Macrophage GIT1 Contributes to Bone Regeneration by Regulating Inflammatory Responses in an ERK/NRF2-Dependent Way.
ANIMAL MODELS
CELLS OF BONE
GENETIC ANIMAL MODELS
INJURY/FRACTURE HEALING
ORTHOPEDICS
STROMAL/STEM CELLS
Journal
Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research
ISSN: 1523-4681
Titre abrégé: J Bone Miner Res
Pays: United States
ID NLM: 8610640
Informations de publication
Date de publication:
10 2020
10 2020
Historique:
received:
13
02
2020
revised:
19
05
2020
accepted:
21
05
2020
pubmed:
28
5
2020
medline:
16
6
2021
entrez:
28
5
2020
Statut:
ppublish
Résumé
Despite the best treatment, approximately 10% of fractures still face undesirable repair. Recently, many studies have focused on the importance of macrophages in bone repair; however, the cellular mechanisms by which they work are not yet fully understood. In this study, we explored the functions of macrophage G-protein-coupled receptor interacting protein 1 (GIT1) in healing a tibial monocortical defect model. Using GIT1
Identifiants
pubmed: 32460388
doi: 10.1002/jbmr.4099
pmc: PMC7689802
doi:
Substances chimiques
Cell Cycle Proteins
0
GTPase-Activating Proteins
0
Git1 protein, mouse
0
NF-E2-Related Factor 2
0
Extracellular Signal-Regulated MAP Kinases
EC 2.7.11.24
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
2015-2031Informations de copyright
© 2020 The Authors. Journal of Bone and Mineral Research published by American Society for Bone and Mineral Research.
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