Discovery of seven novel mutations of gyrB, parC and parE in Salmonella Typhi and Paratyphi strains from Jiangsu Province of China.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
30 04 2020
Historique:
received: 22 11 2019
accepted: 15 04 2020
entrez: 2 5 2020
pubmed: 2 5 2020
medline: 15 12 2020
Statut: epublish

Résumé

To investigate the prevalence of Salmonella Typhi and Paratyphi resistance to quinolones and characterize the underlying mechanism in Jiangsu Province of China. Antimicrobial susceptibility testing was performed using Kirby-Bauer disc diffusion system. Quinolone resistance-determining region (QRDR), plasmid-mediated quinolone resistance (PMQR) determinant genes were detected by PCR and sequencing. Out of 239 Salmonella isolates, 164 were S. Typhi and 75 were S. Paratyphi. 128 (53.6%) Salmonella isolates were resistant to nalidixic acid; 11 (4.6%) isolates to ciprofloxacin and 66 (27.6%) isolates were intermediate to ciprofloxacin. QRDR were present in 69 S. Typhi isolates, among which mutation at codon 83 (n = 45) and 133 (n = 61) predominated. In S. Paratyphi, the most common mutations were detected in gyrA at codon 83(n = 24) and parC: T57S (n = 8). Seven mutations were first reported in Salmonella isolates including gyrB: S426G, parC: D79G and parE: [S498T, E543K, V560G, I444S, Y434S]. PMQR genes including qnrD1, qnrA1, qnrB4, aac (6')-Ib-cr4 and qnrS1 were detected in 1, 2, 3, 7 and 9 isolates, relatively. High resistance to quinolones in Salmonella remains a serious problem in Jiangsu, China. The presence of the novel mutations increases the complexity of quinolone-resistant genotypes and poses a threat to public health. Subject terms: Salmonella Typhi, Salmonella Paratyphi, antimicrobial resistance, QRDR, PMQR.

Identifiants

pubmed: 32355184
doi: 10.1038/s41598-020-64346-0
pii: 10.1038/s41598-020-64346-0
pmc: PMC7193621
doi:

Substances chimiques

Anti-Bacterial Agents 0
Bacterial Proteins 0
Quinolones 0
Nalidixic Acid 3B91HWA56M
Ciprofloxacin 5E8K9I0O4U
DNA Gyrase EC 5.99.1.3

