Targeted cryoprecipitate transfusion in severe traumatic haemorrhage.


Journal

Injury
ISSN: 1879-0267
Titre abrégé: Injury
Pays: Netherlands
ID NLM: 0226040

Informations de publication

Date de publication:
Sep 2020
Historique:
received: 04 02 2020
revised: 14 05 2020
accepted: 29 05 2020
pubmed: 20 6 2020
medline: 20 5 2021
entrez: 20 6 2020
Statut: ppublish

Résumé

Severe traumatic haemorrhage is the leading cause of death in young adults. Trauma Induced Coagulopathy is a complex and multifactorial phenomenon associated with severe traumatic haemorrhage. Fibrinogen is one of the first coagulation factors to become depleted in TIC and evidence suggests that severely injured trauma patients with hypofibrinogenaemia have poor outcomes. It is postulated that early fibrinogen replacement can improve clinical outcomes. This study investigated cryoprecipitate transfusion in hyopfibrinogeneamic trauma patients. This retrospective, single center, observational study investigated the use of cryoprecipitate in severely injured trauma patients admitted to an Australian Level I Trauma Centre. The primary outcome was time to administration of cryoprecipitate after identification of hypofibrinogenaemia using ROTEM (FIBTEM A5). Data collected included demographics, ISS, laboratory values of coagulation and blood product usage. 71 patients received cryoprecipitate with a median time of 61 minutes [IQR 37-93] from FIBTEM A5 result to initial cryoprecipitate administration. At 24 hours following admission to ED, Clauss Fibrinogen levels increased by 1.30g/L [IQR 0.45-1.85] and FIBTEM A5 assay increased by 8mm [IQR 3.0-11.3]. Changes in both variables were highly significant (p<0.001) and Clauss Fibrinogen versus FIBTEM A5 values showed moderate to strong correlation (R=0.75-0.80). This study demonstrated that early administration of cryoprecipitate was both feasible and efficacious in fibrinogen replacement in severe traumatic haemorrhage. High-level evidence supporting cryoprecipitate or fibrinogen concentrate replacement with regards to efficacy and feasibility is required to guide future clinical practice. This study provided baseline data to inform the design of further clinical trials investigating fibrinogen replacement in traumatic haemorrhage.

Sections du résumé

BACKGROUND BACKGROUND
Severe traumatic haemorrhage is the leading cause of death in young adults. Trauma Induced Coagulopathy is a complex and multifactorial phenomenon associated with severe traumatic haemorrhage. Fibrinogen is one of the first coagulation factors to become depleted in TIC and evidence suggests that severely injured trauma patients with hypofibrinogenaemia have poor outcomes. It is postulated that early fibrinogen replacement can improve clinical outcomes. This study investigated cryoprecipitate transfusion in hyopfibrinogeneamic trauma patients.
METHODS METHODS
This retrospective, single center, observational study investigated the use of cryoprecipitate in severely injured trauma patients admitted to an Australian Level I Trauma Centre. The primary outcome was time to administration of cryoprecipitate after identification of hypofibrinogenaemia using ROTEM (FIBTEM A5). Data collected included demographics, ISS, laboratory values of coagulation and blood product usage.
RESULTS RESULTS
71 patients received cryoprecipitate with a median time of 61 minutes [IQR 37-93] from FIBTEM A5 result to initial cryoprecipitate administration. At 24 hours following admission to ED, Clauss Fibrinogen levels increased by 1.30g/L [IQR 0.45-1.85] and FIBTEM A5 assay increased by 8mm [IQR 3.0-11.3]. Changes in both variables were highly significant (p<0.001) and Clauss Fibrinogen versus FIBTEM A5 values showed moderate to strong correlation (R=0.75-0.80).
CONCLUSION CONCLUSIONS
This study demonstrated that early administration of cryoprecipitate was both feasible and efficacious in fibrinogen replacement in severe traumatic haemorrhage. High-level evidence supporting cryoprecipitate or fibrinogen concentrate replacement with regards to efficacy and feasibility is required to guide future clinical practice. This study provided baseline data to inform the design of further clinical trials investigating fibrinogen replacement in traumatic haemorrhage.

Identifiants

pubmed: 32553426
pii: S0020-1383(20)30472-1
doi: 10.1016/j.injury.2020.05.044
pii:
doi:

Substances chimiques

Fibrinogen 9001-32-5

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1949-1955

Informations de copyright

Copyright © 2020. Published by Elsevier Ltd.

Déclaration de conflit d'intérêts

Declaration of Competing Interest JW has received educational and research support from CSL Behring and Haemoview Diagnostics. The other authors declare no conflict of interest.

Auteurs

Debbie Ho (D)

Principal House Officer, Department of Surgery, Gold Coast Hospital and Health Service; Lecturer, Griffith University, Australia. Electronic address: debbie.ho@health.qld.gov.au.

Erick Chan (E)

Principal House Officer, Department of Surgery, Gold Coast Hospital and Health Service; Lecturer, Griffith University, Australia. Electronic address: Erick.Chan@health.qld.gov.au.

Don Campbell (D)

Deputy Director, Department of Trauma, Gold Coast Hospital and Health Service; Senior Lecturer, Griffith University, Australia. Electronic address: Don.Campbell@health.qld.gov.au.

Elizabeth Wake (E)

Research Coordinator, Department of Trauma, Gold Coast Hospital and Health Service, Australia. Electronic address: Elizabeth.Wake@health.qld.gov.au.

Kerin Walters (K)

Research Coordinator, Intensive Care Unit, Gold Coast Hospital and Health Service, Australia. Electronic address: Kerin.Walters@health.qld.gov.au.

Andrew C Bulmer (AC)

Associate Professor, School of Medical Science, Griffith University, Australia. Electronic address: a.bulmer@griffith.edu.au.

James McCullough (J)

Staff Specialist, Intensive Care Unit, Gold Coast Hospital and Health Service, Australia. Electronic address: James.McCullough@health.qld.gov.au.

Martin Wullschleger (M)

Director, Department of Trauma and Surgery, Gold Coast Hospital and Health Service, Australia; Professor, Griffith University, Australia. Electronic address: Martin.Wullschleger@health.qld.gov.au.

James Winearls (J)

Staff Specialist, Intensive Care Unit, Gold Coast Hospital and Health Service, Australia; Consultant Intensivist, St Andrew's War Memorial Hospital, Australia; Senior Lecturer, University of Queensland, Australia. Electronic address: James.Winearls@health.qld.gov.au.

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