Genetic factors associated with response to as-needed aflibercept therapy for typical neovascular age-related macular degeneration and polypoidal choroidal vasculopathy.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
28 04 2020
Historique:
received: 16 01 2020
accepted: 15 04 2020
entrez: 30 4 2020
pubmed: 30 4 2020
medline: 26 11 2020
Statut: epublish

Résumé

In the present study, we investigated the association between susceptible genetic variants to age-related macular degeneration (AMD) and response to as-needed intravitreal aflibercept injection (IAI) therapy for exudative AMD including both typical neovascular AMD and polypoidal choroidal vasculopathy (PCV) over 12-months. A total of 234 patients with exudative AMD were initially treated with 3 monthly IAI and thereafter as-needed IAI over 12 months. Seven variants of 6 genes including ARMS2 A69S (rs10490924), CFH (I62V:rs800292 and rs1329428), C2-CFB-SKIV2L(rs429608), C3 (rs2241394), CETP (rs3764261) and ADAMTS-9 (rs6795735) were genotyped for all participants using TaqMan technology. After adjusting for age, gender, baseline BCVA and AMD subtype, A (protective) allele of C2-CFB-SKIV2L rs429608 was associated with visual improvement at 12-month (P = 0.003). Retreatment was associated with T(risk) allele of ARMS2 A69S (P = 2.0 × 10

Identifiants

pubmed: 32346038
doi: 10.1038/s41598-020-64301-z
pii: 10.1038/s41598-020-64301-z
pmc: PMC7189239
doi:

Substances chimiques

Recombinant Fusion Proteins 0
aflibercept 15C2VL427D
Receptors, Vascular Endothelial Growth Factor EC 2.7.10.1

Types de publication

Clinical Trial Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

7188

Subventions

Organisme : MEXT | Japan Society for the Promotion of Science (JSPS)
ID : 19K18841
Pays : International

