Elevated Troponin in Patients With Coronavirus Disease 2019: Possible Mechanisms.
COVID-19
Coronavirus
Myocardial injury
Myocarditis
SARS
Troponin
Journal
Journal of cardiac failure
ISSN: 1532-8414
Titre abrégé: J Card Fail
Pays: United States
ID NLM: 9442138
Informations de publication
Date de publication:
Jun 2020
Jun 2020
Historique:
received:
07
04
2020
revised:
14
04
2020
accepted:
15
04
2020
pubmed:
22
4
2020
medline:
10
7
2020
entrez:
22
4
2020
Statut:
ppublish
Résumé
Coronavirus disease 2019 (COVID-19) is a pandemic that has affected more than 1.8 million people worldwide, overwhelmed health care systems owing to the high proportion of critical presentations, and resulted in more than 100,000 deaths. Since the first data analyses in China, elevated cardiac troponin has been noted in a substantial proportion of patients, implicating myocardial injury as a possible pathogenic mechanism contributing to severe illness and mortality. Accordingly, high troponin levels are associated with increased mortality in patients with COVID-19. This brief review explores the available evidence regarding the association between COVID-19 and myocardial injury.
Identifiants
pubmed: 32315733
pii: S1071-9164(20)30357-2
doi: 10.1016/j.cardfail.2020.04.009
pmc: PMC7166030
pii:
doi:
Substances chimiques
Cytokines
0
Troponin
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
470-475Commentaires et corrections
Type : CommentIn
Informations de copyright
Copyright © 2020 Elsevier Inc. All rights reserved.
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