Preclinical Assessment of the Analgesic Pharmacology of NKTR-181 in Rodents.


Journal

Cellular and molecular neurobiology
ISSN: 1573-6830
Titre abrégé: Cell Mol Neurobiol
Pays: United States
ID NLM: 8200709

Informations de publication

Date de publication:
Jul 2021
Historique:
received: 05 11 2019
accepted: 16 02 2020
pubmed: 29 2 2020
medline: 15 12 2021
entrez: 29 2 2020
Statut: ppublish

Résumé

Pharmacological evaluation of the mu-opioid receptor (MOR) agonist properties of NKTR-181 in rodent models. Graded noxious stimulus intensities were used in rats to establish the antinociceptive potency and efficacy of NKTR-181 relative to morphine, fentanyl, and oxycodone. Characteristics of MOR agonist actions, as measured by antinociceptive tolerance and cross-tolerance, as well as opioid-induced hyperalgesia (OIH) and naloxone-precipitated withdrawal in NKTR-181- and morphine-dependent in mice, were compared. NKTR-181 showed dose- and time-related antinociception with similar maximal effects to morphine in the rat and mouse hot-water tail-flick test. No sex or species differences were observed in NKTR-181 or morphine antinociception. Rats treated with NKTR-181 and morphine exhibited decreases in both potency and maximal efficacy as nociceptive stimulus intensity was increased from a water temperature of 50 °C to 54 °C. Evaluation of antinociception at a high stimulus intensity revealed that oxycodone and fentanyl exhibited greater efficacy than either NKTR-181 or morphine. The relative potency difference between NKTR-181 and morphine across all tail-flick studies was determined to be 7.6-fold (90% confidence interval, 2.6, 21.5). The peak antinociceptive effect of NKTR-181 was delayed compared to that of the other opioids and cumulative drug effects were not observed. Repeated treatment with escalating, approximately equi-analgesic doses of NKTR-181 or morphine, produced antinociceptive tolerance and cross-tolerance. Under these pharmacological conditions, OIH and naloxone-precipitated physical dependence were similar for NKTR-181 and morphine. NKTR-181 had a slower onset, but similar efficacy, to morphine in the models studied supporting reduced abuse potential while maintaining analgesic effect in comparison with current opioids.

Identifiants

pubmed: 32107752
doi: 10.1007/s10571-020-00816-3
pii: 10.1007/s10571-020-00816-3
doi:

Substances chimiques

Analgesics, Opioid 0
Morphinans 0
NKTR-181 0
Receptors, Opioid, mu 0
Morphine 76I7G6D29C

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

949-960

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Auteurs

Caroline M Kopruszinski (CM)

Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA.

Juliana Swiokla (J)

Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA.

Yeon Sun Lee (YS)

Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA.

Edita Navratilova (E)

Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA.

Laurie VanderVeen (L)

Nektar Therapeutics, San Francisco, CA, USA.

Miao Yang (M)

Nektar Therapeutics, San Francisco, CA, USA.

Yi Liu (Y)

Nektar Therapeutics, San Francisco, CA, USA.

Takahiro Miyazaki (T)

Nektar Therapeutics, San Francisco, CA, USA.

William K Schmidt (WK)

NorthStar Consulting, Davis, CA, USA.

Jonathan Zalevsky (J)

Nektar Therapeutics, San Francisco, CA, USA.

Frank Porreca (F)

Department of Pharmacology, College of Medicine, University of Arizona, Tucson, AZ, USA. frankp@medadmin.arizona.edu.

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