publication

Date de publication:
04 2020
Historique:
received: 23 07 2019
accepted: 12 02 2020
revised: 06 02 2020
pubmed: 29 2 2020
medline: 3 8 2021
entrez: 29 2 2020
Statut: ppublish

Résumé

Increasing evidence links epigenetic variation to anthropometric and metabolic measures. Leptin signalling regulates appetite and energy expenditure, and in pregnancy is important for nutrient supply to the foetus. Maternal metabolic health and foetal growth are linked to infant blood leptin gene (LEP) methylation, which has been cross-sectionally associated with adolescent obesity. Despite this, few studies have explored the relationship between infant LEP methylation and childhood anthropometry, or the impact of genetic variation on these relationships. Using a prospective birth cohort, we investigated whether blood LEP promoter methylation at birth and 12 months predicts weight and adiposity at 4-years. Locus-specific methylation data was analysed by partial correlation tests and multivariable linear regression. There was weak evidence of an association of birth LEP methylation with anthropometry measures at 4 years. Methylation at a specific site (cg19594666) at 12 months was inversely associated with 4-year weight (r = -0.11, p = 0.02) and body-mass index (BMI) (r = -0.13, p = 0.007), which persisted following adjustment for weight at birth and at 12 months. Neither association was influenced by genotype. We report the first evidence of an association between LEP methylation in infancy and childhood weight. Replication in additional cohorts is required to determine if this relationship persists.

Identifiants

pubmed: 32107462
doi: 10.1038/s41366-020-0553-3
pii: 10.1038/s41366-020-0553-3
doi:

Substances chimiques

LEP protein, human 0
Leptin 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

842-847

Investigateurs

Peter Vuillermin (P)
Fiona Collier (F)
Anne-Louise Ponsonby (AL)
John Carlin (J)
Katie Allen (K)
Mimi Tang (M)
Richard Saffery (R)
Sarath Ranganathan (S)
David Burgner (D)
Terry Dwyer (T)
Peter Sly (P)

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Auteurs

Toby Mansell (T)

Murdoch Children's Research Institute, Parkville, VIC, Australia.
Department of Paediatrics, University of Melbourne, Parkville, VIC, Australia.

Anne-Louise Ponsonby (AL)

Murdoch Children's Research Institute, Parkville, VIC, Australia.
Department of Paediatrics, University of Melbourne, Parkville, VIC, Australia.
The Florey Institute of Neuroscience and Mental Health, Parkville, VIC, Australia.

Fiona Collier (F)

Murdoch Children's Research Institute, Parkville, VIC, Australia.
School of Medicine, Deakin University, Geelong, VIC, Australia.
Child Health Research Unit, Barwon Health, Geelong, VIC, Australia.

David Burgner (D)

Murdoch Children's Research Institute, Parkville, VIC, Australia.
Department of Paediatrics, University of Melbourne, Parkville, VIC, Australia.
Department of Paediatrics, Monash University, Clayton, VIC, Australia.

Angela Pezic (A)

Murdoch Children's Research Institute, Parkville, VIC, Australia.

Peter Vuillermin (P)

Murdoch Children's Research Institute, Parkville, VIC, Australia.
School of Medicine, Deakin University, Geelong, VIC, Australia.
Child Health Research Unit, Barwon Health, Geelong, VIC, Australia.

Joanne Ryan (J)

Murdoch Children's Research Institute, Parkville, VIC, Australia.
School of Public Health & Preventive Medicine, Monash University, Melbourne, VIC, Australia.

Richard Saffery (R)

Murdoch Children's Research Institute, Parkville, VIC, Australia. richard.saffery@mcri.edu.au.
Department of Paediatrics, University of Melbourne, Parkville, VIC, Australia. richard.saffery@mcri.edu.au.

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