Th22 cells are efficiently recruited in the gut by CCL28 as an alternative to CCL20 but do not compensate for the loss of Th17 cells in treated HIV-1-infected individuals.
Antiretroviral Therapy, Highly Active
Biomarkers
Chemokine CCL20
/ metabolism
Chemokines, CC
/ metabolism
Chemotaxis, Leukocyte
/ genetics
Cytokines
/ metabolism
HIV Infections
/ drug therapy
HIV-1
/ immunology
Host-Pathogen Interactions
/ genetics
Humans
Inflammation Mediators
/ metabolism
Intestinal Mucosa
/ immunology
T-Lymphocyte Subsets
/ immunology
Journal
Mucosal immunology
ISSN: 1935-3456
Titre abrégé: Mucosal Immunol
Pays: United States
ID NLM: 101299742
Informations de publication
Date de publication:
01 2021
01 2021
Historique:
received:
21
09
2019
accepted:
24
03
2020
revised:
19
02
2020
pubmed:
30
4
2020
medline:
13
10
2021
entrez:
30
4
2020
Statut:
ppublish
Résumé
Gut CD4
Identifiants
pubmed: 32346082
doi: 10.1038/s41385-020-0286-6
pii: S1933-0219(22)00122-2
doi:
Substances chimiques
Biomarkers
0
CCL20 protein, human
0
CCL28 protein, human
0
Chemokine CCL20
0
Chemokines, CC
0
Cytokines
0
Inflammation Mediators
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
219-228Références
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