Renal Fibrosis Is Significantly Attenuated Following Targeted Disruption of
Animals
Blood Pressure
CD40 Antigens
/ genetics
Cell Line
Disease Models, Animal
Fibrosis
Glomerular Filtration Rate
Humans
Inflammation Mediators
/ metabolism
Ischemia
/ genetics
Kidney
/ blood supply
Kidney Tubules, Proximal
/ metabolism
Male
Mutation
Oxidative Stress
Plasminogen Activator Inhibitor 1
/ metabolism
Rats, Inbred Dahl
Renal Artery Obstruction
/ genetics
Signal Transduction
CD40
renal fibrosis
renal ischemia
Journal
Journal of the American Heart Association
ISSN: 2047-9980
Titre abrégé: J Am Heart Assoc
Pays: England
ID NLM: 101580524
Informations de publication
Date de publication:
07 04 2020
07 04 2020
Historique:
entrez:
24
3
2020
pubmed:
24
3
2020
medline:
9
3
2021
Statut:
ppublish
Résumé
Background Renal artery stenosis is a common cause of renal ischemia, contributing to the development of chronic kidney disease. To investigate the role of local CD40 expression in renal artery stenosis, Goldblatt 2-kidney 1-clip surgery was performed on hypertensive Dahl salt-sensitive rats (S rats) and genetically modified S rats in which CD40 function is abolished (
Identifiants
pubmed: 32200719
doi: 10.1161/JAHA.119.014072
pmc: PMC7428653
doi:
Substances chimiques
CD40 Antigens
0
Inflammation Mediators
0
Plasminogen Activator Inhibitor 1
0
Serpine1 protein, rat
0
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
e014072Subventions
Organisme : NHLBI NIH HHS
ID : R01 HL137004
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL105649
Pays : United States
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