Preventive effect of fluvastatin on the development of medication-related osteonecrosis of the jaw.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
27 03 2020
Historique:
received: 03 11 2019
accepted: 01 03 2020
entrez: 30 3 2020
pubmed: 30 3 2020
medline: 1 12 2020
Statut: epublish

Résumé

Medication-related osteonecrosis of the jaw (MRONJ) occurs in patients undergoing oral surgery while medicated with bisphosphonate, denosumab or anti-angiogenic agents. We employed a MRONJ-like rat model to investigate whether injecting fluvastatin at extraction sites prevents MRONJ-like lesion. A MRONJ-like model was created by treating rats with zoledronate and dexamethasone, extracting teeth, and immediately injecting fluvastatin at the extraction site. The experimental group comprised three subgroups treated with low (0.1 mg/kg; FS-L), medium (1.0 mg/kg; FS-M) and high concentrations (10 mg/kg; FS-H) of fluvastatin. Necrotic bone exposure was significantly lower in the FS-M (p = 0.028) and FS-H (p = 0.041) groups than in the MRONJ group. The distance between the edges of the epithelial surfaces was significantly shorter in the FS-M (p = 0.042) and FS-H (p = 0.041) groups. The area of necrotic bone and the necrotic bone ratio were significantly smaller in the FS-H group (p = 0.041 and p = 0.042 respectively). Bone volume fraction calculated on μ-CT images was significantly larger in the FS-H group than in the MRONJ group (p = 0.021). Our findings suggest that a single local injection of fluvastatin following tooth extraction can potentially reduce the chance of developing MRONJ-like lesion in rats.

Identifiants

pubmed: 32221325
doi: 10.1038/s41598-020-61724-6
pii: 10.1038/s41598-020-61724-6
pmc: PMC7101417
doi:

Substances chimiques

Angiogenesis Inhibitors 0
Bone Density Conservation Agents 0
Diphosphonates 0
Denosumab 4EQZ6YO2HI
Fluvastatin 4L066368AS
Zoledronic Acid 6XC1PAD3KF

