Chondrocytes respond to an altered heparan sulfate composition with distinct changes of heparan sulfate structure and increased levels of chondroitin sulfate.


Journal

Matrix biology : journal of the International Society for Matrix Biology
ISSN: 1569-1802
Titre abrégé: Matrix Biol
Pays: Netherlands
ID NLM: 9432592

Informations de publication

Date de publication:
11 2020
Historique:
received: 26 08 2019
revised: 11 03 2020
accepted: 12 03 2020
pubmed: 24 3 2020
medline: 24 8 2021
entrez: 24 3 2020
Statut: ppublish

Résumé

Heparan sulfate (HS) regulates the activity of many signaling molecules critical for the development of endochondral bones. Even so, mice with a genetically altered HS metabolism display a relatively mild skeletal phenotype compared to the defects observed in other tissues and organs pointing to a reduced HS dependency of growth-factor signaling in chondrocytes. To understand this difference, we have investigated the glycosaminoglycan (GAG) composition in two mouse lines that produce either reduced levels of HS (Ext1

Identifiants

pubmed: 32201365
pii: S0945-053X(20)30029-9
doi: 10.1016/j.matbio.2020.03.006
pii:
doi:

Substances chimiques

Chondroitin Sulfates 9007-28-7
Heparitin Sulfate 9050-30-0
N-Acetylglucosaminyltransferases EC 2.4.1.-
exostosin-1 EC 2.4.1.224
Sulfotransferases EC 2.8.2.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

43-59

Informations de copyright

Copyright © 2020 The Authors. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration Competing of Interest The authors declare no conflict of interest.

Auteurs

Velina Bachvarova (V)

Department of Developmental Biology, Faculty of Biology and Centre for Medical Biotechnology, University of Duisburg-Essen, Universitätsstr 1-5,45117 Essen, Germany. Electronic address: velina.bachvarova@uni-due.de.

Tabea Dierker (T)

Department of Medical Biochemistry and Microbiology, and Science for Life Laboratory, Uppsala University, Box 582, Uppsala, Sweden. Electronic address: tabea.dierker@imbim.uu.se.

Jeffrey Esko (J)

Department of Cellular and Molecular Medicine, UCSD, United States. Electronic address: jesko@ucsd.edu.

Daniel Hoffmann (D)

Department of Bioinformatics and Computational Biophysics, Faculty of Biology and Centre for Medical Biotechnology, University of Duisburg-Essen, Germany. Electronic address: daniel.hoffmann@uni-due.de.

Lena Kjellen (L)

Department of Medical Biochemistry and Microbiology, and Science for Life Laboratory, Uppsala University, Box 582, Uppsala, Sweden. Electronic address: lena.kjellen@imbim.uu.se.

Andrea Vortkamp (A)

Department of Developmental Biology, Faculty of Biology and Centre for Medical Biotechnology, University of Duisburg-Essen, Universitätsstr 1-5,45117 Essen, Germany. Electronic address: andrea.vortkamp@uni-due.de.

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Classifications MeSH