Salter-Harris type II fractures of the distal tibia: Residual postreduction displacement and outcomes-a STROBE compliant study.
Journal
Medicine
ISSN: 1536-5964
Titre abrégé: Medicine (Baltimore)
Pays: United States
ID NLM: 2985248R
Informations de publication
Date de publication:
Feb 2020
Feb 2020
Historique:
entrez:
3
3
2020
pubmed:
3
3
2020
medline:
17
3
2020
Statut:
ppublish
Résumé
We assessed factors associated with premature physeal closure (PPC) and outcomes after closed reduction of Salter-Harris type II (SH-II) fractures of the distal tibia. We reviewed patients with SH-II fractures of the distal tibia treated at our center from 2010 to 2015 with closed reduction and a non-weightbearing long-leg cast. Patients were categorized by immediate postreduction displacement: minimal, <2 mm; moderate, 2 to 4 mm; or severe, >4 mm. Demographic data, radiographic data, and Lower Extremity Functional Scale (LEFS) scores were recorded.Fifty-nine patients (27 girls, 31 right ankles, 26 concomitant fibula fractures) were included, with a mean (±SD) age at injury of 12.0 ± 2.2 years. Mean maximum fracture displacements were 6.6 ± 6.5 mm initially, 2.7 ± 2.0 mm postreduction, and 0.4 ± 0.7 mm at final follow-up. After reduction, displacement was minimal in 23 patients, moderate in 21, and severe in 15. Fourteen patients developed PPC, with no significant differences between postreduction displacement groups. Patients with high-grade injury mechanisms and/or initial displacement ≥4 mm had 12-fold and 14-fold greater odds, respectively, of PPC. Eighteen patients responded to the LEFS survey (mean 4.0 ± 2.1 years after injury). LEFS scores did not differ significantly between postreduction displacement groups (P = .61).The PPC rate in this series of SH-II distal tibia fractures was 24% and did not differ by postreduction displacement. Initial fracture displacement and high-grade mechanisms of injury were associated with PPC. LEFS scores did not differ significantly by postreduction displacement.Level of Evidence: Level IV, case series.
Identifiants
pubmed: 32118764
doi: 10.1097/MD.0000000000019328
pii: 00005792-202002280-00064
pmc: PMC7478605
doi:
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
e19328Références
Clin J Pain. 2012 Nov-Dec;28(9):766-74
pubmed: 22688602
Int Orthop. 2010 Apr;34(4):547-52
pubmed: 19662414
J Bone Joint Surg Am. 1978 Dec;60(8):1046-50
pubmed: 721852
J Orthop Trauma. 1987;1(1):63-7
pubmed: 3506588
Acta Orthop Belg. 2016 Dec;82(4):814-820
pubmed: 29182123
J Pediatr Orthop. 2006 May-Jun;26(3):322-8
pubmed: 16670543
Acta Orthop. 2018 Feb;89(1):108-112
pubmed: 28925312
Clin Orthop Relat Res. 1978 Oct;(136):230-3
pubmed: 103673
J Pediatr Orthop. 2009 Jun;29(4):356-61
pubmed: 19461377
J Pediatr Orthop. 2013 Jul-Aug;33(5):524-9
pubmed: 23752150
J Pediatr Orthop. 2003 Nov-Dec;23(6):733-9
pubmed: 14581776
J Trauma. 1972 Apr;12(4):275-81
pubmed: 5018408
J Pediatr Orthop. 1983 Jul;3(3):319-25
pubmed: 6409927
Phys Ther. 1999 Apr;79(4):371-83
pubmed: 10201543
Orthopedics. 2012 Dec;35(12):1046-9
pubmed: 23218619