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

5620

Références

Cremers, S., Drake, M. T., Ebetino, F. H., Bilezikian, J. P. & Russell, R. G. G. Pharmacology of bisphosphonates. Br. J. Clin. Pharmacol. 85, 1052–1062, https://doi.org/10.1111/bcp.13867 (2019).
doi: 10.1111/bcp.13867 pubmed: 30650219 pmcid: 30650219
Marx, R. E. Pamidronate (Aredia) and zoledronate (Zometa) induced avascular necrosis of the jaws: A growing epidemic [1]. J. Oral Maxillofac. Surg. 61, 1115–1117, https://doi.org/10.1016/S0278-2391(03)00720-1 (2003).
doi: 10.1016/S0278-2391(03)00720-1 pubmed: 12966493 pmcid: 12966493
Ruggiero, S. L., Mehrotra, B., Rosenberg, T. J. & Engroff, S. L. Osteonecrosis of the Jaws Associated with the Use of Bisphosphonates: A Review of 63 Cases. J. Oral Maxillofac. Surg. 62, 527–534, https://doi.org/10.1016/j.joms.2004.02.004 (2004).
doi: 10.1016/j.joms.2004.02.004 pubmed: 15122554 pmcid: 15122554
Migliorati, C. A., Schubert, M. M., Peterson, D. E. & Seneda, L. M. Bisphosphonate-associated osteonecrosis of mandibular and maxillary bone - An emerging oral complication of supportive cancer therapy. Cancer 104, 83–93, https://doi.org/10.1002/cncr.21130 (2005).
doi: 10.1002/cncr.21130 pubmed: 15929121 pmcid: 15929121
Bagan, J. V. et al. Jaw osteonecrosis associated with bisphosphonates: multiple exposed areas and its relationship to teeth extractions. Study of 20 cases. Oral Oncol. 42, 327–329, https://doi.org/10.1016/j.oraloncology.2005.08.001 (2006).
doi: 10.1016/j.oraloncology.2005.08.001 pubmed: 16275156 pmcid: 16275156
Aghaloo, T. L., Felsenfeld, A. L. & Tetradis, S. Osteonecrosis of the Jaw in a Patient on Denosumab. J. Oral Maxillofac. Surg. 68, 959–963, https://doi.org/10.1016/j.joms.2009.10.010 (2010).
doi: 10.1016/j.joms.2009.10.010 pubmed: 20149510 pmcid: 20149510
Taylor, K. H., Middlefell, L. S. & Mizen, K. D. Osteonecrosis of the jaws induced by anti-RANK ligand therapy. Br. J. Oral Maxillofac. Surg. 48, 221–223, https://doi.org/10.1016/j.bjoms.2009.08.030 (2010).
doi: 10.1016/j.bjoms.2009.08.030 pubmed: 19836866 pmcid: 19836866
Ruggiero, S. L. et al. American Association of Oral and Maxillofacial Surgeons Position Paper on Medication-Related Osteonecrosis of the Jaw-2014 Update. J. Oral Maxillofac. Surg. 72, 1938–1956, https://doi.org/10.1016/j.joms.2014.04.031 (2014).
doi: 10.1016/j.joms.2014.04.031 pubmed: 25234529 pmcid: 25234529
Khan, A. A. et al. Case-Based Review of Osteonecrosis of the Jaw (ONJ) and Application of the International Recommendations for Management From the International Task Force on ONJ. J. Clin. Densitom. 20, 8–24, https://doi.org/10.1016/j.jocd.2016.09.005 (2017).
doi: 10.1016/j.jocd.2016.09.005 pubmed: 27956123 pmcid: 27956123
Ristow, O. et al. Is the conservative non-surgical management of medication-related osteonecrosis of the jaw an appropriate treatment option for early stages? A long-term single-center cohort study. J. Craniomaxillofac. Surg. 47, 491–499, https://doi.org/10.1016/j.jcms.2018.12.014 (2019).
doi: 10.1016/j.jcms.2018.12.014 pubmed: 30642734 pmcid: 30642734
Melea, P. I. et al. Conservative treatment of bisphosphonate-related osteonecrosis of the jaw in multiple myeloma patients. Int J Dent 2014, 427273, https://doi.org/10.1155/2014/427273 (2014).
doi: 10.1155/2014/427273 pubmed: 25045353 pmcid: 25045353
Bagan, J. et al. Medication-related osteonecrosis of the jaw associated with bisphosphonates and denosumab in osteoporosis. Oral Dis. 22, 324–329, https://doi.org/10.1111/odi.12447 (2016).
