TRAF5 Deficiency Ameliorates the Severity of Dextran Sulfate Sodium Colitis by Decreasing TRAF2 Expression in Nonhematopoietic Cells.
Animals
Cells, Cultured
Colitis
/ chemically induced
Colon
/ metabolism
Cytokines
/ metabolism
Dextran Sulfate
/ pharmacology
Disease Models, Animal
Epithelial Cells
/ metabolism
Fibroblasts
/ metabolism
Inflammation
/ genetics
Intestinal Mucosa
/ metabolism
Mice
Mice, Inbred C57BL
Mice, Knockout
TNF Receptor-Associated Factor 2
/ metabolism
TNF Receptor-Associated Factor 5
/ genetics
Transfection
Journal
ImmunoHorizons
ISSN: 2573-7732
Titre abrégé: Immunohorizons
Pays: United States
ID NLM: 101708159
Informations de publication
Date de publication:
10 03 2020
10 03 2020
Historique:
received:
24
01
2020
accepted:
20
02
2020
entrez:
12
3
2020
pubmed:
12
3
2020
medline:
20
11
2020
Statut:
epublish
Résumé
TNFR-associated factor 5 (TRAF5) is a cytosolic adaptor protein and functions as an inflammatory regulator. However, the in vivo function of TRAF5 remains unclear, and how TRAF5 controls inflammatory responses in the intestine is not well understood. In this study, we found that intestinal epithelial cells from
Identifiants
pubmed: 32156688
pii: 4/3/129
doi: 10.4049/immunohorizons.2000007
doi:
Substances chimiques
Cytokines
0
TNF Receptor-Associated Factor 2
0
TNF Receptor-Associated Factor 5
0
TRAF2 protein, mouse
0
Dextran Sulfate
9042-14-2
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
129-139Commentaires et corrections
Type : ErratumIn
Informations de copyright
Copyright © 2020 The Authors.