Neurofilament-light in former athletes: a potential biomarker of neurodegeneration and progression.


Journal

European journal of neurology
ISSN: 1468-1331
Titre abrégé: Eur J Neurol
Pays: England
ID NLM: 9506311

Informations de publication

Date de publication:
07 2020
Historique:
received: 25 03 2020
accepted: 02 04 2020
pubmed: 14 4 2020
medline: 29 6 2021
entrez: 14 4 2020
Statut: ppublish

Résumé

This study aimed to evaluate serum neurofilament light chain (NF-L) levels in former professional contact sports athletes with multiple concussions (ExPro) as a potential biomarker of neurodegeneration and predictor of white-matter (WM) abnormality progression. Concentrations of NF-L in the serum of fifty-two cognitively normal ExPro and twenty-one healthy controls (HC) with no history of concussions were measured using single molecule array (Simoa) technology. Both groups underwent neuroimaging at the time of serum collection. Eighteen of the participants in the ExPro underwent follow-up imaging after 2 years. Levels of serum NF-L were not significantly different between the ExPro and HC. However, in the ExPro group, NF-L levels were positively correlated with the mean diffusivity (MD) of corpus callosum (CC) and fornix, and total ventricular volume. Moreover, NF-L levels in the ExPro group at the first visit were positively correlated with the amount of increase in CC MD at the 2-year follow-up. NF-L levels reflect neuronal changes in the ExPro group and predict the extent of decrease in white matter integrity over time. Serum NF-L might be a biomarker of neurodegeneration and WM abnormality progression in ExPro.

Sections du résumé

BACKGROUND/OBJECTIVE
This study aimed to evaluate serum neurofilament light chain (NF-L) levels in former professional contact sports athletes with multiple concussions (ExPro) as a potential biomarker of neurodegeneration and predictor of white-matter (WM) abnormality progression.
METHODS
Concentrations of NF-L in the serum of fifty-two cognitively normal ExPro and twenty-one healthy controls (HC) with no history of concussions were measured using single molecule array (Simoa) technology. Both groups underwent neuroimaging at the time of serum collection. Eighteen of the participants in the ExPro underwent follow-up imaging after 2 years.
RESULTS
Levels of serum NF-L were not significantly different between the ExPro and HC. However, in the ExPro group, NF-L levels were positively correlated with the mean diffusivity (MD) of corpus callosum (CC) and fornix, and total ventricular volume. Moreover, NF-L levels in the ExPro group at the first visit were positively correlated with the amount of increase in CC MD at the 2-year follow-up.
CONCLUSIONS
NF-L levels reflect neuronal changes in the ExPro group and predict the extent of decrease in white matter integrity over time. Serum NF-L might be a biomarker of neurodegeneration and WM abnormality progression in ExPro.

Identifiants

pubmed: 32281206
doi: 10.1111/ene.14251
doi:

Substances chimiques

Biomarkers 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1170-1177

Subventions

Organisme : Toronto General and Western Hospital Foundation
Pays : International

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2020 European Academy of Neurology.

Références

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Auteurs

F Taghdiri (F)

Tanz Centre for Research in Neurodegenerative Diseases, University of Toronto, Ontario, Toronto, Canada.

N Multani (N)

Tanz Centre for Research in Neurodegenerative Diseases, University of Toronto, Ontario, Toronto, Canada.

M Ozzoude (M)

Tanz Centre for Research in Neurodegenerative Diseases, University of Toronto, Ontario, Toronto, Canada.

A Tarazi (A)

Division of Neurology, Krembil Neuroscience Centre, Toronto, Ontario, Canada.
Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.

M Khodadadi (M)

Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.

R Wennberg (R)

Division of Neurology, Krembil Neuroscience Centre, Toronto, Ontario, Canada.
Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.
Institute of Medical Science, University of Toronto, Toronto, Ontario, Canada.

D Mikulis (D)

Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.
Institute of Medical Science, University of Toronto, Toronto, Ontario, Canada.
Division of Neuroradiology, Joint Department of Medical Imaging, University Health Network, Toronto, Ontario, Canada.

R Green (R)

Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.
Department of Rehabilitation Sciences, University of Toronto, Toronto, Ontario, Canada.

B Colella (B)

Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.
Department of Rehabilitation Sciences, University of Toronto, Toronto, Ontario, Canada.

K D Davis (KD)

Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.
Institute of Medical Science, University of Toronto, Toronto, Ontario, Canada.
Department of Surgery, University of Toronto, Toronto, Ontario, Canada.
Division of Brain, Imaging and Behaviour-systems Neuroscience, Krembil Brain Institute, University Health Network, Toronto, Ontario, Canada.

K Blennow (K)

Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, The Sahlgrenska Academy at the University of Gothenburg, Mölndal, Sweden.
Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.

H Zetterberg (H)

Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry, The Sahlgrenska Academy at the University of Gothenburg, Mölndal, Sweden.
Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.
Department of Neurodegenerative Disease, UCL Institute of Neurology, Queen Square, London, UK.
UK Dementia Research Institute at UCL, University College London, London, UK.

C Tator (C)

Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.
Division of Neurosurgery, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.

M C Tartaglia (MC)

Tanz Centre for Research in Neurodegenerative Diseases, University of Toronto, Ontario, Toronto, Canada.
Division of Neurology, Krembil Neuroscience Centre, Toronto, Ontario, Canada.
Canadian Concussion Centre, Toronto Western Hospital, Krembil Brain Institute, University Health Network, Toronto, Canada.
Institute of Medical Science, University of Toronto, Toronto, Ontario, Canada.

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