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

7359

Références

Crump, J. A. & Mintz, E. D. Global trends in typhoid and paratyphoid Fever. Clin. Infect. Dis. 50, 241–246 (2010).
pubmed: 20014951 pmcid: 2798017 doi: 10.1086/649541
Salerno-Goncalves, R., Kayastha, D., Fasano, A., Levine, M. M. & Sztein, M. B. Crosstalk between leukocytes triggers differential immune responses against Salmonella enterica serovars Typhi and Paratyphi. PLoS Neglected Trop. Dis. 13, e0007650 (2019).
doi: 10.1371/journal.pntd.0007650
Crump, J. A., Luby, S. P. & Mintz, E. D. The global burden of typhoid fever. Bull. W. H. O. 82, 346–53 (2004).
pubmed: 15298225
Rowe, B., Ward, L. R. & Threlfall, E. J. Multidrug-Resistant Salmonella typhi: A Worldwide Epidemic. Clin. Infect. Dis. 24, S106–S109 (1997).
pubmed: 8994789 doi: 10.1093/clinids/24.Supplement_1.S106
Wain, J. et al. Molecular Analysis of incHI1 Antimicrobial Resistance Plasmids from Salmonella Serovar Typhi Strains Associated with Typhoid Fever. Antimicrob. Agents Chemother. 47, 2732–2739 (2003).
pubmed: 12936967 pmcid: 182646 doi: 10.1128/AAC.47.9.2732-2739.2003
Wain, J., Hendriksen, R. S., Mikoleit, M. L., Keddy, K. H. & Ochiai, R. L. Typhoid fever. Lancet. 385, 1136–1145 (2015).
pubmed: 25458731 doi: 10.1016/S0140-6736(13)62708-7
Chitnis, S., Chitnis, V., Hemvani, N. & Chitnis, D. S. Ciprofloxacin therapy for typhoid fever needs reconsideration. J. Infec. Chemother. 12, 402–404 (2006).
doi: 10.1007/s10156-006-0472-9
Menezes, G. A., Harish, B. N., Khan, M. A., Goessens, W. & Hays, J. P. Antimicrobial resistance trends in blood culture positive Salmonella Paratyphi A isolates from Pondicherry, India. Indian. J. Med. Microbiol. 16, 222–7 (2016).
doi: 10.4103/0255-0857.180352
Rahman, B. A. et al. Multi-drug resistance and reduced susceptibility to ciprofloxacin among Salmonella enterica serovar Typhi isolates from the Middle East and Central Asia. New Microbes New Infect. 2, 88–92 (2014).
pubmed: 25356352 pmcid: 4184576 doi: 10.1002/nmi2.46
Giraud, E., Brisabois, A., Martel, J. L. & Chaslus-Dancla, E. Comparative studies of mutations in animal isolates and experimental in vitro- and in vivo selected mutants of Salmonella spp. suggest a counterselection of highly fluoroquinolone-resistant strains in the field. Antimicrob. Agents. Chemother. 43, 2131–2137 (1999).
pubmed: 10471553 pmcid: 89435 doi: 10.1128/AAC.43.9.2131
Park, C. H., Robicsek, A., Jacoby, G. A., Sahm, D. & Hooper, D. C. Prevalence in the United States of aac(6′)-Ib-cr encoding a ciprofloxacin modifying enzyme. Antimicrob. Agents. Chemother. 50, 3953–3955 (2006).
pubmed: 16954321 pmcid: 1635235 doi: 10.1128/AAC.00915-06
Cui, X. et al. Prevalence and antimicrobial resistance of Shigella flexneri serotype 2 variant in China. Frontiers in Microbiology. 6, 435 (2015).
pubmed: 25999941 pmcid: 4423435 doi: 10.3389/fmicb.2015.00435
Hooper, D. C. Mechanisms of fluoroquinolone resistance. Drug Resist Updat. 2, 38–55 (1999).
pubmed: 11504468 doi: 10.1054/drup.1998.0068
Nordmann, P. & Poirel, L. Emergence of plasmid-mediated resistance to quinolones in Enterobacteriaceae. J. Antimicrob. Chemother. 56, 463–469 (2005).
pubmed: 16020539 doi: 10.1093/jac/dki245
Poirel, L., Cattoir, V. & Nordmann, P. Plasmid-mediated quinolone resistance; interactions between human, animal, and environmental ecologies. Front Microbiol. 3, 24 (2012).
pubmed: 22347217 pmcid: 3270319 doi: 10.3389/fmicb.2012.00024
Punjabi, N. H. et al. Enteric fever burden in North Jakarta, Indonesia: a prospective, community-based study. J. Infect. Dev. Ctries. 7, 781–787 (2013).
pubmed: 24240034 doi: 10.3855/jidc.2629
Lugito, N. P. H. & Cucunawangsih. Antimicrobial Resistance of Salmonella enterica Serovars Typhi and Paratyphi Isolates from a General Hospital in Karawaci, Tangerang, Indonesia: A Five-Year Review. Int. J. Microbiol. 2017, 6215136 (2017).
Day, M. R. et al. Comparison of phenotypic and WGS-derived antimicrobial resistance profiles of Salmonella enterica serovars Typhi and Paratyphi. J. Antimicrob. Chemother. 73, 365–372 (2017).
doi: 10.1093/jac/dkx379
Misra, R. et al. Antimicrobial susceptibility pattern and sequence analysis of DNA gyrase and DNA topoisomerase IV in Salmonella enterica serovars Typhi and Paratyphi A isolates with decreased susceptibility to ciprofloxacin. Trans. R. Soc. Trop. Med. Hyg. 110, 472–479 (2016).
pubmed: 27618918 doi: 10.1093/trstmh/trw051 pmcid: 27618918
Pramod, P., Binod, L., Ritu, A., Sharad, B. & Prakash, P. Enteric fever caused by Salmonella enterica serovar paratyphi A: an emerging health concern in Nepal. Afr. J. Microbiol. Res. 10, 1784–1791 (2016).
Verma, S., Thakur, S., Kanga, A., Singh, G. & Gupta, P. Emerging Salmonella Paratyphi A enteric fever and changing trends in antimicrobial resistance pattern of Salmonella in Shimla. Indian J. Med. Microbiol. 28, 51–53 (2010).
pubmed: 20061765 doi: 10.4103/0255-0857.58730
Dutta, S. et al. Antimicrobial Resistance, Virulence Profiles and Molecular Subtypes of Salmonella enterica Serovars Typhi and Paratyphi A Blood Isolates from Kolkata, India during 2009–2013. PLoS ONE. 9, e101347 (2014).
pubmed: 25098613 pmcid: 4123848 doi: 10.1371/journal.pone.0101347
Chau, T. T. et al. Antimicrobial Drug Resistance of Salmonella enterica Serovar Typhi in Asia and Molecular Mechanism of Reduced Susceptibility to the Fluoroquinolones. Antimicrobial Agents and Chemotherapy. 51, 4315–4323 (2007).
pubmed: 17908946 pmcid: 2167998 doi: 10.1128/AAC.00294-07
