Allopregnanolone induces antidepressant-like effects through BDNF-TrkB signaling independent from AMPA receptor activation in a rat learned helplessness model of depression.


Journal

Behavioural brain research
ISSN: 1872-7549
Titre abrégé: Behav Brain Res
Pays: Netherlands
ID NLM: 8004872

Informations de publication

Date de publication:
15 07 2020
Historique:
received: 28 02 2020
revised: 12 04 2020
accepted: 20 04 2020
pubmed: 22 5 2020
medline: 26 10 2021
entrez: 22 5 2020
Statut: ppublish

Résumé

Allopregnanolone (ALLO, 3α5α-tetrahydroprogesterone) was found to be effective for depressed patients. Animal models of depression indicate that ALLO is associated with the pathophysiology of depression. Traditional antidepressant drugs produce antidepressant effects via the monoamine system, with consequent up-regulation of brain-derived neurotrophic factor (BDNF). This study was designed to examine whether the antidepressant effects of ALLO involve BDNF-TrkB signaling or α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor activation on the learned helplessness paradigm. The antidepressant-like effect of ALLO infusion into the cerebral ventricle was blocked by coinfusion of TrkB inhibitor ANA-12, but not by co-administration of AMPA receptor antagonist 2,3-dihydroxy-6-nitro-7-sulfoamoylbenzo(f)quinoxaline (NBQX). Thus, the antidepressant-like effect of ALLO involves BDNF signaling independent from AMPA receptor activation.

Identifiants

pubmed: 32437888
pii: S0166-4328(20)30369-7
doi: 10.1016/j.bbr.2020.112670
pii:
doi:

Substances chimiques

ANA 12 compound 0
Antidepressive Agents 0
Azepines 0
Benzamides 0
Brain-Derived Neurotrophic Factor 0
Excitatory Amino Acid Antagonists 0
Quinoxalines 0
Receptors, AMPA 0
2,3-dioxo-6-nitro-7-sulfamoylbenzo(f)quinoxaline 118876-58-7
Pregnanolone BXO86P3XXW
Receptor, trkB EC 2.7.10.1

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

112670

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest None.

Auteurs

Yukihiko Shirayama (Y)

Department of Psychiatry, Teikyo University Chiba Medical Center, Ichihara, Japan; Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba, Japan. Electronic address: shirayama@rapid.ocn.ne.jp.

Yuko Fujita (Y)

Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba, Japan.

Yasunori Oda (Y)

Department of Psychiatry, Chiba University Graduate School of Medicine, Chiba, Japan.

Masaaki Iwata (M)

Department of Neuropsychiatry, Faculty of Medicine, Tottori University, Yonago, Japan.

Katsumasa Muneoka (K)

Department of Psychiatry, Teikyo University Chiba Medical Center, Ichihara, Japan.

Kenji Hashimoto (K)

Division of Clinical Neuroscience, Chiba University Center for Forensic Mental Health, Chiba, Japan.

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Classifications MeSH