Astrocytes in Atp1a2-deficient heterozygous mice exhibit hyperactivity after induction of cortical spreading depression.
Animals
Astrocytes
/ metabolism
Calcium
/ analysis
Cations, Divalent
/ analysis
Cerebral Cortex
/ cytology
Cortical Spreading Depression
/ genetics
Disease Models, Animal
Female
Heterozygote
Humans
Intravital Microscopy
Mice
Mice, Transgenic
Microscopy, Fluorescence, Multiphoton
Migraine with Aura
/ genetics
Neurons
/ metabolism
Sodium-Potassium-Exchanging ATPase
/ deficiency
Stereotaxic Techniques
Atp1a2
CSD
FHM2
G-CaMP7
astrocyte
calcium imaging
migraine
Journal
FEBS open bio
ISSN: 2211-5463
Titre abrégé: FEBS Open Bio
Pays: England
ID NLM: 101580716
Informations de publication
Date de publication:
06 2020
06 2020
Historique:
received:
19
12
2019
revised:
01
03
2020
accepted:
25
03
2020
pubmed:
3
4
2020
medline:
20
7
2021
entrez:
3
4
2020
Statut:
ppublish
Résumé
The ATP1A2 coding α2 subunit of Na,K-ATPase, which is predominantly located in astrocytes, is a causative gene of familial hemiplegic migraine type 2 (FHM2). FHM2 model mice (Atp1a2
Identifiants
pubmed: 32237043
doi: 10.1002/2211-5463.12848
pmc: PMC7262908
doi:
Substances chimiques
Cations, Divalent
0
Atp1a2 protein, mouse
EC 3.6.1.-
Sodium-Potassium-Exchanging ATPase
EC 7.2.2.13
Calcium
SY7Q814VUP
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1031-1043Informations de copyright
© 2020 The Authors. Published by FEBS Press and John Wiley & Sons Ltd.
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