Zhuang, L. et al. Epidemiologic characteristics of typhoid and paratyphoid fever on related drug resistance and molecular types regarding Salmonella typhi and S. paratyphi, in Jiangsu province. Zhonghua liu xing bing xue za zhi. 33, 1269–1272 (212).
Threlfall, E. J., Ward, L. R., Skinner, J. A., Smith, H. R. & Lacey, S. Ciprofloxacin-resistant Salmonella typhi and treatment failure. Lancet. 353, 1590–1591 (1999).
pubmed: 10334265 doi: 10.1016/S0140-6736(99)01001-6
Lunguya, O. et al. Antimicrobial resistance in invasive non-typhoid Salmonella from the Democratic Republic of the Congo: emergence of decreased fluoroquinolone susceptibility and extended-spectrum beta lactamases. PLoS Neglected Trop. Dis. 7, e2103 (2013).
doi: 10.1371/journal.pntd.0002103
Joaquim, R. Mechanisms of resistance to quinolones: target alterations, decreased accumulation and DNA gyrase protection. J. Antimicrob. Chemother. 5, 1109–17 (2003).
Wasyl, D., Hoszowski, A. & Zajc, M. Prevalence and characterisation of quinolone resistance mechanisms in Salmonella spp. Vet. Microbiol. 171, 307–314 (2014).
pubmed: 24613291 doi: 10.1016/j.vetmic.2014.01.040
Eguale, T. et al. Genetic markers associated with resistance to beta-lactam and quinolone antimicrobials in non-typhoidal Salmonella isolates from humans and animals in central Ethiopia. Antimicrob Resist Infect Control. 6, 13 (2017).
pubmed: 28105330 pmcid: 5240271 doi: 10.1186/s13756-017-0171-6
Al-Emran, H. M. et al. A Multicountry Molecular Analysis of Salmonella enterica Serovar Typhi With Reduced Susceptibility to Ciprofloxacin in Sub-Saharan Africa. Clin. Infect. Dis. 62(suppl 1), S42–S46 (2016).
pubmed: 26933020 pmcid: 4772832 doi: 10.1093/cid/civ788
García, C. et al. Intermediate susceptibility to ciprofloxacin among Salmonella enterica serovar Typhi isolates in Lima, Peru. J. Clin. Microbiol. 52, 968–970 (2014).
pubmed: 24371234 pmcid: 3957797 doi: 10.1128/JCM.02663-13
Acheampong, G. et al. Chromosomal and plasmid-mediated fluoroquinolone resistance in human Salmonella enterica infection in Ghana. BMC Infect. Dis. 19, 898 (2019).
pubmed: 31660876 pmcid: 6819380 doi: 10.1186/s12879-019-4522-1
Smith, A. M., Govender, N. & Keddy, K. H. Short Report: Quinolone-Resistant Salmonella Typhi in South Africa, 2003–2007. Epidemiol. Infect. 138, 86–90 (2010).
pubmed: 19563699 doi: 10.1017/S0950268809990331
Eaves, D. J. et al. Prevalence of Mutations within the Quinolone Resistance-Determining Region of gyrA, gyrB, parC, and parE and Association with Antibiotic Resistance in Quinolone-Resistant Salmonella enterica. Antimicrob. Agents Chemother. 48, 4012–4015 (2004).
pubmed: 15388468 pmcid: 521866 doi: 10.1128/AAC.48.10.4012-4015.2004
Jacoby, G. A. Mechanisms of Resistance to Quinolones. Clin. Infect. Dis. 41, S120–S126 (2005).
pubmed: 15942878 doi: 10.1086/428052
Piekarska, K. et al. Co-existence of plasmid-mediated quinolone resistance determinants and mutations in gyrA and parC among fluoroquinolone-resistant clinical Enterobacteriaceae isolated in a tertiary hospital in Warsaw, Poland. Int. J. Antimicrob. Agents. 45, 238–243 (2015).
pubmed: 25468717 doi: 10.1016/j.ijantimicag.2014.09.019
Thong, K. L., Ngoi, S. T., Chai, L. C. & Teh, C. S. Quinolone Resistance Mechanisms Among Salmonella enterica in Malaysia. Microb. Drug Resist. 22, 259–272 (2016).
pubmed: 26683630 doi: 10.1089/mdr.2015.0158
Giedraitienė, A., Vitkauskienė, A., Naginienė, R. & Pavilonis, A. Antibiotic resistance mechanisms of clinically important bacteria. Medicina. 47, 137–146 (2011).
pubmed: 21822035 doi: 10.3390/medicina47030019
Martínez-Martínez, L., Pascual, A. & Jacoby, G. A. Quinolone resistance from a transferable plasmid. Lancet. 351, 797 (1998).
pubmed: 9519952 doi: 10.1016/S0140-6736(97)07322-4
Cattoir, V. et al. Prevalence of qnr genes in Salmonella in France. J. Antimicrob. Chemother. 59, 751–754 (2007).
pubmed: 17307773 doi: 10.1093/jac/dkl547
Kim, J. H., Cho, J. K. & Kim, K. S. Prevalence and characterization of plasmid-mediated quinolone resistance genes in Salmonella isolated from poultry in Korea. Avian Pathol. 42, 221–229 (2013).
pubmed: 23607509 doi: 10.1080/03079457.2013.779636
Luo, Y. et al. Prevalence of β-lactamases and 16S rRNA methylase genes amongst clinical Klebsiella pneumoniae isolates carrying plasmid-mediated quinolone resistance determinants. Int. J. Antimicrob. Agents. 37, 352–355 (2011).
doi: 10.1016/j.ijantimicag.2010.12.018
Jiang, H. X. et al. Multiple transmissible genes encoding fluoroquinolone and third-generation cephalosporin resistance co-located in non-typhoidal Salmonella isolated from food-producing animals in China. Int. J. Antimicrob. Agents. 43, 242–247 (2014).
doi: 10.1016/j.ijantimicag.2013.12.005
Wong, M. H., Yan, M., Chan, E. W., Biao, K. & Chen, S. Emergence of Clinical Salmonella enterica Serovar Typhimurium Isolates with Concurrent Resistance to Ciprofloxacin, Ceftriaxone, and Azithromycin. Antimicrob. Agents Chemother. 58, 3752–3756 (2014).
pubmed: 24752251 pmcid: 4068579 doi: 10.1128/AAC.02770-13
Doublet, B. et al. Extended-spectrum β-lactamase- and AmpC β-lactamase-producing D-tartrate-positive Salmonella enterica serovar Paratyphi B from broilers and human patients in Belgium, 2008–2010. J. Antimicrob. Chemother. 69, 1257–1264 (2014).
pubmed: 24379303 doi: 10.1093/jac/dkt504
Qamar, F. N., Azmatullah, A., Kazi, A. M., Khan, E. & Zaidi, A. K. A three year review of antimicrobial resistance of Salmonella enterica serovars Typhi and Paratyphi A in Pakistan. Int J Antimicrobial Agents. 8, 981–986 (2014).

