The Use of Transdermal Estrogen in Castrate-resistant, Steroid-refractory Prostate Cancer.


Journal

Clinical genitourinary cancer
ISSN: 1938-0682
Titre abrégé: Clin Genitourin Cancer
Pays: United States
ID NLM: 101260955

Informations de publication

Date de publication:
06 2020
Historique:
received: 12 05 2019
revised: 25 08 2019
accepted: 10 09 2019
pubmed: 17 3 2020
medline: 16 6 2021
entrez: 16 3 2020
Statut: ppublish

Résumé

Androgen-deprivation therapy is the mainstay of treatment for metastatic prostate cancer. Corticosteroids and estrogens are also useful agents in castration-resistant prostate cancer (CRPC). However, oral estrogens are associated with thromboembolic events, which limits their use, and transdermal estrogens may offer a safer alternative. This study was carried out to determine the safety and effectiveness of transdermal estrogens in CRPC. Forty-one patients with CRPC and steroid-resistant prostate cancer were eligible for this dose-escalation study of transdermal estradiol. A starting dose of 50 mcg/24 hours was applied and increased if prostate-specific antigen (PSA) rose > 5 ng/mL in steps to 300 mcg/24 hours. The primary endpoint was PSA response, and secondary outcomes included incidence of thromboembolic events and progression-free survival. Patients who progressed were offered diethylstilbestrol. Five (13%) of 40 patients had > 50% PSA reduction for at least 1 month at any transdermal estradiol dose. No venous-thromboembolic events were observed, and responses plateaued at 200 mcg/24 hours. A correlation between PSA response and rising sex hormone binding globulin was seen. Fifty percent of patients subsequently responded to low-dose diethylstilbestrol. Transdermal estradiol appears to be a low toxicity treatment option to control CRPC after failure of steroid therapy. Modulation of sex hormone binding globulin by transdermal estradiol may be one mechanism of action of estrogens on CRPC. Oral estrogens remain effective after the use of transdermal estradiol.

Sections du résumé

BACKGROUND
Androgen-deprivation therapy is the mainstay of treatment for metastatic prostate cancer. Corticosteroids and estrogens are also useful agents in castration-resistant prostate cancer (CRPC). However, oral estrogens are associated with thromboembolic events, which limits their use, and transdermal estrogens may offer a safer alternative. This study was carried out to determine the safety and effectiveness of transdermal estrogens in CRPC.
PATIENTS AND METHODS
Forty-one patients with CRPC and steroid-resistant prostate cancer were eligible for this dose-escalation study of transdermal estradiol. A starting dose of 50 mcg/24 hours was applied and increased if prostate-specific antigen (PSA) rose > 5 ng/mL in steps to 300 mcg/24 hours. The primary endpoint was PSA response, and secondary outcomes included incidence of thromboembolic events and progression-free survival. Patients who progressed were offered diethylstilbestrol.
RESULTS
Five (13%) of 40 patients had > 50% PSA reduction for at least 1 month at any transdermal estradiol dose. No venous-thromboembolic events were observed, and responses plateaued at 200 mcg/24 hours. A correlation between PSA response and rising sex hormone binding globulin was seen. Fifty percent of patients subsequently responded to low-dose diethylstilbestrol.
CONCLUSION
Transdermal estradiol appears to be a low toxicity treatment option to control CRPC after failure of steroid therapy. Modulation of sex hormone binding globulin by transdermal estradiol may be one mechanism of action of estrogens on CRPC. Oral estrogens remain effective after the use of transdermal estradiol.

Identifiants

pubmed: 32171601
pii: S1558-7673(19)30288-5
doi: 10.1016/j.clgc.2019.09.019
pii:
doi:

Substances chimiques

Estrogens 0
Steroids 0

Types de publication

Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e217-e223

Subventions

Organisme : Department of Health
ID : NIHR-RP-011-053
Pays : United Kingdom

Informations de copyright

Crown Copyright © 2019. Published by Elsevier Inc. All rights reserved.

Auteurs

Katherine Smith (K)

Barts Cancer Institute, Saint Bartholomew's Hospital, London, UK.

Myria Galazi (M)

Barts Cancer Institute, Saint Bartholomew's Hospital, London, UK.

Mark R Openshaw (MR)

Department of Medical Oncology, Charing Cross Hospital, Imperial College NHS Trust, London, UK.

Peter Wilson (P)

Barts Cancer Institute, Saint Bartholomew's Hospital, London, UK.

Shah J Sarker (SJ)

Centre for Experimental Cancer Medicine, Queen Mary University of London, London, UK.

Neale O'Brien (N)

Barts Cancer Institute, Saint Bartholomew's Hospital, London, UK.

Constantine Alifrangis (C)

Barts Cancer Institute, Saint Bartholomew's Hospital, London, UK.

Justin Stebbing (J)

Department of Medical Oncology, Charing Cross Hospital, Imperial College NHS Trust, London, UK.

Jonathan Shamash (J)

Barts Cancer Institute, Saint Bartholomew's Hospital, London, UK. Electronic address: jonathan.shamash@bartshealth.nhs.uk.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH