Parkin contributes to synaptic vesicle autophagy in Bassoon-deficient mice.


Journal

eLife
ISSN: 2050-084X
Titre abrégé: Elife
Pays: England
ID NLM: 101579614

Informations de publication

Date de publication:
04 05 2020
Historique:
received: 03 03 2020
accepted: 02 05 2020
pubmed: 5 5 2020
medline: 18 3 2021
entrez: 5 5 2020
Statut: epublish

Résumé

Mechanisms regulating the turnover of synaptic vesicle (SV) proteins are not well understood. They are thought to require poly-ubiquitination and degradation through proteasome, endo-lysosomal or autophagy-related pathways. Bassoon was shown to negatively regulate presynaptic autophagy in part by scaffolding Atg5. Here, we show that increased autophagy in

Identifiants

pubmed: 32364493
doi: 10.7554/eLife.56590
pii: 56590
pmc: PMC7224700
doi:
pii:

Substances chimiques

Bsn protein, mouse 0
Membrane Glycoproteins 0
Nerve Tissue Proteins 0
Sv2b protein, mouse 0
Vesicle-Associated Membrane Protein 2 0
vesicle-associated membrane protein 2, mouse 0
Ubiquitin-Protein Ligases EC 2.3.2.27
parkin protein EC 2.3.2.27

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Federal Government of Germany
ID : SFB958
Pays : International
Organisme : Federal Government of Germany
ID : SFB779/B09
Pays : International
Organisme : BMBF
ID : 20150065
Pays : International

Informations de copyright

© 2020, Hoffmann-Conaway et al.

Déclaration de conflit d'intérêts

SH, MB, KS, AA, KR, CB, KT, TT, KS, CR, EG, CG, CM No competing interests declared

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Auteurs

Sheila Hoffmann-Conaway (S)

German Center for Neurodegenerative Diseases (DZNE), Berlin, Germany.

Marisa M Brockmann (MM)

Charité - Universitätsmedizin Berlin, Institute of Neurobiology, Berlin, Germany.
NeuroCure Cluster of Excellence, Charité - Universitätsmedizin Berlin, Berlin, Germany.

Katharina Schneider (K)

German Center for Neurodegenerative Diseases (DZNE), Berlin, Germany.

Anil Annamneedi (A)

Leibniz Institute for Neurobiology, Magdeburg, Germany.
Center for Behavioral Brain Sciences (CBBS), Magdeburg, Germany.
Institute of Biology (IBIO), Otto von Guericke University Magdeburg, Magdeburg, Germany.

Kazi Atikur Rahman (KA)

German Center for Neurodegenerative Diseases (DZNE), Berlin, Germany.
Einstein Center for Neurosciences Berlin, Berlin, Germany.

Christine Bruns (C)

German Center for Neurodegenerative Diseases (DZNE), Berlin, Germany.

Kathrin Textoris-Taube (K)

Charité - Universitätsmedizin Berlin, Institute of Biochemistry, Core Facility High Throughput Mass Spectrometry, Berlin, Germany.

Thorsten Trimbuch (T)

Charité - Universitätsmedizin Berlin, Institute of Neurobiology, Berlin, Germany.
NeuroCure Cluster of Excellence, Charité - Universitätsmedizin Berlin, Berlin, Germany.

Karl-Heinz Smalla (KH)

Leibniz Institute for Neurobiology, Magdeburg, Germany.
Center for Behavioral Brain Sciences (CBBS), Magdeburg, Germany.

Christian Rosenmund (C)

Charité - Universitätsmedizin Berlin, Institute of Neurobiology, Berlin, Germany.
NeuroCure Cluster of Excellence, Charité - Universitätsmedizin Berlin, Berlin, Germany.

Eckart D Gundelfinger (ED)

Leibniz Institute for Neurobiology, Magdeburg, Germany.
Center for Behavioral Brain Sciences (CBBS), Magdeburg, Germany.
Molecular Neurobiology, Medical Faculty, Otto von Guericke University, Magdeburg, Germany.

Craig Curtis Garner (CC)

German Center for Neurodegenerative Diseases (DZNE), Berlin, Germany.
Charité - Universitätsmedizin Berlin, Institute of Neurobiology, Berlin, Germany.
NeuroCure Cluster of Excellence, Charité - Universitätsmedizin Berlin, Berlin, Germany.

Carolina Montenegro-Venegas (C)

Leibniz Institute for Neurobiology, Magdeburg, Germany.
Center for Behavioral Brain Sciences (CBBS), Magdeburg, Germany.
Institute for Pharmacology and Toxicology, Medical Faculty, Otto von Guericke University, Magdeburg, Germany.

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Classifications MeSH