doi: 10.1111/odi.12447 pubmed: 26818808 pmcid: 26818808
Kakehashi, H. et al. Administration of teriparatide improves the symptoms of advanced bisphosphonate-related osteonecrosis of the jaw: preliminary findings. Int. J. Oral Maxillofac. Surg. 44, 1558–1564, https://doi.org/10.1016/j.ijom.2015.07.018 (2015).
doi: 10.1016/j.ijom.2015.07.018 pubmed: 26304604 pmcid: 26304604
Jung, J. et al. Short-Term Teriparatide and Recombinant Human Bone Morphogenetic Protein-2 for Regenerative Approach to Medication-Related Osteonecrosis of the Jaw: A Preliminary Study. J. Bone Miner. Res. 32, 2445–2452, https://doi.org/10.1002/jbmr.3237 (2017).
doi: 10.1002/jbmr.3237 pubmed: 28815779 pmcid: 28815779
Heggendorn, F. L. et al. Bisphosphonate-related osteonecrosis of the jaws: Report of a case using conservative protocol. Spec. Care Dentist. 36, 43–47, https://doi.org/10.1111/scd.12143 (2016).
doi: 10.1111/scd.12143 pubmed: 26782365 pmcid: 26782365
Mauceri, R. et al. Conservative Surgical Treatment of Bisphosphonate-Related Osteonecrosis of the Jaw with Er,Cr:YSGG Laser and Platelet-Rich Plasma: A Longitudinal Study. Biomed Res Int 2018, 3982540, https://doi.org/10.1155/2018/3982540 (2018).
doi: 10.1155/2018/3982540 pubmed: 30211221 pmcid: 30211221
Freiberger, J. J. et al. What is the role of hyperbaric oxygen in the management of bisphosphonate-related osteonecrosis of the jaw: A randomized controlled trial of hyperbaric oxygen as an adjunct to surgery and antibiotics. J. Oral Maxillofac. Surg. 70, 1573–1583, https://doi.org/10.1016/j.joms.2012.04.001 (2012).
doi: 10.1016/j.joms.2012.04.001 pubmed: 22698292 pmcid: 22698292
Curi, M. M. et al. Bisphosphonate-Related Osteonecrosis of the Jaws-An Initial Case Series Report of Treatment Combining Partial Bone Resection and Autologous Platelet-Rich Plasma. J. Oral Maxillofac. Surg. 69, 2465–2472, https://doi.org/10.1016/j.joms.2011.02.078 (2011).
doi: 10.1016/j.joms.2011.02.078 pubmed: 21763050 pmcid: 21763050
Lesclous, P. et al. Bisphosphonate-associated osteonecrosis of the jaw: A key role of inflammation? Bone 45, 843–852, https://doi.org/10.1016/j.bone.2009.07.011 (2009).
doi: 10.1016/j.bone.2009.07.011 pubmed: 19631301 pmcid: 19631301
Zirk, M. et al. Microbial diversity in infections of patients with medication-related osteonecrosis of the jaw. Clin. Oral Investig. 23, 2143–2151, https://doi.org/10.1007/s00784-018-2655-z (2019).
doi: 10.1007/s00784-018-2655-z pubmed: 30276516 pmcid: 30276516
Allen, M. R. & Burr, D. B. Mandible Matrix Necrosis in Beagle Dogs After 3 Years of Daily Oral Bisphosphonate Treatment. J. Oral Maxillofac. Surg. 66, 987–994, https://doi.org/10.1016/j.joms.2008.01.038 (2008).
doi: 10.1016/j.joms.2008.01.038 pubmed: 18423290 pmcid: 18423290
Fournier, P. et al. Bisphosphonates inhibit angiogenesis in vitro and testosterone-stimulated vascular regrowth in the ventral prostate in castrated rats. Cancer Res. 62, 6538–6544 (2002).
pubmed: 12438248 pmcid: 12438248
Santini, D. et al. Zoledronic acid induces significant and long-lasting modifications of circulating angiogenic factors in cancer patients. Clin. Cancer Res. 9, 2893–2897 (2003).
pubmed: 12912933 pmcid: 12912933
Gkouveris, I. et al. Vasculature submucosal changes at early stages of osteonecrosis of the jaw (ONJ). Bone 123, 234–245, https://doi.org/10.1016/j.bone.2019.03.031 (2019).
doi: 10.1016/j.bone.2019.03.031 pubmed: 30953717 pmcid: 30953717
Ravosa, M. J., Ning, J., Liu, Y. & Stack, M. S. Bisphosphonate effects on the behaviour of oral epithelial cells and oral fibroblasts. Arch. Oral Biol. 56, 491–498, https://doi.org/10.1016/j.archoralbio.2010.11.003 (2011).
doi: 10.1016/j.archoralbio.2010.11.003 pubmed: 21146154 pmcid: 21146154
Soydan, S. S. et al. Effects of alendronate and pamidronate on apoptosis and cell proliferation in cultured primary human gingival fibroblasts. Hum. Exp. Toxicol. 34, 1073–1082, https://doi.org/10.1177/0960327115569808 (2015).
doi: 10.1177/0960327115569808 pubmed: 25636638 pmcid: 25636638
Zhang, Q. et al. IL-17-mediated M1/M2 macrophage alteration contributes to pathogenesis of bisphosphonate-related osteonecrosis of the jaws. Clin. Cancer Res. 19, 3176–3188, https://doi.org/10.1158/1078-0432.Ccr-13-0042 (2013).
doi: 10.1158/1078-0432.Ccr-13-0042 pubmed: 23616636 pmcid: 23616636
Zhu, W. et al. Zoledronic acid promotes TLR-4-mediated M1 macrophage polarization in bisphosphonate-related osteonecrosis of the jaw. FASEB J. 33, 5208–5219, https://doi.org/10.1096/fj.201801791RR (2019).
doi: 10.1096/fj.201801791RR pubmed: 30624969 pmcid: 30624969
Mundy, G. et al. Stimulation of bone formation in vitro and in rodents by statins. Science 286, 1946–1949, https://doi.org/10.1126/science.286.5446.1946 (1999).
doi: 10.1126/science.286.5446.1946 pubmed: 10583956 pmcid: 10583956
Ayukawa, Y., Okamura, A. & Koyano, K. Simvastatin promotes osteogenesis around titanium implants. Clin. Oral Implants Res. 15, 346–350, https://doi.org/10.1046/j.1600-0501.2003.01015.x (2004).
doi: 10.1046/j.1600-0501.2003.01015.x pubmed: 15142098 pmcid: 15142098
Sakoda, K. et al. Simvastatin decreases IL-6 and IL-8 production in epithelial cells. J. Dent. Res. 85, 520–523, https://doi.org/10.1177/154405910608500608 (2006).
doi: 10.1177/154405910608500608 pubmed: 16723648 pmcid: 16723648
Hassan, H. M., Al-Gayyar, M. M. H., El-Gayar, A. M. & Ibrahim, T. M. Effect of simvastatin on inflammatory cytokines balance in air pouch granuloma model. Inflammation and Allergy - Drug Targets 13, 74–79, https://doi.org/10.2174/1871528112666131230012026 (2014).
doi: 10.2174/1871528112666131230012026 pubmed: 24372369 pmcid: 24372369
Jerwood, S. & Cohen, J. Unexpected antimicrobial effect of statins. J. Antimicrob. Chemother. 61, 362–364, https://doi.org/10.1093/jac/dkm496 (2008).
doi: 10.1093/jac/dkm496 pubmed: 18086693 pmcid: 18086693
Ko, H. H. T., Lareu, R. R., Dix, B. R. & Hughes, J. D. In vitro antibacterial effects of statins against bacterial pathogens causing skin infections. Eur. J. Clin. Microbiol. Infect. Dis. 37, 1125–1135, https://doi.org/10.1007/s10096-018-3227-5 (2018).
doi: 10.1007/s10096-018-3227-5 pubmed: 29569046 pmcid: 29569046
Altieri, D. C. Statins’ benefits begin to sprout. J. Clin. Invest. 108, 365–366, https://doi.org/10.1172/jci13556 (2001).
doi: 10.1172/jci13556 pubmed: 11489928 pmcid: 11489928
Zhang, Y. et al. Simvastatin augments the efficacy of therapeutic angiogenesis induced by bone marrow-derived mesenchymal stem cells in a murine model of hindlimb ischemia. Mol. Biol. Rep. 39, 285–293, https://doi.org/10.1007/s11033-011-0737-y (2012).
doi: 10.1007/s11033-011-0737-y pubmed: 21553056 pmcid: 21553056
Yasunami, N. et al. Acceleration of hard and soft tissue healing in the oral cavity by a single transmucosal injection of fluvastatin-impregnated poly (lactic-co-glycolic acid) microspheres. An in vitro and rodent in vivo study. Biomed. Mater. 11, 015001, https://doi.org/10.1088/1748-6041/11/1/015001 (2015).
doi: 10.1088/1748-6041/11/1/015001
The ARRIVE Guidelines: Animal Research: Reporting of In Vivo Experiments, https://www.nc3rs.org.uk/sites/default/files/documents/Guidelines/NC3Rs%20ARRIVE%20Guidelines%202013.pdf .
