Graft patency and progression of coronary artery disease after CABG assessed by angiography-derived fractional flow reserve.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
01 10 2020
Historique:
received: 28 11 2019
revised: 15 04 2020
accepted: 27 04 2020
pubmed: 4 5 2020
medline: 15 5 2021
entrez: 4 5 2020
Statut: ppublish

Résumé

Graft occlusion after coronary artery bypass graft surgery (CABG) has been associated with native coronary artery competitive flow. The present study aims to characterize the functional progression of coronary artery disease (CAD) in native vessels after CABG, and to assess the relationship between preoperative FFR as derived from angiography and graft occlusion. Multicenter study of consecutive patients undergoing CABG between 2013 and 2018, in whom a follow-up angiogram had been performed. Serial vessel-fractional flow reserve (vFFR) analyses were obtained in each major native coronary vessel before and after CABG, excluding post-anastomotic segments and graft conduits. In 73 patients, serial angiograms were suitable for vFFR analysis, including 118 grafted (86 arterial and 32 saphenous grafts) and 64 non-grafted vessels. The median time between CABG and follow-up angiography was 2.4 years [IQR 1.5, 3.3]. Functional CAD progression, by means of decline in vFFR, was observed in grafted but not in non-grafted vessels (delta vFFR in grafted vessels 0.10 [IQR 0.05, 0.18] vs. 0.01 [IQR -0.01, 0.03], in non-grafted vessels, p < 0.001). Preoperative vFFR predicted graft occlusion (AUC: 0.66, 95% CI 0.52 to 0.80, p = 0.031). In patients undergoing CABG, preoperative vFFR derived from conventional angiograms without use of pressure wire was able to predict graft occlusion. Graft occlusion was more frequent in vessels with high vFFR values. Grafted native coronary vessels exhibited accelerated functional CAD progression, whereas in non-grafted native coronaries the functional status remained unchanged.

Sections du résumé

BACKGROUND
Graft occlusion after coronary artery bypass graft surgery (CABG) has been associated with native coronary artery competitive flow.
OBJECTIVES
The present study aims to characterize the functional progression of coronary artery disease (CAD) in native vessels after CABG, and to assess the relationship between preoperative FFR as derived from angiography and graft occlusion.
METHODS
Multicenter study of consecutive patients undergoing CABG between 2013 and 2018, in whom a follow-up angiogram had been performed. Serial vessel-fractional flow reserve (vFFR) analyses were obtained in each major native coronary vessel before and after CABG, excluding post-anastomotic segments and graft conduits.
RESULTS
In 73 patients, serial angiograms were suitable for vFFR analysis, including 118 grafted (86 arterial and 32 saphenous grafts) and 64 non-grafted vessels. The median time between CABG and follow-up angiography was 2.4 years [IQR 1.5, 3.3]. Functional CAD progression, by means of decline in vFFR, was observed in grafted but not in non-grafted vessels (delta vFFR in grafted vessels 0.10 [IQR 0.05, 0.18] vs. 0.01 [IQR -0.01, 0.03], in non-grafted vessels, p < 0.001). Preoperative vFFR predicted graft occlusion (AUC: 0.66, 95% CI 0.52 to 0.80, p = 0.031).
CONCLUSIONS
In patients undergoing CABG, preoperative vFFR derived from conventional angiograms without use of pressure wire was able to predict graft occlusion. Graft occlusion was more frequent in vessels with high vFFR values. Grafted native coronary vessels exhibited accelerated functional CAD progression, whereas in non-grafted native coronaries the functional status remained unchanged.

Identifiants

pubmed: 32360649
pii: S0167-5273(19)35905-4
doi: 10.1016/j.ijcard.2020.04.083
pii:
doi:

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

19-25

Informations de copyright

Copyright © 2020. Published by Elsevier B.V.

Déclaration de conflit d'intérêts

Declaration of competing interest CC report receiving research grants from Biosensor, Heart Flow Inc. and Abbott Vascular; and consultancy fees from Heart Flow Inc. and Philips Volcano. JS reports a research grant provided by Cardiopath Ph.D program. TM report receiving consultancy fees from Heart Flow Inc. EM reports institutional grant support from Abbott Vascular, Boston Scientific, and Biotronik. DA reports institutional fees as a speaker and clinical research grants from GE, Bracco, and Heartflow. BDB reports receiving consultancy fees on his behalf from Boston Scientific and Abbott Vascular. The other authors have nothing to disclose. This study was supported by the VZW Cardiovascular Research Centre, Aalst, Belgium.

Auteurs

Carlo Gigante (C)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium; Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Takuya Mizukami (T)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium; Centrum voor Hart- en Vaatziekten, Universitair Ziekenhuis Brussel, Brussels, Belgium; Department of Cardiology, Showa University Fujigaoka Hospital, Kanagawa, Japan.

Jeroen Sonck (J)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium; Department of Advanced Biomedical Sciences, University of Naples, Federico II, Naples, Italy.

Sakura Nagumo (S)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium; Department of Cardiology, Showa University Fujigaoka Hospital, Kanagawa, Japan.

Alessandra Tanzilli (A)

La Sapienza University, Rome, Italy.

Jozef Bartunek (J)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium.

Marc Vanderheyden (M)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium.

Eric Wyffels (E)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium.

Emanuele Barbato (E)

Department of Advanced Biomedical Sciences, University of Naples, Federico II, Naples, Italy.

Giulio Pompilio (G)

Centro Cardiologico Monzino, IRCCS, Milan, Italy; Department of Clinical Sciences and Community Health, Cardiovascular Section, University of Milan, Milan, Italy.

Saima Mushtaq (S)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Antonio Bartorelli (A)

Centro Cardiologico Monzino, IRCCS, Milan, Italy; Department of Biomedical and Clinical Sciences "Luigi Sacco", University of Milan, Milan, Italy.

Bernard De Bruyne (B)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium; Department of Cardiology, Lausanne University Center Hospital, Switzerland.

Daniele Andreini (D)

Centro Cardiologico Monzino, IRCCS, Milan, Italy; Department of Clinical Sciences and Community Health, Cardiovascular Section, University of Milan, Milan, Italy.

Carlos Collet (C)

Cardiovascular Center Aalst, OLV Clinic, Aalst, Belgium. Electronic address: carloscollet@gmail.com.

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