Expression of Olig2, Nestin, NogoA and AQP4 have no impact on overall survival in IDH-wildtype glioblastoma.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2020
Historique:
received: 19 06 2019
accepted: 03 02 2020
entrez: 12 3 2020
pubmed: 12 3 2020
medline: 8 7 2020
Statut: epublish

Résumé

Despite many years of research efforts and clinical trials the prognosis of patients diagnosed with glioblastoma remains very poor. The oligodendrocyte transcription factor 2 (Olig2) was identified as a marker for glioma stem cells, which are believed to be responsible for glioma recurrence and therapy resistance. In this retrospective analysis we assessed the prognostic value of oligodendroglial and glioma stem cell markers in 113 IDH-wildtype glioblastomas. Immunohistochemical staining for Olig2, NogoA, AQP4 and Nestin was performed in combination with sequencing of IDH1 and IDH2 as well as promotor methylation analysis of the MGMT gene. Even though differences in overall survival according to Olig2 expression were observed, univariate and multivariate survival analysis did not reveal a firm significant prognostic impact of Olig2, NogoA, AQP4 or Nestin expression. Additionally, no differences in the expression of these markers depending on clinical status, age or gender were found. The established independent prognostic factors age<65, Karnofsky Performance Status> = 70 and methylated MGMT gene promoter were significant in the multivariate analysis. In conclusion expression of oligodendroglial and glioma stem cell markers do not have an independent prognostic effect in IDH-wildtype glioblastoma.

Identifiants

pubmed: 32160197
doi: 10.1371/journal.pone.0229274
pii: PONE-D-19-17358
pmc: PMC7065747
doi:

Substances chimiques

AQP4 protein, human 0
Aquaporin 4 0
Biomarkers, Tumor 0
NES protein, human 0
Nestin 0
Nogo Proteins 0
OLIG2 protein, human 0
Oligodendrocyte Transcription Factor 2 0
IDH2 protein, human EC 1.1.1.41
Isocitrate Dehydrogenase EC 1.1.1.41
IDH1 protein, human EC 1.1.1.42.

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0229274

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Felix Behling (F)

Institute of Neuropathology, University Medical Center Goettingen, Goettingen, Germany.
Department of Neurosurgery, University Hospital Tuebingen, Tuebingen, Germany.
Center for CNS Tumors, Comprehensive Cancer Center Tuebingen-Stuttgart, University Hospital Tuebingen, Tuebingen, Germany.

Alonso Barrantes-Freer (A)

Institute of Neuropathology, University Medical Center Goettingen, Goettingen, Germany.
Department of Neuropathology, Leipzig University Medicine, Leipzig, Germany.

Carl Ludwig Behnes (CL)

Institute of Pathology, University Medical Center Goettingen, Goettingen, Germany.

Florian Stockhammer (F)

Department of Neurosurgery, University Medical Center Goettingen, Goettingen, Germany.

Veit Rohde (V)

Department of Neurosurgery, University Medical Center Goettingen, Goettingen, Germany.

Antonia Adel-Horowski (A)

Department of Neurosurgery, University Medical Center Goettingen, Goettingen, Germany.

Odir Antonio Rodríguez-Villagra (OA)

Neuroscience Research Center, University of Costa Rica, San José, Costa Rica.
Institute for Psychological Research, University of Costa Rica, San José, Costa Rica.

Miguel Angel Barboza (MA)

Neurosciences Department, Hospital Dr. Rafael A. Calderón Guardia, CCSS, University of Costa Rica, San José, Costa Rica.

Wolfgang Brück (W)

Institute of Neuropathology, University Medical Center Goettingen, Goettingen, Germany.

Ulrich Lehmann (U)

Institute of Pathology, Hannover Medical School, Hannover, Germany.

Christine Stadelmann (C)

Institute of Neuropathology, University Medical Center Goettingen, Goettingen, Germany.

Christian Hartmann (C)

Department of Neuropathology, Institute of Pathology, Hannover Medical School, Hannover, Germany.

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