Kaibuchi, N., Iwata, T., Yamato, M., Okano, T. & Ando, T. Multipotent mesenchymal stromal cell sheet therapy for bisphosphonate-related osteonecrosis of the jaw in a rat model. Acta Biomater. 42, 400–410, https://doi.org/10.1016/j.actbio.2016.06.022 (2016).
doi: 10.1016/j.actbio.2016.06.022 pubmed: 27326918 pmcid: 27326918
Kuroshima, S., Entezami, P., McCauley, L. K. & Yamashita, J. Early effects of parathyroid hormone on bisphosphonate/steroid-associated compromised osseous wound healing. Osteoporos. Int. 25, 1141–1150, https://doi.org/10.1007/s00198-013-2570-8 (2014).
doi: 10.1007/s00198-013-2570-8 pubmed: 24301537 pmcid: 24301537
Araújo, M. G., Wennström, J. L. & Lindhe, J. Modeling of the buccal and lingual bone walls of fresh extraction sites following implant installation. Clin. Oral Implants Res. 17, 606–614, https://doi.org/10.1111/j.1600-0501.2006.01315.x (2006).
doi: 10.1111/j.1600-0501.2006.01315.x pubmed: 17092217 pmcid: 17092217
Ogata, K. et al. Evaluation of the therapeutic effects of conditioned media from mesenchymal stem cells in a rat bisphosphonate-related osteonecrosis of the jaw-like model. Bone 74, 95–105, https://doi.org/10.1016/j.bone.2015.01.011 (2015).
doi: 10.1016/j.bone.2015.01.011 pubmed: 25613174 pmcid: 25613174
Saad, F. et al. Incidence, risk factors, and outcomes of osteonecrosis of the jaw: Integrated analysis from three blinded active-controlled phase III trials in cancer patients with bone metastases. Ann. Oncol. 23, 1341–1347, https://doi.org/10.1093/annonc/mdr435 (2012).
doi: 10.1093/annonc/mdr435 pubmed: 21986094 pmcid: 21986094
Aghaloo, T. L. et al. RANKL inhibitors induce osteonecrosis of the jaw in mice with periapical disease. J. Bone Miner. Res. 29, 843–854, https://doi.org/10.1002/jbmr.2097 (2014).
doi: 10.1002/jbmr.2097 pubmed: 24115073 pmcid: 24115073
Vukelic, S. et al. Farnesyl pyrophosphate inhibits epithelialization and wound healing through the glucocorticoid receptor. J. Biol. Chem. 285, 1980–1988, https://doi.org/10.1074/jbc.M109.016741 (2010).
doi: 10.1074/jbc.M109.016741 pubmed: 19903814 pmcid: 19903814
Kureishi, Y. et al. The HMG-CoA reductase inhibitor simvastatin activates the protein kinase Akt and promotes angiogenesis in normocholesterolemic animals. Nat. Med. 6, 1004–1010, https://doi.org/10.1038/79510 (2000).
doi: 10.1038/79510 pubmed: 10973320 pmcid: 10973320
Greenwood, J., Steinman, L. & Zamvil, S. S. Statin therapy and autoimmune disease: From protein prenylation to immunomodulation. Nature Reviews Immunology 6, 358–370, https://doi.org/10.1038/nri1839 (2006).
doi: 10.1038/nri1839 pubmed: 16639429 pmcid: 16639429
Thangamani, S. et al. Exploring simvastatin, an antihyperlipidemic drug, as a potential topical antibacterial agent. Sci. Rep. 5, 16407, https://doi.org/10.1038/srep16407 (2015).
doi: 10.1038/srep16407 pubmed: 26553420 pmcid: 26553420
Gupta, M. & Kumar, A. Comparison of Minimum Inhibitory Concentration (MIC) value of statin drugs: A Systematic Review. Anti-Infective Agents 17, 4–19, https://doi.org/10.2174/2211352516666180629124433 (2018).
doi: 10.2174/2211352516666180629124433
Jain, M. K. & Ridker, P. M. Anti-inflammatory effects of statins: Clinical evidence and basic mechanisms. Nature Reviews Drug Discovery 4, 977–987, https://doi.org/10.1038/nrd1901 (2005).
doi: 10.1038/nrd1901 pubmed: 16341063 pmcid: 16341063
