The Mincle/Syk/NF-κB Signaling Circuit Is Essential for Maintaining the Protumoral Activities of Tumor-Associated Macrophages.


Journal

Cancer immunology research
ISSN: 2326-6074
Titre abrégé: Cancer Immunol Res
Pays: United States
ID NLM: 101614637

Informations de publication

Date de publication:
08 2020
Historique:
received: 09 10 2019
revised: 08 04 2020
accepted: 04 06 2020
pubmed: 14 6 2020
medline: 20 1 2021
entrez: 14 6 2020
Statut: ppublish

Résumé

Tumor-associated macrophages (TAM) have important roles in cancer progression, but the signaling behind the formation of protumoral TAM remains understudied. Here, by single-cell RNA sequencing, we revealed that the pattern recognition receptor Mincle was highly expressed in TAM and significantly associated with mortality in patients with non-small cell lung cancer. Cancer cells markedly induced Mincle expression in bone marrow-derived macrophages (BMDM), thus promoting cancer progression in invasive lung carcinoma LLC and melanoma B16F10

Identifiants

pubmed: 32532809
pii: 2326-6066.CIR-19-0782
doi: 10.1158/2326-6066.CIR-19-0782
doi:

Substances chimiques

CLEC4D protein, human 0
Clecsf8 protein, mouse 0
Cytokines 0
Lectins, C-Type 0
Membrane Proteins 0
NF-kappa B 0
Receptors, Immunologic 0
Syk Kinase EC 2.7.10.2

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1004-1017

Informations de copyright

©2020 American Association for Cancer Research.

Auteurs

Chunjie Li (C)

Department of Medicine and Therapeutics, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shatin, Hong Kong.
Department of Head and Neck Oncology, West China Hospital of Stomatology, Sichuan University, Chengdu, Sichuan, China.

Vivian Weiwen Xue (VW)

Department of Anatomical and Cellular Pathology, State Key Laboratory of Translational Oncology, The Chinese University of Hong Kong, Shatin, Hong Kong.

Qing-Ming Wang (QM)

Department of Medicine and Therapeutics, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shatin, Hong Kong.

Guang-Yu Lian (GY)

Department of Medicine and Therapeutics, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shatin, Hong Kong.

Xiao-Ru Huang (XR)

Department of Medicine and Therapeutics, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shatin, Hong Kong.
Guangdong-Hong Kong Joint Laboratory on Immunological and Genetic Kidney Diseases, Guangdong Academy of Medical Sciences, Guangdong Provincial People's Hospital, Guangzhou, China.

Tin-Lap Lee (TL)

Reproduction, Development and Endocrinology Program, School of Biomedical Sciences, The Chinese University of Hong Kong, Shatin, Hong Kong.

Ka-Fai To (KF)

Department of Anatomical and Cellular Pathology, State Key Laboratory of Translational Oncology, The Chinese University of Hong Kong, Shatin, Hong Kong.

Patrick Ming-Kuen Tang (PM)

Department of Anatomical and Cellular Pathology, State Key Laboratory of Translational Oncology, The Chinese University of Hong Kong, Shatin, Hong Kong. hylan@cuhk.edu.hk patrick.tang@cuhk.edu.hk.

Hui-Yao Lan (HY)

Department of Medicine and Therapeutics, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Shatin, Hong Kong. hylan@cuhk.edu.hk patrick.tang@cuhk.edu.hk.

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Classifications MeSH