TERT C228T mutation in non-malignant bladder urothelium is associated with intravesical recurrence for patients with non-muscle invasive bladder cancer.


Journal

Molecular oncology
ISSN: 1878-0261
Titre abrégé: Mol Oncol
Pays: United States
ID NLM: 101308230

Informations de publication

Date de publication:
10 2020
Historique:
received: 13 04 2020
revised: 28 05 2020
accepted: 05 06 2020
pubmed: 14 6 2020
medline: 11 9 2021
entrez: 14 6 2020
Statut: ppublish

Résumé

Telomerase reverse transcriptase (TERT) promoter mutations are frequently found in tumors or urine from patients with urothelial carcinoma (UC). TERT promoter mutations are also detected in urine from patients with no evidence of cancer but are associated with subsequent UC development. Mutations in the TERT promoter are thought to be present in nonmalignant urothelium (NMU) during early stages of tumor formation prior to pathological change, but this has not been proven directly. In this proof-of-concept study, we investigated the clinical utility of TERT promoter mutation analysis in NMU of patients with non-muscle-invasive bladder cancer (NMIBC). This single-institute study included 53 primary tumors and 428 systematic bladder biopsy specimens from 54 patients with NMIBC. All patients underwent systematic random biopsy and transurethral resection of the bladder tumor. Genomic DNA was analyzed for TERT C228T and C250T mutations using droplet digital PCR (ddPCR). The association between TERT promoter mutation of NMU and bladder recurrence was examined by the Kaplan-Meier method and Cox proportional hazards model. Of the 54 patients, 16 (29.6%) had a TERT C228T mutation and three (5.6%) had a TERT C250T mutation in NMU. Of 428 biopsy specimens, the TERT C228T mutation was detected in 9% (31/364) of normal urothelium, 27% (4/15) of urothelial dysplasia (UD), 50% (9/18) of UD suspicious for carcinoma in situ (CIS), and 58% (18/31) of CIS. During follow-up (median: 3.7 years), 22 (40.7%) patients experienced bladder recurrence and five (9.3%) experienced disease progression. Cox proportional hazard analysis showed that TERT C228T mutation in NMU was significantly associated with bladder recurrence after adjustment for cofounding factors (P = 0.0128). Thus, TERT C228T mutation was detected in NMU, which was a reliable independent prognostic factor of bladder tumor recurrence.

Identifiants

pubmed: 32533903
doi: 10.1002/1878-0261.12746
pmc: PMC7530786
doi:

Substances chimiques

TERT protein, human EC 2.7.7.49
Telomerase EC 2.7.7.49

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2375-2383

Informations de copyright

© 2020 The Authors. Published by FEBS Press and John Wiley & Sons Ltd.

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Auteurs

Yujiro Hayashi (Y)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Kazutoshi Fujita (K)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.
Department of Urology, Kindai University Faculty of Medicine, Osakasayama, Japan.

Satoshi Nojima (S)

Department of Pathology, Osaka University Graduate School of Medicine, Suita, Japan.

Eisuke Tomiyama (E)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Makoto Matsushita (M)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Yoko Koh (Y)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Kosuke Nakano (K)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Cong Wang (C)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Yu Ishizuya (Y)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Taigo Kato (T)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.
Department of Urological Immuno-oncology, Osaka University Graduate School of Medicine, Suita, Japan.

Koji Hatano (K)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Atsunari Kawashima (A)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Takeshi Ujike (T)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Motohide Uemura (M)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.
Department of Urological Immuno-oncology, Osaka University Graduate School of Medicine, Suita, Japan.

Ryoichi Imamura (R)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

Eiichi Morii (E)

Department of Pathology, Osaka University Graduate School of Medicine, Suita, Japan.

Norio Nonomura (N)

Department of Urology, Osaka University Graduate School of Medicine, Suita, Japan.

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