Cardiomyocyte-Specific Deletion of Orai1 Reveals Its Protective Role in Angiotensin-II-Induced Pathological Cardiac Remodeling.
Angiotensin II
/ metabolism
Angiotensins
/ metabolism
Animals
Calcium
/ metabolism
Calcium Signaling
Carrier Proteins
/ metabolism
Female
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Microfilament Proteins
/ metabolism
Muscle Proteins
/ metabolism
Myocardium
/ metabolism
Myocytes, Cardiac
/ metabolism
ORAI1 Protein
/ genetics
Stromal Interaction Molecule 1
/ metabolism
Ventricular Remodeling
/ genetics
Orai1 proteins
calcium
cardiac function
cardiac remodeling
neurohumoral stimulus
Journal
Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052
Informations de publication
Date de publication:
28 04 2020
28 04 2020
Historique:
received:
26
03
2020
revised:
22
04
2020
accepted:
24
04
2020
entrez:
2
5
2020
pubmed:
2
5
2020
medline:
4
3
2021
Statut:
epublish
Résumé
Pathological cardiac remodeling correlates with chronic neurohumoral stimulation and abnormal Ca
Identifiants
pubmed: 32354146
pii: cells9051092
doi: 10.3390/cells9051092
pmc: PMC7290784
pii:
doi:
Substances chimiques
Angiotensins
0
Carrier Proteins
0
Microfilament Proteins
0
Muscle Proteins
0
Myoz2 protein, mouse
0
ORAI1 Protein
0
Orai1 protein, mouse
0
Stim1 protein, mouse
0
Stromal Interaction Molecule 1
0
Angiotensin II
11128-99-7
Calcium
SY7Q814VUP
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
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