Li, X., Cui, Q., Kao, C., Wang, G. J. & Balian, G. Lovastatin inhibits adipogenic and stimulates osteogenic differentiation by suppressing PPARgamma2 and increasing Cbfa1/Runx2 expression in bone marrow mesenchymal cell cultures. Bone 33, 652–659,  https://doi.org/10.1016/s8756-3282(03)00239-4 (2003).
doi: 10.1016/s8756-3282(03)00239-4 pubmed: 14555271 pmcid: 14555271
Ohnaka, K. et al. Pitavastatin enhanced BMP-2 and osteocalcin expression by inhibition of Rho-associated kinase in human osteoblasts. Biochem. Biophys. Res. Commun. 287, 337–342, https://doi.org/10.1006/bbrc.2001.5597 (2001).
doi: 10.1006/bbrc.2001.5597 pubmed: 11554731 pmcid: 11554731
Oryan, A., Kamali, A. & Moshiri, A. Potential mechanisms and applications of statins on osteogenesis: Current modalities, conflicts and future directions. J. Control. Release 215, 12–24, https://doi.org/10.1016/j.jconrel.2015.07.022 (2015).
doi: 10.1016/j.jconrel.2015.07.022 pubmed: 26226345 pmcid: 26226345
Galus, R., Wlodarski, P. K. & Wlodarski, K. H. Fluvastatin increases heterotopically induced ossicles in mice. Clin. Exp. Pharmacol. Physiol. 33, 388–390, https://doi.org/10.1111/j.1440-1681.2006.04380.x (2006).
doi: 10.1111/j.1440-1681.2006.04380.x pubmed: 16620306 pmcid: 16620306
Galus, R., Wlodarski, P. & Wlodarski, K. Influence of fluvastatin on bone formation induced by demineralized bone matrix in mice. Pharmacol. Rep. 58, 443–447 (2006).
pubmed: 16845222 pmcid: 16845222
Kuroshima, S. & Yamashita, J. Chemotherapeutic and antiresorptive combination therapy suppressed lymphangiogenesis and induced osteonecrosis of the jaw-like lesions in mice. Bone 56, 101–109, https://doi.org/10.1016/j.bone.2013.05.013 (2013).
doi: 10.1016/j.bone.2013.05.013 pubmed: 23727433 pmcid: 23727433
García, M. J., Reinoso, R. F., Sánchez Navarro, A. & Prous, J. R. Clinical pharmacokinetics of statins. Methods Find. Exp. Clin. Pharmacol. 25, 457–481 (2003).
doi: 10.1358/mf.2003.25.6.769652 pubmed: 12949632 pmcid: 12949632
Ayukawa, Y. et al. Simvastatin enhances bone formation around titanium implants in rat tibiae. J. Oral Rehabil. 37, 123–130, https://doi.org/10.1111/j.1365-2842.2009.02011.x (2010).
doi: 10.1111/j.1365-2842.2009.02011.x pubmed: 19889034 pmcid: 19889034

Auteurs

Naomi Adachi (N)

Section of Implant and Rehabilitative Dentistry, Division of Oral Rehabilitation, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.

Yasunori Ayukawa (Y)

Section of Implant and Rehabilitative Dentistry, Division of Oral Rehabilitation, Faculty of Dental Science, Kyushu University, Fukuoka, Japan. ayukawa@dent.kyushu-u.ac.jp.

Noriyuki Yasunami (N)

Section of Implant and Rehabilitative Dentistry, Division of Oral Rehabilitation, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.
Department of Reconstructive Science, School of Dental Medicine, University of Connecticut, CT, USA.

Akihiro Furuhashi (A)

Section of Implant and Rehabilitative Dentistry, Division of Oral Rehabilitation, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.

Mikio Imai (M)

Section of Implant and Rehabilitative Dentistry, Division of Oral Rehabilitation, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.

Koma Sanda (K)

Section of Implant and Rehabilitative Dentistry, Division of Oral Rehabilitation, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.

Ikiru Atsuta (I)

Division of Advanced Dental Devices and Therapeutics, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.

Kiyoshi Koyano (K)

Section of Implant and Rehabilitative Dentistry, Division of Oral Rehabilitation, Faculty of Dental Science, Kyushu University, Fukuoka, Japan.

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