Impact of dipyridamole on adenosine dosing in pediatric and young adult patients after heart transplantation.


Journal

Pediatric transplantation
ISSN: 1399-3046
Titre abrégé: Pediatr Transplant
Pays: Denmark
ID NLM: 9802574

Informations de publication

Date de publication:
05 2020
Historique:
received: 08 09 2019
revised: 08 02 2020
accepted: 12 02 2020
pubmed: 12 3 2020
medline: 10 4 2021
entrez: 12 3 2020
Statut: ppublish

Résumé

Relative contraindications to adenosine use have included heart transplant and dipyridamole. We previously demonstrated the safety and efficacy of adenosine-induced atrioventricular (AV) block in healthy young heart transplant recipients while suspending dipyridamole therapy (dual antiplatelet agent). This prospective follow-up study evaluated the safety and efficacy of adenosine use in the same cohort of heart transplant recipients while on dipyridamole. Adenosine was incrementally dosed until AV block occurred (maximum 200 mcg/kg up to 12 mg). The primary outcome was clinically significant asystole (≥12 seconds). Secondary outcomes included maximal adenosine dose, AV block duration, dysrhythmias, and clinical symptoms. Outcomes were compared to the parent study. Thirty of 39 eligible patients (5-24 years) were tested. No patient (0%, CI 0%-8%) experienced clinically significant asystole. AV block occurred in 29/30 patients (97%, CI 86%-100%). The median dose causing AV block was 50mcg/kg (vs 100 mcg/kg off dipyridamole; P = .011). Seventeen patients (57%, CI 39%-72%) required less adenosine to achieve AV block on dipyridamole; six (20%) required more. AV block occurred at doses ≥25 mcg/kg in all patients. In pairwise comparison to prior testing off dipyridamole, no significant change occurred in AV block duration, frequency of cardiac ectopy, or incidence of reported symptoms. No atrial fibrillation/flutter occurred. AV block often occurs at twofold lower adenosine doses in healthy young heart transplant recipients taking oral dipyridamole, compared with previous testing of this cohort off dipyridamole. Results suggest that initial dosing of 25 mcg/kg (maximum 0.8 mg) with stepwise escalation poses low risk of prolonged asystole on dipyridamole.

Sections du résumé

BACKGROUND
Relative contraindications to adenosine use have included heart transplant and dipyridamole. We previously demonstrated the safety and efficacy of adenosine-induced atrioventricular (AV) block in healthy young heart transplant recipients while suspending dipyridamole therapy (dual antiplatelet agent). This prospective follow-up study evaluated the safety and efficacy of adenosine use in the same cohort of heart transplant recipients while on dipyridamole.
METHODS
Adenosine was incrementally dosed until AV block occurred (maximum 200 mcg/kg up to 12 mg). The primary outcome was clinically significant asystole (≥12 seconds). Secondary outcomes included maximal adenosine dose, AV block duration, dysrhythmias, and clinical symptoms. Outcomes were compared to the parent study.
RESULTS
Thirty of 39 eligible patients (5-24 years) were tested. No patient (0%, CI 0%-8%) experienced clinically significant asystole. AV block occurred in 29/30 patients (97%, CI 86%-100%). The median dose causing AV block was 50mcg/kg (vs 100 mcg/kg off dipyridamole; P = .011). Seventeen patients (57%, CI 39%-72%) required less adenosine to achieve AV block on dipyridamole; six (20%) required more. AV block occurred at doses ≥25 mcg/kg in all patients. In pairwise comparison to prior testing off dipyridamole, no significant change occurred in AV block duration, frequency of cardiac ectopy, or incidence of reported symptoms. No atrial fibrillation/flutter occurred.
CONCLUSIONS
AV block often occurs at twofold lower adenosine doses in healthy young heart transplant recipients taking oral dipyridamole, compared with previous testing of this cohort off dipyridamole. Results suggest that initial dosing of 25 mcg/kg (maximum 0.8 mg) with stepwise escalation poses low risk of prolonged asystole on dipyridamole.

Identifiants

pubmed: 32157785
doi: 10.1111/petr.13689
pmc: PMC9610548
mid: NIHMS1842941
doi:

Substances chimiques

Anti-Arrhythmia Agents 0
Dipyridamole 64ALC7F90C
Adenosine K72T3FS567

Types de publication

Clinical Trial Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e13689

Subventions

Organisme : NHLBI NIH HHS
ID : K23 HL133454
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR000040
Pays : United States
Organisme : NIH HHS
ID : K23 HL133454
Pays : United States
Organisme : NIH HHS
ID : UL1 TR000040
Pays : United States

Informations de copyright

© 2020 Wiley Periodicals, Inc.

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Auteurs

Michael B Satzer (MB)

Division of Pediatric Cardiology, Columbia University Irving Medical Center, New York, NY, USA.

Jonathan N Flyer (JN)

Division of Pediatric Cardiology, Columbia University Irving Medical Center, New York, NY, USA.
Department of Pediatrics, Division of Pediatric Cardiology, The Robert Larner, M.D. College of Medicine at The University of Vermont, Burlington, VT, USA.

Warren A Zuckerman (WA)

Division of Pediatric Cardiology, Columbia University Irving Medical Center, New York, NY, USA.

Leonardo Liberman (L)

Division of Pediatric Cardiology, Columbia University Irving Medical Center, New York, NY, USA.

Marc E Richmond (ME)

Division of Pediatric Cardiology, Columbia University Irving Medical Center, New York, NY, USA.

Brett R Anderson (BR)

Division of Pediatric Cardiology, Columbia University Irving Medical Center, New York, NY, USA.

Linda J Addonizio (LJ)

Division of Pediatric Cardiology, Columbia University Irving Medical Center, New York, NY, USA.

Eric S Silver (ES)

Division of Pediatric Cardiology, Columbia University Irving Medical Center, New York, NY, USA.

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Classifications MeSH