Auteurs

Huimin Qian (H)

Department of Acute Infectious Disease Prevention and Control, Jiangsu Provincial Center for Disease Prevention and Control, Nanjing, 210029, China.

Siyun Cheng (S)

Xuzhou Medical University School of Medical Technology, Xuzhou, 221004, China.

Guoye Liu (G)

Xuzhou Medical University School of Medical Technology, Xuzhou, 221004, China.

Zhongming Tan (Z)

Department of Acute Infectious Disease Prevention and Control, Jiangsu Provincial Center for Disease Prevention and Control, Nanjing, 210029, China.

Chen Dong (C)

Department of Acute Infectious Disease Prevention and Control, Jiangsu Provincial Center for Disease Prevention and Control, Nanjing, 210029, China.

Jinfeng Bao (J)

Xuzhou Medical University School of Medical Technology, Xuzhou, 221004, China.

Jie Hong (J)

Department of Acute Infectious Disease Prevention and Control, Jiangsu Provincial Center for Disease Prevention and Control, Nanjing, 210029, China.

Dazhi Jin (D)

Centre of Laboratory Medicine, Zhejiang Provincial People Hospital, People's Hospital of Hangzhou Medical College, Hangzhou, Zhejiang, 310014, China.
School of Laboratory Medicine, Hangzhou Medical College, Hangzhou, Zhejiang, 310053, China.

Changjun Bao (C)

Department of Acute Infectious Disease Prevention and Control, Jiangsu Provincial Center for Disease Prevention and Control, Nanjing, 210029, China. bao2000_cn@163.com.

Bing Gu (B)

Xuzhou Medical University School of Medical Technology, Xuzhou, 221004, China. gb20031129@163.com.
Department of Laboratory Medicine, Affiliated Hospital of Xuzhou Medical University, Xuzhou, 221002, China. gb20031129@163.com.

Articles similaires

Vancomycin-associated DRESS demonstrates delay in AST abnormalities.

Ahmed Hussein, Kateri L Schoettinger, Jourdan Hydol-Smith et al.
1.00
Humans Drug Hypersensitivity Syndrome Vancomycin Female Male
Photosynthesis Ribulose-Bisphosphate Carboxylase Carbon Dioxide Molecular Dynamics Simulation Cyanobacteria
T-Lymphocytes, Regulatory Lung Neoplasms Proto-Oncogene Proteins p21(ras) Animals Humans

Pathogenic mitochondrial DNA mutations inhibit melanoma metastasis.

Spencer D Shelton, Sara House, Luiza Martins Nascentes Melo et al.
1.00
DNA, Mitochondrial Humans Melanoma Mutation Neoplasm Metastasis

Classifications MeSH