Références

Kawasaki, R. et al. The prevalence of age-related macular degeneration in Asians: a systematic review and meta-analysis. Ophthalmology 117, 921–927, https://doi.org/10.1016/j.ophtha.2009.10.007 (2010).
doi: 10.1016/j.ophtha.2009.10.007 pubmed: 20110127
Sakurada, Y. et al. Prevalence and Genetic Characteristics of Geographic Atrophy among Elderly Japanese with Age-Related Macular Degeneration. PLoS One 11, e0149978, https://doi.org/10.1371/journal.pone.0149978 (2016).
doi: 10.1371/journal.pone.0149978 pubmed: 26918864 pmcid: 4769020
Fritsche, L. G. et al. Seven new loci associated with age-related macular degeneration. Nat Genet 45(433-439), 439e431–432, https://doi.org/10.1038/ng.2578 (2013).
doi: 10.1038/ng.2578
Cheng, C. Y. et al. New loci and coding variants confer risk for age-related macular degeneration in East Asians. Nat Commun 6, 6063, https://doi.org/10.1038/ncomms7063 (2015).
doi: 10.1038/ncomms7063 pubmed: 25629512 pmcid: 4317498
Huang, L. et al. A missense variant in FGD6 confers increased risk of polypoidal choroidal vasculopathy. Nat Genet 48, 640–647, https://doi.org/10.1038/ng.3546 (2016).
doi: 10.1038/ng.3546 pubmed: 27089177
Heier, J. S. et al. Intravitreal aflibercept (VEGF trap-eye) in wet age-related macular degeneration. Ophthalmology 119, 2537–2548, https://doi.org/10.1016/j.ophtha.2012.09.006 (2012).
doi: 10.1016/j.ophtha.2012.09.006 pubmed: 23084240
Lalwani, G. A. et al. A variable-dosing regimen with intravitreal ranibizumab for neovascular age-related macular degeneration: year 2 of the PrONTO Study. Am J Ophthalmol 148(43-58), e41, https://doi.org/10.1016/j.ajo.2009.01.024 (2009).
doi: 10.1016/j.ajo.2009.01.024
Group, C. R. et al. Ranibizumab and bevacizumab for neovascular age-related macular degeneration. N Engl J Med 364, 1897–1908, https://doi.org/10.1056/NEJMoa1102673 (2011).
doi: 10.1056/NEJMoa1102673
Yamashiro, K. et al. A prospective multicenter study on genome wide associations to ranibizumab treatment outcome for age-related macular degeneration. Sci Rep 7, 9196, https://doi.org/10.1038/s41598-017-09632-0 (2017).
doi: 10.1038/s41598-017-09632-0 pubmed: 28835685 pmcid: 5569099
Sakurada, Y. et al. Association of LOC387715 A69S with vitreous hemorrhage in polypoidal choroidal vasculopathy. Am J Ophthalmol 145, 1058–1062, https://doi.org/10.1016/j.ajo.2008.02.007 (2008).
doi: 10.1016/j.ajo.2008.02.007 pubmed: 18400199
Sakurada, Y. et al. Angiographic lesion size associated with LOC387715 A69S genotype in subfoveal polypoidal choroidal vasculopathy. Retina 29, 1522–1526, https://doi.org/10.1097/IAE.0b013e3181af0d72 (2009).
doi: 10.1097/IAE.0b013e3181af0d72 pubmed: 19898184
Sakurada, Y. et al. Role of complement factor H I62V and age-related maculopathy susceptibility 2 A69S variants in the clinical expression of polypoidal choroidal vasculopathy. Ophthalmology 118, 1402–1407, https://doi.org/10.1016/j.ophtha.2010.12.010 (2011).
doi: 10.1016/j.ophtha.2010.12.010 pubmed: 21397333
Brantley, M. A. Jr. et al. Clinical phenotypes associated with the complement factor H Y402H variant in age-related macular degeneration. Am J Ophthalmol 144, 404–408, https://doi.org/10.1016/j.ajo.2007.05.018 (2007).
doi: 10.1016/j.ajo.2007.05.018 pubmed: 17631852 pmcid: 2140051
Hagstrom, S. A. et al. Pharmacogenetics for genes associated with age-related macular degeneration in the Comparison of AMD Treatments Trials (CATT). Ophthalmology 120, 593–599, https://doi.org/10.1016/j.ophtha.2012.11.037 (2013).
doi: 10.1016/j.ophtha.2012.11.037 pubmed: 23337555 pmcid: 3633658
Valverde-Megias, A. et al. ARMS2 A69S polymorphism is associated with the number of ranibizumab injections needed for exudative age-related macular degeneration in a pro re nata regimen during 4 years of follow-up. Graefes Arch Clin Exp Ophthalmol 255, 2091–2098, https://doi.org/10.1007/s00417-017-3748-0 (2017).
doi: 10.1007/s00417-017-3748-0 pubmed: 28744656
Sugiyama, A. et al. Retreatment of Exudative Age-Related Macular Degeneration after Loading 3-Monthly Intravitreal Ranibizumab. Ophthalmologica 239, 52–59, https://doi.org/10.1159/000480439 (2018).
doi: 10.1159/000480439 pubmed: 29045945
Miki, A. et al. Common variants in the complement factor H gene confer genetic susceptibility to central serous chorioretinopathy. Ophthalmology 121, 1067–1072, https://doi.org/10.1016/j.ophtha.2013.11.020 (2014).
doi: 10.1016/j.ophtha.2013.11.020 pubmed: 24365176
de Jong, E. K. et al. Chronic central serous chorioretinopathy is associated with genetic variants implicated in age-related macular degeneration. Ophthalmology 122, 562–570, https://doi.org/10.1016/j.ophtha.2014.09.026 (2015).
doi: 10.1016/j.ophtha.2014.09.026 pubmed: 25439433
Hosoda, Y. et al. CFH and VIPR2 as susceptibility loci in choroidal thickness and pachychoroid disease central serous chorioretinopathy. Proc Natl Acad Sci USA 115, 6261–6266, https://doi.org/10.1073/pnas.1802212115 (2018).
doi: 10.1073/pnas.1802212115 pubmed: 29844195
Yoneyama, S. et al. Genetic Factors Associated with Choroidal Vascular Hyperpermeability and Subfoveal Choroidal Thickness in Polypoidal Choroidal Vasculopathy. Retina 36, 1535–1541, https://doi.org/10.1097/IAE.0000000000000964 (2016).
doi: 10.1097/IAE.0000000000000964 pubmed: 26745149
Abedi, F. et al. Genetic influences on the outcome of anti-vascular endothelial growth factor treatment in neovascular age-related macular degeneration. Ophthalmology 120, 1641–1648, https://doi.org/10.1016/j.ophtha.2013.01.014 (2013).
doi: 10.1016/j.ophtha.2013.01.014 pubmed: 23582991
Yuan, D. et al. Genetic association with response to intravitreal ranibizumab for neovascular age-related macular degeneration in the Han Chinese population. Ophthalmologica 230, 227–232, https://doi.org/10.1159/000355068 (2013).
doi: 10.1159/000355068 pubmed: 24080590
Fukuda, Y. et al. Clinical and genetic characteristics of pachydrusen in patients with exudative age-related macular degeneration. Sci Rep 9, 11906, https://doi.org/10.1038/s41598-019-48494-6 (2019).
doi: 10.1038/s41598-019-48494-6 pubmed: 31417165 pmcid: 31417165
Yoneyama, S. et al. Genetic variants in the SKIV2L gene in exudative age-related macular degeneration in the Japanese population. Ophthalmic Genet 35, 151–155, https://doi.org/10.3109/13816810.2014.921313 (2014).
doi: 10.3109/13816810.2014.921313 pubmed: 24865191

Auteurs

Seigo Yoneyama (S)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

Yoichi Sakurada (Y)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan. sakurada@yamanashi.ac.jp.

Wataru Kikushima (W)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

Atsushi Sugiyama (A)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

Mio Matsubara (M)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

Yoshiko Fukuda (Y)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

Naohiko Tanabe (N)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

Ravi Parikh (R)

New York University School of Medicine, New York, NY, USA.
Manhattan Retina and Eye Consultants, New York, NY, USA.

Fumihiko Mabuchi (F)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

Kenji Kashiwagi (K)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

Hiroyuki Iijima (H)

Department of Ophthalmology, University of Yamanashi, Chuo Yamanashi, Japan.

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